Peds Hour 9 PDF

PE a eEi of pregnancy late preterm birth 35ᵗʰ to 37ᵗʰ weeks may not need to go to the Neonatal intensive care unit health conditions of the mother may contribute to preterm birth preeclampsia diabetes heart disease kidney disease multiples cervical...

PE a eEi of pregnancy late preterm birth 35ᵗʰ to 37ᵗʰ weeks may not need to go to the Neonatal intensive care unit health conditions of the mother may contribute to preterm birth preeclampsia diabetes heart disease kidney disease multiples cervical complications drug use preeclampsia or premature rupture may also cause premature birth often the cause is unknown in crisis need to get baby out Nalintensivecare the level of caTsdictatedby gestational at the  ge time of birth congenital issues level 1 lowest basic care level 4 highest most advanced care the patient name in the system is usually listed as last name male or female baby a or baby b for twins babies do not have a legal name until birth certificate is received Potential Problems of the tEemmaFF E sreaP.tn sion feeding difficulties tube fed then breast bottle fed developmental delay build up of bilirubin causes Kernicters phototherapy vision problems premature eyes retinopathy of prematurity even at 40 weeks gestation babies don't have vision  P 9 hearing problems respiratory distress syndrome RespiratoryDistress abream.EE ta fctE most often in babies born before their due date usually before 28 weeks of pregnancy Less often RDS can affect full term newborns more common in NICU babies PEadtmishad.IE surfactant immature muscles and ribs lung compliance alveoli cannot support shape to exchange 02 and CO2 RDS S S inspection tachyprea retractions grunting flaring hares pale purplish red color auscultation diminished breath sounds crackles use small side of stethoscope treatment interventions positioning raise HOB turn on side place prone environment adjust stimuli feeding stimulates bathing breathing process monitor CPAP ET tube 02 levels ABO's Potential problems bronchopulmonary dysplasia BPD retrolental fibroplasia vasoconstriction of retinal vessels prevention monitor ABG's and 02 levels Bronchopulmonar.PL Pl8 EF warns most often those born prematurely and needing oxygen therapy in BPD the and the airways bronchi lungs are damaged causing tissue destruction dysplasia in the tiny air sacs of the lung alveoli alveoli don't return to normal size the the child the lower the muscle tone younger and symptoms Signs rapid breathing 60 70 breaths min labored breathing drawing in of the lower chest while breathing in wheezing need more continuous 02 there is aneed for continued oxygentherapy after the gestational age of 36 weeks difficulty breathing repeated lunginfections that may require hospitalization d t ineffective breathing Diagnosis diagnosis Chest X rays of the lungs of babies with respiratory distress syndrome often look like ground glass and those with BPD often look spongy  Blood tests this shows how much oxygen is in the bloodstream and helpsidentify any infection ABG's take very small amounts of blood heel stick squeeze out and put in tube Treatment there is no specific cure for BPD but treatment focuses on minimizing further lung damage and providing support for the infant's lungs allowing them to heal and grow Newborn suffering from BPS are frequently treated in a hospital setting where they can be continuously monitored treat assess monitor reassess REae.IY.EE retinopathy of prematurity a disease entity initially referred to as retriental fibroplasia RLF describes a disorder occurring in premature low birth weight infants in this condition blood vessel development in the retina is abnormal Initially the abnormal  vessels develop in the retinal periphery more likely to have retinal tear detachment or vision problems and signs symptoms common symptoms of retrolental fibroplasia in preterm infants are visual disturbance retinal detachment absent pupillary light reflexes potential blindness dilated or twisted eye vessels opaque retrolental eye membrane retinal edema retinal hemorrhages diagnosis an optometrist makes the diagnosis treatment laser is mosteffective at earlier stages vitrectomy removal of nitrous layer lensectomy removal of lens if left untreated their eyes wander shake or make other unusual movements  their eyes don't follow objects their pupils look white they have trouble recognizing faces retinal detachment distortion lazy eye can lead to blindness Nantizing Entercolitis ned necro cause FEE.EE an underdeveloped premature intestine too little oxygen on blood flows to the intestine at birth or later to the intestinal lining injury heavy growth of bacteria in the intestine that erodes the intestinal wall viral or bacterial infection of the intestine formula feeding babies who get breast milk have a lower risk of NEC  and symptoms signs a swollen on tender belly red blue or gray discoloration of the belly pale trouble feeding food stays in the stomach longer than expected no BM's constipation diarrhea and dark or bloody stools or poop fecal matter can go back into stomach beinglethargic or less active a low on unstable body temperature green vomit containing bile apnea pauses in breathing bradycardia slowed heart rate hypotension low blood pressure fecal matter blood flow dead colon may need to be removed interventions treatments A treatment for NEC may include the following STOP l intervention  g g No tube nose inserting a nasogastric into the stomach to keep the stomachempty intravenous fluids IV for nutrition and fluid replacement antibiotics for infection immunocompromised can't infection fight frequent X rays to monitor the progress of the disease extra oxygen or mechanically assisted breathing isolation procedures such as protective gowns and gloves to keep any infection from spreading in more severe cases babies with NEC may require surgery to remove diseasedintestine or bowel bowel resection connecting part of the intestine or bowel to an ostomy opening on the abdomen