Coronary Vascular Disorders PDF

Disorders Coronary vascular most rommon cause is atherosclerosis accumulation of lipid or fatty substances fibrous tissue manifestations caused by myocardial ischemia...

Disorders Coronary vascular most rommon cause is atherosclerosis accumulation of lipid or fatty substances fibrous tissue manifestations caused by myocardial ischemia related to location and degree of vessel obstruation angina stable vs unstable MI heart failure sudden cardiac death Risk factors metabolic syndrome major risks cholesterol abnormalities tabascouse hypertension Diabetes Pectoris Angina chest pain brought on by myocardial ischemia T due to  flow coronary blood Types of anging stable predictable unstable occurs w rest medital emerg variant Vaso spasm blood flow in coronary artery refractory frequent despite meds lifestyle changes surgery necessary is a for Ml Angina warning sign pain may radiate to neck jaw shoulders back or arms usually left other symptoms SOB dizzines dyspnea nausea vomiting Tx work load of hrt beta blockers calcium channel blockers oxygen re perfusion therapy meds Nitroglycerin vasodilator Beta blockers hit rate  V V conduction of impulses calcium channel hit rate I contractility antiplatelet antiloagulant aspirin used if allergy clopidogrel and ticlopidine of aspirin Heparin unstable IIB 111 a agents Glycoprotein angia PCI percutaneous coronary intervention Collaborative Problems acute pulmonary edema pink Frothy Sputum heart Failure shock cardiogenic a cardiac arrest dysrhythmias infarction myocardial  Process The care of patient Nursing tx prevention of angina reduition of anxiety awareness of disease process of prescribed understanding care adherence to self care and absence of tomplications meds exercise Or therpy diet Interventions tx of pain is priority and sit a rest in bed stop activity Assessment includes v5 observe for distress and assess resp pain ECG is assessed or obtained in hospital Oxygen Nitroglycerin and other meds reduce stress anxiety Patient teaching NTG 2 pills sins seek care if emery not working call apart at 3rd pill  Infarction Myocardial destruction results from reduce blood flow due to rupture of atherosclerosis and plaque blocking of artery by thrombus unstable angina plaque ruptures but artery not completely occulated acute coronary syndrome unstable angina myocardial infarction inverted T wave Ischemia ST elevation injury Q wave abnormal doward spike tissue death MI  manifestations and Diagnosis 02 transport results out mi myoglobin Neg rule Pos results not necessarily MI return to normal 12hr5 Mi 3 hrs Peak 4 12 Troponin regulates contractility w in process 6hr5 found in mi levels are for to 2 weeks remain up 73 4 hrs Peak 4 24 return to normal 1 34 to hrt muscle CK MB happen injury w isoenzyme 74 8 Peak 10 24 return to normal mass assay T2 3 Peak 10 18 3 4 days of mi Management my caudial oz demand Or therapy Beta blockers slow hrt reduce O2 consumption slow less 02 fibrinolytics dissolve thrombus watch for reprefussion dysrythmias Ace in hib within 24ns reduces scar tissue mortality rate BP 02 demands ACE inhibits scar tissue  Treatment of MI ECG O2 therapy meds morphine beta blocke aspirin Nitroglycerin BP 02 reduces scar tissue ACE or ticlopidine heprin LMWH clopidogrel a inhibitor glycoprotein Ib bed rest cardiac death few Sudden common mi is most hrs after collaborative Problems Shock after me a lot lardiogenic effusion and Cardiac tamponade Pericardial Fidd blood collecting collecting around HRT KEY