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Reactive Hyperemia: What is it?
Reactive Hyperemia: What is it?
The increase in blood flow following a brief period of occlusion, leading to heightened tissue oxygenation and temporary redness.
Septic Autolysis: What is it?
Septic Autolysis: What is it?
The liquefaction and breakdown of a thrombus, involving the release of inflammatory mediators and potentially causing complications.
Left Heart Clots: What can they cause?
Left Heart Clots: What can they cause?
Blood clots originating in the left heart chambers may cause ischemic damage to the kidneys, due to the blockage of blood flow.
Thromboxane: Its role in arterial hyperemia?
Thromboxane: Its role in arterial hyperemia?
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Arterial Hyperemia: Temperature change?
Arterial Hyperemia: Temperature change?
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Plasmin: Role in hyperemia?
Plasmin: Role in hyperemia?
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Neurotonic Hyperemia: What is it?
Neurotonic Hyperemia: What is it?
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Obstruction Ischemia: What is it?
Obstruction Ischemia: What is it?
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Diapedesis: What is it?
Diapedesis: What is it?
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Caisson Disease: Cause?
Caisson Disease: Cause?
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Septic Autolysis: Thrombosis complication?
Septic Autolysis: Thrombosis complication?
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Embolism: What is it?
Embolism: What is it?
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Arterial Hyperemia: Complication?
Arterial Hyperemia: Complication?
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Ischemia: Signs?
Ischemia: Signs?
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Venous Hypertension: What is it?
Venous Hypertension: What is it?
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Thrombus Formation: Trigger?
Thrombus Formation: Trigger?
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Neuroparalytic Arterial Hyperemia: What is it?
Neuroparalytic Arterial Hyperemia: What is it?
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Neuroparalytic Arterial Hyperemia: Cause?
Neuroparalytic Arterial Hyperemia: Cause?
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Arterial Hyperemia: Excluding working hyperemia?
Arterial Hyperemia: Excluding working hyperemia?
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Thrombosis: What is it?
Thrombosis: What is it?
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Diapedesis: Role in inflammation?
Diapedesis: Role in inflammation?
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Fluid Removal from Abdominal Cavity: What can occur?
Fluid Removal from Abdominal Cavity: What can occur?
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Arterial Hyperemia: Skin appearance?
Arterial Hyperemia: Skin appearance?
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Working Hyperemia: What is it?
Working Hyperemia: What is it?
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Ischemia: Impact on organs?
Ischemia: Impact on organs?
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Septic Autolysis: Risk?
Septic Autolysis: Risk?
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Venous Hyperemia: Cause?
Venous Hyperemia: Cause?
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Lymphokinesis Insufficiency: Main sign?
Lymphokinesis Insufficiency: Main sign?
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Microcirculation Vessels: Size?
Microcirculation Vessels: Size?
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Ischemia: Blood filling?
Ischemia: Blood filling?
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Neurotonic Arterial Hyperemia: Cause?
Neurotonic Arterial Hyperemia: Cause?
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Physiological Hyperemia: When does it occur?
Physiological Hyperemia: When does it occur?
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Hemorrhagic Syndrome: Radiation disease cause?
Hemorrhagic Syndrome: Radiation disease cause?
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Arterial Hyperemia: Metabolism change?
Arterial Hyperemia: Metabolism change?
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Sladge-phenomenon: What is it?
Sladge-phenomenon: What is it?
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Histamine: Its role?
Histamine: Its role?
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Working Hyperemia: What is it?
Working Hyperemia: What is it?
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Histamine: Its role?
Histamine: Its role?
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Thromboembolism: What is it?
Thromboembolism: What is it?
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Ischemia: Most sensitive organ?
Ischemia: Most sensitive organ?
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Air Embolism: Cause?
Air Embolism: Cause?
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Neurotonic Hyperemia: Example?
Neurotonic Hyperemia: Example?
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Stasis: What is it?
Stasis: What is it?
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Venous Hyperemia: Role in local circulation disorders?
Venous Hyperemia: Role in local circulation disorders?
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Immune Thrombocytopenia: What is it?
Immune Thrombocytopenia: What is it?
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Septic Autolysis: Risk?
Septic Autolysis: Risk?
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Angiospastic Ischemia: What is it?
Angiospastic Ischemia: What is it?
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Left Heart Clots: What can they cause?
Left Heart Clots: What can they cause?
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Ischemia: Signs?
Ischemia: Signs?
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Venous Hyperemia: Description?
Venous Hyperemia: Description?
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Ischemia: Signs?
Ischemia: Signs?
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Hemorrhagic Syndrome: Radiation disease cause?
Hemorrhagic Syndrome: Radiation disease cause?
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Ileofemoral Thrombosis: What is it?
Ileofemoral Thrombosis: What is it?
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Thromboembolism: What is it?
Thromboembolism: What is it?
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Obstructive: What does it describe?
Obstructive: What does it describe?
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Aorta: What is it?
Aorta: What is it?
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Thromboembolism: What is it?
Thromboembolism: What is it?
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Obstructive: What does it describe?
Obstructive: What does it describe?
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Aorta: What is it?
Aorta: What is it?
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Thromboembolism: What is it?
Thromboembolism: What is it?
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Study Notes
Question 1
- Increased blood flow after brief limitation is called reactive hyperemia.
Question 2
- Purulent melting of a thrombus is called septic autolysis.
Question 3
- Blood clots in the left side of the heart can cause pulmonary artery embolism.
Question 4
- Bradykinin is a mediator of inflammation, and it does not cause arterial hyperemia. Prostacyclin doesn't cause arterial hyperemia.
Question 5
- Local temperature typically increases in arterial hyperemia.
Question 6
- Plasmin is not a product of alteration that causes arteriol hyperemia.
Question 7
- Neurotonic arterial hyperemia can develop following a trigeminal neuralgia case.
Question 8
- Venous hyperemia is the condition defined by increased blood volume in tissues resulting from decreased venous outflow.
Question 9
- Pale skin and reduced local temperature are signs of arterial ischemia.
Question 10
- Embolism at caisson works is caused by nitrogen.
Question 11
- The adverse effect of thrombosis is septic autolysis.
Question 12
- The occlusion of lymphatic or blood vessels by foreign particles not normally present in blood or lymph is called embolism.
Question 13
- A common complication of arterial hyperemia is necrosis.
Question 14
- Pale skin and decreased local temperature are signs of ischemia.
Question 15
- Increased blood supply to an organ or tissue due to difficulties in venous drainage is called venous hyperemia.
Question 16
- Thrombus formation begins with vessel endothelium damage and the release of various factors, including serotonin.
Question 17
- Neurotonic arterial hyperemia can develop after vasoconstrictor nerves are cut.
Question 18
- Neuroparalytic arterial hyperemia is a possible result of medication blockade.
Question 19
- Ischemia is most harmful to the brain.
Question 20
- Ultraviolet erythema is a pathological type of arterial hyperemia, but not all arterial hyperemia cases are pathological.
Question 21
- Inflammatory hyperemia, thermal hyperemia, and ultraviolet erythema belong to pathological arterial hyperemia.
Question 22
- Intravital blood or lymph coagulation within blood vessels and the heart is called thrombosis.
Question 23
- Removal of ascitic fluid can cause various circulatory issues in the abdominal cavity, including arterial hyperemia in short term.
Question 24
- Reactive hyperemia is the increase of blood flow following a period of reduced blood flow.
Question 25
- Arterial hyperemia causes red skin coloration.
Question 26
- Arterial hyperemia comes from increased blood filling in the arterioles.
Question 27
- Histamine increases the permeability of microcirculatory blood vessels.
Question 28
- Thrombosis of blood vessels causes venous hyperemia.
Question 29
- Septic autolysis of a thrombus is a dangerous outcome of thrombogenesis.
Question 30
- The term "sludge phenomenon" refers to the aggregation of red blood cells.
Question 31
- The organs most at risk during ischemia are the heart, lungs, kidneys, and intestines.
Question 32
- Increased histamine in tissue due to injury leads to redness, swelling and pain
Question 33
- Venous hyperemia is characterized by fluid accumulation in tissues.
Question 34
- Tissue thromboplastin is the III factor of blood coagulation.
Question 35
- Thromboembolism is the most likely pulmonary artery embolism caused by thigh bone fracture.
Question 36
- Ischemia results from reduced blood flow, not increased flow.
Question 37
- Venous hyperemia develops due to vessel thrombosis.
Question 38
- Edema is a typical symptom of lymphokinesis insufficiency.
Question 39
- Fatty embolism is a potential complication of intravenous fat infusion.
Question 40
- An embolus from the right heart can pass to the left through a ventricular septal defect.
Question 41
- Fatty embolism is the likely complication of accidental oily solution infusion from the incorrect administration.
Question 42
- Gas embolism is a potential problem related to caisson disease.
Question 43
- Capillary vessel diameters are normally less than 100 micrometers.
Question 44
- Blood flow within tissues and organs decreases during ischemia.
Question 45
- Irritation of parasympathetic nerve fibers can cause Neurotonic arterial hyperemia.
Question 46
- Physiological hyperemia happens due to chemical compound effects.
Question 47
- Histamine increases the permeability of microcirculatory blood vessels.
Question 48
- Adhesion of red blood cells to one another is called the "sludge phenomenon."
Question 49
- Metabolism increases in the instance of arterial hyperemia.
Question 50
- The brain is the most sensitive organ concerning ischemia risk.
Question 51
- Thromboembolism is an example of endogenous blood embolism.
Question 52
- Histamine is a substance that boosts capillary wall permeability.
Question 53
- The enhanced blood supply needed for enhanced organ function is called working hyperemia.
Question 54
- Thromboembolism is a likely embolism in large vein damage with negative blood pressure.
Question 55
- Neurotonic arterial hyperemia is a typical cause of prosopalgia.
Question 56
- Stasis is the cessation of blood flow inside vessels, especially capillaries.
Question 57
- The wound described shows venous hyperemia.
Question 58
- Thrombocytopenia is a disorder linked to a reduction in platelets owing to their excessive destruction. This type of thrombocytopenia belongs to the immune-mediated categories.
Question 59
- Septic autolysis is the most hazardous consequence of thrombogenesis.
Question 60
- Angiospastic ischemia is the cause of circulatory issues during emotional outbursts.
Question 61
- Blood clot formation in the heart chambers, specifically on heart valves, is associated with septic endocarditis.
Question 62
- Embolization, typically a thromboembolism, can have severe consequences, including kidney or organ infarction.
Question 63
- Ischemia leads to pale skin and low local temperature.
Question 64
- When peripheral blood circulation disorders induce cyanosis, enlarged volume, and diminished local temperature, it signals venous hyperemia.
Question 65
- Arterial hyperemia displays a pale appearance in the affected skin area and lost/diminished local temperature.
Question 66
- Hemorrhagic syndrome, especially in acute radiation sickness, is very often caused by a reduction in blood clotting factors and increased fibrinolysis factors.
Question 67
- Venous hyperemia is the correct answer to the image identified as Fig. 78
Question 68
- Thromboembolism is a potential outcome of the vascular lesion seen in Figure 79.
Question 69
- The figure, labeled as Fig. 81, suggests a possible cause of ileofemoral thrombosis.
Question 70
- The most common cause of vascular clot formation seen on heart valves is septic endocarditis.
Question 71
- Figure 79 suggests thromboembolism.
Question 72
- The vascular damage in Figure 78 appears to be Obstructive, impeding blood flow.
Question 73
- Aorta is correctly identified as the vessel in Figure 81 because of its peculiar movement.
Question 74
- Thromboembolism is the most probable explanation based on figure 79, not Aseptic autolysis.
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