Diabetes Student Shell NUPD701 Module 10 PDF
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Centennial College
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Summary
This document is a module on endocrine disorders, specifically diabetes. It includes information about diabetic ketoacidosis (DKA), its causes, manifestations, and possible nursing interventions. It covers different aspects, including objectives, management of DKA, treatment, and assessment.
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Endocrine Disorders: Diabetes NUPD701: MODULE 10 1. Apply the nursing process to a client with: Diabetes Mellitus/Diabetic Ketoacidosis 2. Reflect and explore your personal values and beliefs in relation to the...
Endocrine Disorders: Diabetes NUPD701: MODULE 10 1. Apply the nursing process to a client with: Diabetes Mellitus/Diabetic Ketoacidosis 2. Reflect and explore your personal values and beliefs in relation to the metalanguage (metaparadigms) of nursing. Learning 3. Apply the metalanguage Objectives (metaparadigms) to a patient with: Diabetes Mellitus/Diabetic Ketoacidosis 4. Explore how all the patterns of knowing influence nurses and nursing practice. 5. Apply all the patterns of knowing to a patient with: Diabetes Mellitus/Diabetic Ketoacidosis DM- take away Similarities Treatments & Characteristic Types and Priorities of s of each Differences care Review: Insulin Hormone produced by pancreas that exerts its effect on metabolism and cellular processes in all tissue and organs Uses active transport to carry glucose into cells - * K+ goes with the insulin & glucose Main metabolic effects: 1. Simulates glucose uptake in skeletal and heart muscle 2. Suppresses liver production of glucose and low-density lipoproteins 1. When body is in starvation -> body release glucagon 3. Keeps blood glucose levels from getting too high and keeps lipids in normal range Review: Hyperglycemia Acts as an Osmotic Diuretic – pulls fluid Hyperglycemia causes fluid and electrolyte imbalances leading to the classic manifestations of DM Polyuria: excess urine loss results from osmotic diuresis – glucose spills into urine pulling more fluid, e-lyte’s are lost, severe water loss Polydipsia: excess thirst Polyphagia: excessive hunger Zena’s story Role of the nurse: Health promotion Maintenance to identification, monitoring and education of Goals for client’s at risk of developing DM Diabetes Assessing acute situations Priorities of care*** What education should you be providing the patient and their significant others? How will you assess if the Nursing patient/family understand the Responsibiliti information you have provided them? es? What means will you use to health teach a patient and/or their significant others if they speak a different language, are of a different culture or they are unable to hear or speak? Diabetic ketoacidosis (DKA) DIABETES NURSING MANAGEMENT Etiology*** know causes Undiagnosed DM Usually patient comes in with DKA before having diagnosis of DM Usually from type 1 Inadequate treatment of existing DM Insulin not taken as prescribed, or omitted Infection Type 1 Dehydration owing to illness with vomiting or diarrhea Change in diet, insulin or exercise regimen – keep sugar high before exercising and make sure you have something sweet after exercise to prevent drop DKA How would you define it? Life threatening Occurs most often in people with type 1 DM Usually triggered by a physical stressor Body starts to break down fat at a fast rate Liver turns the fats into fuel (Ketones) which makes the blood acidic Manifestations Hallmarks: D (DRY & HIGH SUGAR) Dehydrated and high glucose K (Ketones & Kussmaul resp. – rapid and fruity) Fat turns into ketones A (Abdominal Pain) A (Acidosis Metabolic) Diabetic ketoacidosis Characterized by: Hyperglycemia Metabolic acidosis Ketosis Dehydration (volume depletion) What is the clinical presentation of DKA? Dehydration: poor skin turgor, Polyuria, Polydipsia altered LOC, lethargy, Weight loss tachycardia, and hypotension Profuse vomiting are often present on Diffuse abdominal pain examination. Lab findings: Fruity smelling breath BS > 14 mmo/L Kussmaul breathing—a deep, pH < 7.35 labored pattern indicative of a Bicarb < 15 mmol/L hyperventilatory response to Ketones in blood and urine metabolic acidosis R. Van Ness-Otunnu and J. B. Hack 2013 Nursing Assessment What is included in your nursing assessment? Review of systems Subjective Data Objective Data Diagnostic Testing R. Van Ness-Otunnu and J. B. Hack 2013 Nursing Assessment Dry mouth Rapid, weak pulse, orthostatic hypotension Thirst Laboured breathing (Kussmaul’s Abdominal pain respirations) N&V Fever Gradually increasing restlessness, Urinary frequency confusion, lethargy – dehydration and acidosis Serum glucose >14mmol/L Flushed, dry skin Glucosuria, ketonuria – should not have** Eyes appear sunken Key significant from type 1 and type Breath odour of acetone 2 Nursing Diagnosis What are some potential nursing diagnosis, and goals for patients experiencing diabetes hyperglycemic crisis? DKA – what is the goal of treatment What is your absolute priority for DKA? And Why? Explain how the empirical knowledge will guide your care? - *FLUID RESUSITICAN IS KEY* - Normal saline is commonly used - Bring BP and reprofuse organs ( kidneys) - After providing fluid, understand the issue/cause - Check electrolytes ( potassium) with insulin moves into the cell (hypokalemia) What is the goal for DKA? Goals of treatment include: Identifying the underlying cause Replace fluid volume, re-establish euglycemia Resolve ketonemia, correct acidosis Improve mental status Optimize renal perfusion, replete electrolytes & minerals Prevent complications MANAGEMENT OF DKA ** super important DKA & HHS DeBeer et al., 2008 Management IV fluid replacement: Correct water & electrolyte deficits over the next 24-48h to replete ECF & restore ICF volume Begin fluid resuscitation with 0.9Nacl solution until BP stabilized and urine output 30- 60ml/hr ** FIRST** Management of electrolytes: K+ IV to correct hypokalemia IV Na bicarb if severe acidosis (pH