Match the following cancer-related factors with their effects: Chronic inflammation | Angiogenesis TNF | Leukocyte infiltration/activity Dendritic cells | Co-stimulation Endothelia... Match the following cancer-related factors with their effects: Chronic inflammation | Angiogenesis TNF | Leukocyte infiltration/activity Dendritic cells | Co-stimulation Endothelial cell death | Proliferation and differentiation
Understand the Problem
The question is asking us to match various cancer-related factors with their corresponding effects. We will analyze each factor and determine its associated effect based on our knowledge of oncology.
Answer
Chronic inflammation - leukocyte infiltration/activity; Angiogenesis TNF - proliferation and differentiation; Dendritic cells - co-stimulation; Endothelial cell death - angiogenesis.
Chronic inflammation is linked to leukocyte infiltration/activity, angiogenesis TNF is linked to proliferation and differentiation, dendritic cells are linked to co-stimulation, and endothelial cell death is linked to angiogenesis.
Answer for screen readers
Chronic inflammation is linked to leukocyte infiltration/activity, angiogenesis TNF is linked to proliferation and differentiation, dendritic cells are linked to co-stimulation, and endothelial cell death is linked to angiogenesis.
More Information
Chronic inflammation contributes to cancer progression by sustaining cellular proliferation and supporting tumor infiltration by immune cells. TNF is critical for cancer cell proliferation and angiogenesis, while dendritic cells are key to T-cell activation through co-stimulation.
Tips
A common mistake is confusing the specific role of cell types like dendritic cells with generalized immune activities. Differentiating between various cell functions clearly helps avoid this error.
Sources
- Inflammation: A key process in skin tumorigenesis - PMC - pmc.ncbi.nlm.nih.gov
- Monocyte/macrophage infiltration in tumors: modulators - SLB-JLB - jlb.onlinelibrary.wiley.com
- Tumor necrosis factor–like cytokine 1A plays a role in inflammatory - PNAS - pnas.org
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