Oral Lacerations Lecture Notes PDF
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These lecture notes cover oral lacerations, detailing the stages of wound healing for various types of oral injuries. Information about skin anatomy and specific wound management approaches for the face and lips within the oral cavity are also provided. This document is a summary of dental emergencies lecture topics.
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DH 410 EMERGENCIES IN DENTAL PRACTICE SELF-STUDY: INADVERTENT LACERATION OF TISSUES IN THE ORAL CAVITY Skin Anatomy Epidermis keratinized squamous epithelium Avascular Dermis dense, fibro-elastic tissue highly vascular cells of dermis mainly f...
DH 410 EMERGENCIES IN DENTAL PRACTICE SELF-STUDY: INADVERTENT LACERATION OF TISSUES IN THE ORAL CAVITY Skin Anatomy Epidermis keratinized squamous epithelium Avascular Dermis dense, fibro-elastic tissue highly vascular cells of dermis mainly fibroblasts responsible for elaboration of collagen, elastin, ground substance Subcutaneous layer (superficial fascia) connects dermis to underlying tissue contains variable amounts of adipose tissue Wound Healing Stages hemostasis inflammation epithelialization fibroplasia contraction scar maturation Wound Healing Inflammation serves to remove bacteria, foreign debris, and devitalized tissue – a biologic debridement if this stage is prolonged (from infection, foreign material, etc.) will get persistent inflammation and result in poor wound healing Wound Healing Epithelialization insutured wounds, surface of wound develops epithelial covering impermeable to water in 24-48 hours Eschar (dead tissue) and surface debris impair this process by inhibiting the migration of the epithelial cells Wound Healing Fibroplasia by fourth day fibroblasts begin synthesizing collagen, initiating scar formation characterized clinically by pebbled red tissue in wound base Wound Healing Contraction movement of skin edges toward center of defect, primarily in direction of underlying muscle everting skin edges at time of repair accounts for the subsequent wound contraction Wound Healing Scar Maturation amount of scar tissue influenced by physical forces acting across wound strength of wound increases rapidly from day 5-17, more slowly for additional 14 days, and further collagen remodeling / maturation for 2 years strength of scar tissue never quite reaches that of unwounded skin Wound Evaluation – History American College of Emergency Physicians: Clinical policy for the initial approach to patients presenting with penetrating extremity trauma. Ann Emerg Med Vol 33 No. 5 May 1999 Identify all extrinsic and intrinsic factors that jeopardize healing and promote infection mechanism of injury time of injury environment in which wound occurred potential contaminants, foreign bodies species of animal if bite patient’s medical problems / immune status tetanus immunization status handedness / vocation (daily activity) Wound Evaluation - History Risk Factors for wound infection (Singer et al. Risk factors for infection in patients with traumatic lacerations. Academic Emergency Medicine. July 1, 2001; 8(7): 716-20) older age diabetes laceration width presence of foreign body Specific Wounds – Face high vascularity therefore low incidence of infection debride minimally to preserve normal facial contours be more aggressive with layered closure Specific Wounds - Lips Anatomy skin, vermilion border, vermilion, oral mucosa obicularis oris Always inspect intraoral and mucosal lip wounds for foreign bodies – esp. teeth and teeth fragments Lacerations through vermilion border use traction to the lips place first stitch at vermilion border – need perfect alignment then repair obicularis oris then repair skin and remainder of lip Specific Wounds - Lips Through and through lacerations 3 layer closure 1st– mucosal layer with rapidly absorbable suture 2nd – orbicularis oris 3rd – skin Specific Wounds – Intraoral Irrigation as per normal lacerations of buccal mucosa and gingiva heal without repair if wound edges not widely separated Small (