Hemodynamic Disorders Overview
40 Questions
0 Views

Choose a study mode

Play Quiz
Study Flashcards
Spaced Repetition
Chat to Lesson

Podcast

Play an AI-generated podcast conversation about this lesson

Questions and Answers

What is the term for increased blood flow to an organ due to active dilation of its arterioles?

  • Embolism
  • Hyperemia (correct)
  • Congestion
  • Edema
  • Chronic generalized venous congestion can lead to hypoxia and edema.

    True (A)

    What is a primary cause of acute localized venous congestion?

    Sudden obstruction of venous outflow

    _________ is a condition where fat globules obstruct arterioles and capillaries, commonly seen after bone fractures.

    <p>Fat embolism</p> Signup and view all the answers

    Match the type of congestion with its description:

    <p>Acute localized congestion = Results from sudden obstruction of venous outflow Chronic local congestion = Results from gradual obstruction of venous outflow Acute generalized congestion = Caused by acute heart failure Chronic generalized congestion = Stagnation of venous blood in all organs</p> Signup and view all the answers

    Which of the following is NOT a type of embolism?

    <p>Lymphatic obstruction (A)</p> Signup and view all the answers

    Strangulated hernia is an example of chronic localized venous congestion.

    <p>False (B)</p> Signup and view all the answers

    Name one condition that can cause chronic local venous congestion.

    <p>Liver cirrhosis or pregnancy</p> Signup and view all the answers

    What is the fate of small infarcts in the kidney?

    <p>They are replaced by a depressed scar. (D)</p> Signup and view all the answers

    Hemorrhage can occur only due to pathologic causes.

    <p>False (B)</p> Signup and view all the answers

    What is a common type of hemorrhage that occurs in body cavities?

    <p>Hemothorax</p> Signup and view all the answers

    Edema is classified as ____ when it is localized due to conditions like filariasis.

    <p>lymphatic</p> Signup and view all the answers

    Match the following types of edema with their causes:

    <p>Cardiac edema = CHF Nutritional edema = Kwashiorkor Inflammatory edema = Infection or injury Lymphatic edema = Filariasis</p> Signup and view all the answers

    What is NOT a mechanism of natural arrest of hemorrhage?

    <p>Increased blood flow to the area (D)</p> Signup and view all the answers

    Pitting edema is characterized by the accumulation of fluid that can create temporary skin depressions.

    <p>True (A)</p> Signup and view all the answers

    What is the immediate effect of hemorrhage on the body?

    <p>Fall of blood pressure</p> Signup and view all the answers

    Which type of shock is characterized by the heart failing to pump blood effectively?

    <p>Cardiogenic shock (C)</p> Signup and view all the answers

    Septic shock is caused by viral infections.

    <p>False (B)</p> Signup and view all the answers

    What is one major cause of hypovolemic shock?

    <p>Severe hemorrhage</p> Signup and view all the answers

    Non-pitting edema is characterized by the edema fluid being __________ with tissues.

    <p>united</p> Signup and view all the answers

    Match the type of shock to its cause:

    <p>Cardiogenic shock = Acute heart failure Hypovolemic shock = Severe burns Septic shock = Bacterial infection Anaphylactic shock = IgE-mediated hypersensitivity</p> Signup and view all the answers

    Which compensatory mechanism helps to maintain cardiac and cerebral circulation during shock?

    <p>Vasoconstriction of skin and visceral blood vessels (D)</p> Signup and view all the answers

    In the compensatory stage of shock, blood pressure falls significantly.

    <p>False (B)</p> Signup and view all the answers

    What substance is released by mast cells during anaphylactic shock?

    <p>Vasodilators</p> Signup and view all the answers

    Where does a mixed thrombus typically form?

    <p>In the heart (A), In leg veins (B)</p> Signup and view all the answers

    A thrombus can occur after death.

    <p>False (B)</p> Signup and view all the answers

    What process involves the dissolution of a thrombus?

    <p>Resolution</p> Signup and view all the answers

    Inadequate blood supply to an organ or tissue is known as __________.

    <p>ischemia</p> Signup and view all the answers

    Match the type of ischemia with its description:

    <p>Acute ischemia = Due to sudden complete arterial occlusion Chronic ischemia = Due to gradual incomplete arterial occlusion Localized ischemia = Affects a specific organ or tissue Generalized ischemia = Involves decreased cardiac output</p> Signup and view all the answers

    Which of the following is NOT a fate of thrombi?

    <p>Enlargement of the organ (A)</p> Signup and view all the answers

    A wedge-shaped area is a characteristic feature of infarction.

    <p>True (A)</p> Signup and view all the answers

    What is the primary cause of infarction?

    <p>Sudden arterial occlusion</p> Signup and view all the answers

    What is the primary cause of air embolism?

    <p>Air entering uterine blood vessels (A)</p> Signup and view all the answers

    Small amounts of air entering the bloodstream are harmful.

    <p>False (B)</p> Signup and view all the answers

    What clinical feature is commonly associated with amniotic fluid embolism?

    <p>Sudden respiratory distress</p> Signup and view all the answers

    Thrombosis refers to the formation of an _____ mass of blood elements within the circulatory system during life.

    <p>insoluble</p> Signup and view all the answers

    Match the types of thrombi with their characteristics:

    <p>Pale thrombus = Mainly formed of platelets as rheumatic vegetations Red thrombus = Formed of fibrin network entangling platelets and blood cells</p> Signup and view all the answers

    What is a predisposing factor for thrombosis?

    <p>Inflammation of the vascular wall (D)</p> Signup and view all the answers

    Amniotic fluid embolism is a common condition occurring in labor.

    <p>False (B)</p> Signup and view all the answers

    What substance is released during endothelial injury that initiates thrombus formation?

    <p>Tissue factor</p> Signup and view all the answers

    Flashcards

    Hyperemia

    Increased blood flow to an organ or tissue due to dilated arterioles.

    Congestion

    Increased venous blood flow and stagnation in an organ due to outflow obstruction.

    Localized Congestion (Acute)

    Sudden obstruction of venous outflow causing blood buildup in a specific area.

    Generalized Congestion (Chronic)

    Venous blood stagnation in all organs due to gradual obstruction.

    Signup and view all the flashcards

    Embolism

    Circulating intravascular mass (solid, fluid, or gas) obstructing blood flow.

    Signup and view all the flashcards

    Fat Embolism

    Blockage of arterioles by circulating fat globules, often from a bone fracture.

    Signup and view all the flashcards

    Chronic Venous Congestion Causes

    Conditions like mitral/pulmonary stenosis, lung fibrosis that lead to prolonged blood stagnation.

    Signup and view all the flashcards

    Effects of Chronic Venous Congestion

    General effects like hypoxia, cyanosis, and edema, and organ-specific congestion in organs such as lungs, liver, spleen, and kidneys.

    Signup and view all the flashcards

    Amniotic Fluid Embolism

    A rare complication during pregnancy where amniotic fluid enters the mother's bloodstream, leading to severe complications.

    Signup and view all the flashcards

    Thrombosis

    Formation of a blood clot (thrombus) inside a blood vessel during life.

    Signup and view all the flashcards

    Predisposing Factors of Thrombosis

    Conditions increasing the risk of blood clot formation, including inflammation of blood vessel walls, changes in blood composition (e.g., high platelets), and changes in blood flow (e.g., aneurysm).

    Signup and view all the flashcards

    Pale Thrombus

    A thrombus primarily composed of platelets.

    Signup and view all the flashcards

    Red Thrombus

    A blood clot composed of fibrin strands and blood cells.

    Signup and view all the flashcards

    Pulmonary and systemic embolism

    Obstruction of pulmonary or systemic blood circulation by a clot or foreign material.

    Signup and view all the flashcards

    Toxic effects of fatty acids on vascular endothelium

    Harmful effects of fatty acids on the inner lining of blood vessels (endothelium).

    Signup and view all the flashcards

    Cardiac thrombus

    A thrombus formed in the heart, often associated with conditions like rheumatic fever or infective endocarditis.

    Signup and view all the flashcards

    Arterial thrombus

    A thrombus formed within an artery, frequently in coronary or cerebral arteries.

    Signup and view all the flashcards

    Thrombus vs. Clot

    A thrombus forms during life and is attached to a vessel wall, whereas a clot forms during life or after death and is not attached.

    Signup and view all the flashcards

    Ischemia

    Inadequate blood supply to an organ or tissue.

    Signup and view all the flashcards

    Infarction

    Localized tissue death following complete arterial blockage.

    Signup and view all the flashcards

    Acute ischemia

    Sudden and complete blockage of an artery causing tissue damage.

    Signup and view all the flashcards

    Chronic ischemia

    Gradual and incomplete blockage of an artery, leading to a slow decline in tissue health.

    Signup and view all the flashcards

    Infarction Kidney

    A kidney damaged by lack of blood supply due to a blocked artery. After healing, the damaged area is replaced by a depressed scar.

    Signup and view all the flashcards

    Small Infarcts

    Small areas of dead tissue in the kidney are removed by macrophages and replaced with fibrous tissue, leading to a surface depression.

    Signup and view all the flashcards

    Large Infarcts

    Large areas of dead tissue in the kidney become surrounded by a fibrous capsule and may develop calcium deposits.

    Signup and view all the flashcards

    Hemorrhage

    Escape of blood from blood vessels or the heart into surrounding tissues or body cavities.

    Signup and view all the flashcards

    Internal Hemorrhage

    Bleeding into body cavities like the chest (hemothorax), heart sac (hemopericardium), or abdomen (hemoperitoneum).

    Signup and view all the flashcards

    Interstitial Hemorrhage

    Bleeding into the spaces between tissues, causing purple spots (petechiae), larger bruises (ecchymosis), or blood clots (hematomas).

    Signup and view all the flashcards

    Edema

    Excess fluid buildup in the spaces between cells, causing swelling.

    Signup and view all the flashcards

    Pitting Edema

    Swelling that leaves an indentation when pressed, typical in conditions like heart failure, kidney disease, and malnutrition.

    Signup and view all the flashcards

    Shock

    A life-threatening condition where tissues don't get enough blood flow due to low blood volume or circulation problems.

    Signup and view all the flashcards

    What are the types of shock?

    Shock can be categorized as primary (nervous) or secondary (cardiogenic, hypovolemic, septic, anaphylactic).

    Signup and view all the flashcards

    Cardiogenic Shock

    The heart fails to pump blood properly, causing inadequate blood flow to organs and tissues.

    Signup and view all the flashcards

    Hypovolemic Shock

    Shock caused by a significant decrease in blood volume, leading to low blood pressure.

    Signup and view all the flashcards

    Septic Shock

    Severe shock caused by bacterial infection, often with gram-negative bacteria, due to the release of endotoxins.

    Signup and view all the flashcards

    Anaphylactic Shock

    A severe allergic reaction triggered by an antigen, causing rapid release of chemicals that lead to a drop in blood pressure.

    Signup and view all the flashcards

    Compensatory Mechanisms

    The body's natural responses to shock, including vasoconstriction, increased respiration, and hormonal adjustments.

    Signup and view all the flashcards

    Decompensated Stage

    The stage where compensatory mechanisms fail, resulting in a significant drop in blood pressure, hypoxia, and worsening organ damage.

    Signup and view all the flashcards

    Study Notes

    HEMODYNAMIC DISORDERS

    • Hyperemia: Increased blood flow to an organ or tissue due to active dilation of arterioles. This can be physiological (exercise) or pathological (inflammation).

    • Congestion: Increased venous flow and blood stasis in an organ due to venous outflow obstruction. This can be localized (e.g., strangulated hernia) or generalized (e.g., heart failure).

      • Acute localized venous congestion: Results from sudden obstruction of venous outflow (e.g., volvulus, intussusception).
      • Chronic localized venous congestion: Results from gradual obstruction (e.g., liver cirrhosis, pregnancy).
      • Acute generalized venous congestion: Increased venous pressure throughout the viscera (e.g., acute heart failure).
      • Chronic generalized venous congestion: Stagnation of venous blood in all organs and tissues due to various causes (e.g., cardiac issues, lung fibrosis). Results in hypoxia, cyanosis, and edema, as well as organ congestion (lungs, liver, spleen, kidneys).

    EMBOLISM

    • Definition: Intravascular insoluble solid, fluid, or gaseous mass circulating in the blood.

      • Types: Detached thrombi, fat (bone fractures), air (Caisson disease, lung/neck vein injury), amniotic fluid (labor), tumor cells, parasitic ova, foreign body.
    • Fat embolism: Obstruction of arterioles and capillaries by fat globules, often caused by fractured long bones, extensive tissue trauma, or severe liver fatty change.

      • Effects: Depend on the size and quantity of fat globules. Can lead to pulmonary and systemic embolism and toxic effects of fatty acids on vascular endothelium.
    • Air embolism: Obstruction of vascular flow by a large amount of air or gas (50-100 cc).

      • Causes: Air entering uterine blood vessels (abortion, obstructed labor), large neck veins (stab wounds), pleural sac (pneumothorax), uterine tube insufflation.
      • Effects: Small amounts are harmless; large amounts can impair heart action and be fatal.

    AMNIOTIC FLUID EMBOLISM

    • Definition: Introduction of amniotic fluid and its contents into maternal circulation through the utero-placental site.
    • Pathogenesis: Often related to strong uterine contractions causing fetal membrane tears or placental vein rupture. Amniotic fluid components (meconium, lanugo, cells, mucin, fat) can mechanically obstruct maternal pulmonary circulation.
    • Clinical features: Sudden respiratory distress, dyspnea, deep cyanosis, cardiogenic shock, convulsions, and sudden maternal death.

    THROMBOSIS

    • Definition: Formation of an insoluble mass of blood elements within the circulatory system during life.
    • Predisposing factors: Inflammation of the vascular wall, changes in blood composition (thrombocytosis, polycythemia, dehydration, burns), and changes in blood stream (aneurysm).
    • Mechanism of thrombus formation: Endothelial injury, hypercoagulability, and abnormal blood flow interact to form a thrombus.

    ISCHEMIA

    • Definition: Inadequate blood supply to an organ or tissue.
      • Types: Generalized (e.g., congestive heart failure) or localized (e.g., cardiac or renal ischemia).
      • Effects: Can lead to hypertension, chronic renal failure, angina pectoris, myocardial infarction, fainting, and cerebrovascular stroke.
      • Types: Acute (sudden complete arterial occlusion, e.g., embolism) or chronic (gradual incomplete occlusion, e.g., atherosclerosis).

    INFARCTION

    • Definition: Localized area of ischemic necrosis in an organ or tissue due to sudden cutting of its blood supply.
    • Causes: Sudden arterial occlusion
    • Gross picture: Wedge-shaped triangular area, apex at the site of occlusion.
    • Microscopic: Only tissue framework remains after healing.
    • Fate: Small infarcts resolve via macrophage fibrosis; large infarcts form fibrous capsules and may cause dystrophic calcification.

    HEMORRHAGE

    • Definition: Extravasation of blood outside the circulation.
      • Causes: Physiologic (menstruation) and Pathologic (trauma, spontaneous rupture, vasculitis, tumors, hemorrhagic blood diseases, vitamin deficiencies).
      • Mechanisms: Immediate blood pressure drop, local vasoconstriction (platelets, serotonin, thromboxane A2), clot plug formation, and healing.
      • Types: External (e.g., epistaxis), internal (e.g., hemothorax), interstitial (e.g., petechiae, ecchymosis, hematoma).
      • Effects: Local (clotting, organization, hemostasis); systemic (loss of blood volume, anemia, shock if acute and severe).

    EDEMA

    • Definition: Accumulation of fluid in interstitial tissue.
    • Types: Generalized (e.g., cardiac, renal, nutritional, hepatic, allergic, inflammatory, lymphatic); or localised (e.g., inflammatory, lymphatic).
    • Classification: Pitting (soft) edema (fluids displace on pressure); non-pitting (hard) edema (edema can't be displaced as it's part of the structure).

    SHOCK

    • Definition: Widespread hypoperfusion of tissues due to inadequate circulatory volume.
    • Types: Primary (vasovagal), secondary (cardiogenic, hypovolemic, septic, anaphylactic).
    • Nervous shock: Emotional stress, recovery typically occurs.
    • Cardiogenic shock: Heart's inability to pump blood.
    • Hypovolemic shock: Marked drop in blood pressure due to severe blood loss.
    • Septic shock: Bacterial infection with endotoxin release.
    • Anaphylactic shock: Allergic reaction to antigen (e.g., medication).

    COMPENSATORY MECHANISMS IN SHOCK

    • Vasoconstriction of skin and visceral blood vessels: Dilates cerebral & coronary vessels to maintain blood flow to vital organs.
    • Hypoxia: Stimulation of respiratory center to promote hyperventilation.

    CLINICAL STAGES OF SHOCK

    • Compensatory stage: Compensatory mechanisms control blood pressure, recovery occurs.
    • Decompensated stage: Failure of compensatory mechanisms, marked drop in blood pressure and ischemia. With appropriate interventions, this stage is potentially reversible.
    • Irreversible stage: All therapeutic efforts fail to control the condition leading to death.

    Studying That Suits You

    Use AI to generate personalized quizzes and flashcards to suit your learning preferences.

    Quiz Team

    Related Documents

    Chapter 3 Nursingg PDF

    Description

    This quiz covers the concepts of hyperemia and congestion in hemodynamic disorders. Understanding the differences between physiological and pathological states, as well as acute and chronic conditions, is crucial for grasping their clinical implications. Test your knowledge on the causes and effects of these conditions.

    More Like This

    Hemodynamic Disorders Quiz
    10 questions
    Hemodynamic Disorders and Edema Quiz
    16 questions
    Hemodynamic Disorders Lecture 1
    20 questions
    Use Quizgecko on...
    Browser
    Browser