Podcast
Questions and Answers
What is the main difference in the metabolism of normal cells compared to cancer cells?
What is the main difference in the metabolism of normal cells compared to cancer cells?
- Cancer cells break down glucose to pyruvate to produce ATP. (correct)
- Normal cells can proliferate due to overactive oncogenes.
- Cancer cells divert metabolites for energy production through glycolysis.
- Normal cells convert glucose to lactate regardless of oxygen levels.
Which characteristic distinguishes cancer cells from normal cells in terms of immune response avoidance?
Which characteristic distinguishes cancer cells from normal cells in terms of immune response avoidance?
- Cancer cells inhibit T cells by secreting antibodies and cytokines.
- Cancer cells express PDL-1 or PDL-2 antigens to deflect the immune response. (correct)
- Cancer cells stimulate the immune system to destroy foreign particles.
- Cancer cells have a higher sensitivity to DNA damage.
What ability do cancer cells possess that allows them to survive and grow by inducing the formation of new blood vessels?
What ability do cancer cells possess that allows them to survive and grow by inducing the formation of new blood vessels?
- Cancer cells can break through the extracellular matrix during invasion.
- Cancer cells can over-activate oncogenes.
- Cancer cells possess an altered metabolism.
- Cancer cells can induce angiogenesis to create new blood vessels. (correct)
How do cancer cells differ from normal cells in terms of proliferation?
How do cancer cells differ from normal cells in terms of proliferation?
What is the main function of having PDL-1 or PDL-2 antigens in cancer cells?
What is the main function of having PDL-1 or PDL-2 antigens in cancer cells?
How do cancer cells differ from normal cells in terms of their interaction with the immune system?
How do cancer cells differ from normal cells in terms of their interaction with the immune system?
What characteristic of cancer cells allows them to greatly exceed Hayflick's limit?
What characteristic of cancer cells allows them to greatly exceed Hayflick's limit?
Which gene acts as a central regulator of cell death by halting the cell cycle in response to DNA damage?
Which gene acts as a central regulator of cell death by halting the cell cycle in response to DNA damage?
How do most cancer cells avoid normal growth suppressor signals in the G1 checkpoint?
How do most cancer cells avoid normal growth suppressor signals in the G1 checkpoint?
What hallmark of cancer involves the ability of cancer cells to bear mutations in their DNA and still undergo mitosis?
What hallmark of cancer involves the ability of cancer cells to bear mutations in their DNA and still undergo mitosis?
Which enzyme is responsible for elongating chromosomal ends (telomeres) in cancer cells?
Which enzyme is responsible for elongating chromosomal ends (telomeres) in cancer cells?
In normal human cells, what phase of the cell cycle do cells enter after reaching Hayflick's limit?
In normal human cells, what phase of the cell cycle do cells enter after reaching Hayflick's limit?
