24.6 Reabsorption and Secretion in Tubules and Collecting Ducts

What happens to the blood concentration of plasma proteins in an individual with renal disease that results in damage to the filtration membrane?

How does renal disease that decreases filtration at the filtration membrane affect the blood concentration of small plasma proteins?

What is the term used to describe the loss of plasma proteins in the urine due to saturation of cellular structures in the kidney tubules?

What is the potential physiologic outcome of having too low of a blood concentration of plasma proteins?

What is the potential physiologic outcome of having too high of a blood concentration of plasma proteins?

What is the primary mechanism by which renal disease that decreases filtration at the filtration membrane leads to an increase in the blood concentration of small plasma proteins?

What is the primary effect of an increase in urine output?

Which of the following is NOT one of the three major variables that influence blood pressure?

What is the primary mechanism by which potassium is reabsorbed in the proximal convoluted tubule?

What is the primary driving force for the reabsorption of potassium in the proximal convoluted tubule?

Which of the following best describes the overall movement of potassium along the nephron?

What is the primary factor that determines the amount of potassium lost in the urine?

What is the primary mechanism by which water is reabsorbed in the renal tubules?

Which of the following hormones decreases sodium reabsorption in the renal tubules?

Approximately what percentage of the water in the tubular fluid is reabsorbed in the proximal convoluted tubule?

What is the term used to describe the water reabsorption that follows sodium reabsorption in the proximal convoluted tubule?

Which of the following statements about aquaporins is true?

What is the average amount of water excreted daily from the approximately 180 L of water filtered by the kidneys?

What contributes approximately one-half of the solutes of the interstitial fluid concentration gradient?

Where is urea removed from the tubular fluid in the nephron?

Why does urea remain within the tubular fluid until it reaches the collecting duct?

What contributes to the concentration gradient in the interstitial fluid?

Which part of the nephron is responsible for reabsorbing a majority of substances after filtration?

What happens as tubular fluid moves through the nephron loop?

What is the primary mode of potassium reabsorption from the tubular fluid into the blood?

Which of the following solutes can also be passively reabsorbed along with potassium?

Approximately what percentage of potassium in the tubular fluid is reabsorbed in the thick ascending limb of the nephron loop?

Which of the following cell types in the collecting tubules and collecting ducts reabsorbs potassium continuously?

What is the primary stimulant for the release of aldosterone from the adrenal cortex?

Approximately what percentage of calcium in the blood becomes part of the filtrate and then the tubular fluid?

What happens to the blood concentration of plasma proteins in an individual with renal disease that results in higher than normal filtration of plasma proteins?

What is the clinical consequence of renal disease that results in lower than normal filtration of plasma proteins?

What happens when renal disease results in damage to the filtration membrane?

What physiologic outcome can occur when an individual has too low blood concentrations of plasma proteins?

How does renal disease resulting in lower than normal filtration of plasma proteins affect blood concentrations?

What occurs when the kidney's function is altered, causing abnormal changes in the filtration of plasma proteins?

What establishes the concentration gradient of the interstitial fluid in the kidney?

Which cells are mainly responsible for regulating the amount of specific substances excreted in urine?

What is the composition of urine?

Where does the fluid leaving the collecting ducts at the renal papilla go next?

Under normal circumstances, why does urine not contain nutrients like glucose and proteins?

What occurs in the proximal convoluted tubule, nephron loop, distal convoluted tubule, collecting tubules, and collecting ducts?

Which of the following is the primary source of creatinine production in the body?

What is the primary function of urea in the kidneys?

What is the primary mechanism by which urea is reabsorbed in the proximal convoluted tubule?

What percentage of the filtered urea is excreted in the urine?

Which of the following is a key difference between urea and creatinine in terms of their processing in the kidneys?

What is the primary reason for the range in normal blood urea levels?

What is the primary mechanism by which proteins are reabsorbed from the tubular fluid?

What happens to very small proteins like angiotensin II in the tubular fluid?

Why is the term 'transported' used instead of 'reabsorbed' when referring to protein movement?

What is the role of lysosomes in the reabsorption of proteins?

What is the primary reason for reabsorbing proteins from the tubular fluid?

Which of the following statements about protein reabsorption is correct?

What is the primary mechanism by which sodium (Na+) moves from the tubular lumen into the tubular cells of the proximal convoluted tubule (PCT)?

What is the primary function of the Na+/K+ pumps embedded in the basolateral membrane of the tubular cells?

Why is the regulation of sodium output critical for maintaining blood sodium levels within a normal homeostatic range?

What is the primary reason for the substantial energy consumption of the Na+/K+ pumps in the nephron?

Which of the following mnemonic devices is mentioned in the text to help remember the function of aldosterone?

What is the significance of the word roots in the term 'atrial natriuretic peptide'?