Classification of Periodontal Diseases 2017

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Questions and Answers

Which characteristic is NOT a clinical sign of gingivitis?

  • Hypersensitivity to temperature (correct)
  • Subgingival restoration margins
  • Erythema
  • Bleeding on gentle probing

Which systemic factor is known to exacerbate plaque-induced gingivitis?

  • Autoimmune disorders
  • Malnutrition (correct)
  • Elevated calcium levels
  • Hyposalivation

What is the primary cause of plaque-induced gingivitis?

  • Excessive fluoride exposure
  • Systemic diseases
  • Inadequate oral hygiene products
  • Bacterial dental biofilm (correct)

Which of the following symptoms is NOT typically reported by patients with gingivitis?

<p>Frequent headaches (A)</p>
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How is gingivitis primarily diagnosed?

<p>Clinical diagnosis (D)</p>
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Which factor may lead to an increased accumulation of dental plaque in individuals with gingivitis?

<p>Prominent subgingival restoration margins (D)</p>
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In the context of gingivitis, what differentiates generalized gingivitis from localized gingivitis?

<p>Localized occurs only in one quadrant of the mouth (C)</p>
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What is one of the significant factors that can lead to the reversibility of gingivitis?

<p>Effective removal of the etiology (A)</p>
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Which of the following is a clinical sign of gingival inflammation?

<p>Enlargement of the interdental papilla (B)</p>
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Which classification describes conditions that are not caused by dental plaque?

<p>Non-dental biofilm induced gingival lesions (D)</p>
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What is the effect of reducing dental plaque on gingival lesions?

<p>It can limit severity (B)</p>
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Which of the following is NOT a risk factor for gingival lesions?

<p>Good oral hygiene (B)</p>
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Which type of gingivitis is characterized by inflammation independent of plaque?

<p>Non-dental biofilm induced gingivitis (C)</p>
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Which of the following is a characteristic of autoimmune diseases affecting gingival tissue?

<p>Enlargement of the gingival size (D)</p>
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Which of these lesions is classified as a neoplasm associated with gingival tissue?

<p>Fibrous epulis (C)</p>
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What is a common misconception regarding gingival pigmentation?

<p>It always indicates severe gum disease (B)</p>
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Which type of gingival condition is primarily associated with hereditary factors?

<p>Hereditary gingival fibromatosis (D)</p>
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What type of viral infection is primarily characterized by multiple vesicles that rupture to form painful ulcers on the gingiva?

<p>Primary herpetic gingivostomatitis (C)</p>
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Which condition could potentially lead to linear gingival erythema in immunocompromised patients?

<p>Generalized gingival candidiasis (D)</p>
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Which of the following is NOT a recognized bacterial origin of gingival lesions?

<p>Candidiasis (D)</p>
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What is a notable feature of contact allergy-related gingival lesions?

<p>Quick resolution upon allergen removal (D)</p>
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Which type of hypersensitivity reaction may present lesions resembling oral lichen planus or leukoplakia?

<p>Delayed hypersensitivity (A)</p>
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What distinguishes localized gingivitis from generalized gingivitis?

<p>Localized gingivitis affects a specific area, while generalized affects the entire gingiva (D)</p>
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Which organism is primarily responsible for generalized gingival candidiasis?

<p>Candida albicans (B)</p>
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Flashcards

Hereditary Gingival Fibromatosis

A genetic disorder causing fibrotic enlargement of the gums.

Bacterial Gingival Infections

Gingival problems caused by bacteria other than plaque-forming bacteria.

Viral Gingival Infections

Gingival problems caused by viruses, often showing vesicles that rupture into ulcers.

Herpetic Gingivostomatitis

A type of viral infection causing painful ulcers in the mouth, including the gums.

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Fungal Gingival Infections

Gingival problems caused by fungi, often presenting as white or red lesions.

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Generalized Gingival Candidiasis

A fungal infection of the gums, often seen in immunocompromised patients.

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Linear Gingival Erythema

A fungal infection limited to the free gums, not responding to plaque removal.

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Contact Allergy (Gingiva)

Gingival reactions to certain materials (toothpaste, mouthwash) after contact.

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Non-dental biofilm-induced gingivitis

Gingivitis caused by factors other than plaque, such as genetics, infections, or immune disorders.

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Plaque-associated gingivitis

Gingivitis primarily caused by dental plaque accumulation.

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Genetic/developmental disorders (gingivitis)

Gum inflammation stemming from genetic or developmental issues, not plaque.

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Specific infections (gingivitis)

Gum inflammation due to bacterial, viral, or fungal infections.

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Inflammatory and immune conditions (gingivitis)

Gum inflammation associated with hypersensitivity reactions or autoimmune diseases.

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Reactive processes (gingival lesions)

Lesions resulting from a reaction to external or internal stimuli (e.g., trauma, allergy).

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Traumatic lesions (gingiva)

Injuries causing changes in the gum tissue, e.g., toothbrushing, chemical burns, or thermal burns.

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Plaque-induced gingivitis

Inflammation of the gums (gingiva) caused only by dental plaque.

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Gingivitis diagnosis

Gingivitis is diagnosed clinically, not by X-rays.

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Gingivitis symptoms

Clinical signs include redness, swelling, bleeding, pain, and difficulty eating.

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Systemic factors in gingivitis

Factors like hormones (puberty, menstrual cycle) and hyperglycemia can worsen gingivitis.

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Oral factors affecting plaque

Factors like subgingival restorations (fillings) and hyposalivation can make plaque buildup worse.

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Gingivitis reversibility

Removing the cause (plaque) can reverse gingivitis.

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Gingival recession and crown lengthening

Gingival recession is evident by a reduced periodontium and a BOP score of ≥10%.

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Characteristics of gingivitis

Inflammation, redness, swelling, pain, bleeding, and loss of function are common characteristics of gingivitis.

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Study Notes

Attempts at Classification

  • Classification of disease is essential for distinct categories to aid clinical and lab diagnosis, and specific treatment.
  • Ideally, criteria should be based on etiology, histopathology, and genetics, rather than age of onset or disease progression rates.
  • Over the past three decades, there have been four major attempts to classify periodontal disease.
  • The 1999 classification of periodontitis was reclassified in 1999 and used for 19 years.
  • Reclassified into chronic, aggressive (local and generalized), necrotizing, and as a systemic disease manifestation.
  • A 2017 workshop established a classification framework for periodontitis.
  • This framework includes a multidimensional staging and grading system that can adapt to new information.

Classification of Periodontal Diseases and Conditions (2017)

  • 1. Periodontal health and gingival diseases and conditions:
    • a. Periodontal health and gingival health
    • b. Dental biofilm-induced gingivitis
    • c. Non-dental biofilm-induced gingival disease
  • 2. Periodontitis:
    • a. Periodontitis
    • b. Necrotizing periodontal diseases
    • c. Periodontitis as a manifestation of systemic disease
  • 3. Other conditions affecting the periodontium:
    • a. Periodontal abscess and endodontic periodontal lesions
    • b. Mucogingival deformity and conditions
    • c. Traumatic occlusal force
    • d. Tooth and prosthetic-related factors
    • e. Peri-implant disease and conditions
      • a. Peri-implant health
      • b. Peri-implant mucositis
      • c. Peri-implantitis
      • d. Peri-implant soft and hard tissue deficiency

Periodontal Health and Gingival Health

  • Defined by the absence of clinically detectable inflammation.
  • There is a biological level of immune surveillance consistent with clinical gingival health and homeostasis.
  • Clinical gingival health can be assessed using a bleeding-on-probing score (BOP%).
  • Clinical gingival health on an intact periodontium is characterized by the absence of bleeding on probing (less than 10%), lack of symptoms, and attachment and bone loss ≤3mm probing pocket depth.
  • Clinical gingival health on a reduced periodontium includes non-periodontitis patients with recession due to any cause except periodontitis. Characterized by the absence of bleeding on probing (less than 10%), and possible reduced clinical attachment and bone levels with probing pocket depth ≤3mm.

Dental Biofilm-Induced Gingivitis

  • Defined at the site level as an inflammatory lesion resulting from interactions between dental plaque biofilm and the host's inflammatory response,
  • Contained within the gingiva, does not extend to periodontal attachment.
  • Reversible by reducing dental plaque at and apical to the gingival margin.
  • Localized gingivitis = BOP score 10% to ≤30%
  • Generalized gingivitis = BOP score >30%
  • Gingivitis on an intact periodontium characterized by BOP score ≥10%, probing pocket depth ≤3mm, no pseudo pocket, no attachment loss, and no radiographic bone loss.
  • Gingivitis on a reduced periodontium is in a non-periodontitis patient characterized by BOP score ≥10%, probing pocket depth ≤3mm, possible presence of attachment loss and/or radiographic bone loss.

Classification of Dental Biofilm-Induced Gingivitis

  • A. Associated with bacterial dental biofilm only
  • B. Mediated by systemic or local risk factors
      1. Systemic conditions
      • a. Sex steroid hormones
      • b. Hyperglycemia
      • c. Leukemia
      • d. Smoking
      • e. Malnutrition
      1. Oral factors enhancing plaque accumulation
      • a. Prominent subgingival restoration margins
      • b. Hyposalivation
  • C. Drug-influenced gingival enlargements

Dental Plaque-Induced Gingivitis, Associated with Specific Infections

  • Genetic/developmental disorders (Hereditary gingival fibromatosis)
  • Bacterial infections ( Neisseria gonorrhoeae, Treponema pallidum, Streptococcal species , others)
  • Viral infections (Herpes simplex, Varicella-zoster, Epstein-Barr)

Inflammatory and Immune Conditions Gingiva (Hypersensitivity)

  • Contact allergy
  • Plasma cell gingivitis:
  • Erythema multiforme

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