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Dysmetria impairedability tocorrectly measure distance when reaching or stepping Dysphagia impaired or inability to swallow Dystonia involuntarymusclecontraction Ataxia Lossof musclecoordination Typically in LE causingbalane issues Hyperalgesia painsensitiv...

Dysmetria impairedability tocorrectly measure distance when reaching or stepping Dysphagia impaired or inability to swallow Dystonia involuntarymusclecontraction Ataxia Lossof musclecoordination Typically in LE causingbalane issues Hyperalgesia painsensitivity Allodydia Painfulresponse to non nociceptivestim iGlobalii iii Aphasia antmaiinjiitiasiiiiiingioa.siL Lossof all language abilities bothmotor sister region comprehension butdoesntmakesense 811alibi fire toci Motor Apraxia Difficulty iiiiibalize preform learned moter task planning Ischemia Restricted blood flow to an area of tissue causing ad in Oz nutrient What direction of acceleration does semicircularcanalsutricle Saccule detect SemicircularCanals detectangular motion LatHorizontal rotation utilie Sa Ewar.ddeti.PT F'sm ion Leasions to the basalgangliapresent contralaterally or ipsilaterally helowd.fitimiy the Bs What A suppliesthe Anterior Cerebral A somatosensorycortex skcifically If Thet What is nystagmus namedby Nonvoluntaryoscilation of eyes Nambyfastcomponent What is a true statement of nystagmus Thevestibularsystem drives theslowphase orslowcomponent of nystagmus Direct at USE Indiret For Fame SNpets Strigtyma Et SNPC 8 stygie cloth Thulames Feet GpitSNprgft.gg thulans SNprthpi.IE Thalamy is inhibitednot allowingunwanted movement Somebasalnuclei inputarrive in striatum using glutamate as theinhibitory NT False bc glutamate is anexcitatoryNT Inputinto striatum glutamate beingexitatory What istheneuromatrix theory of pain Its a network of neurons in thethalamusCortex limbicsystem makingpainperception influenced byboth sensoryinputs cognitive events w optic tractopticnerve or optic chiasm leasions H4ayhasafg.ptrt Finalstabile Taste tf uiusi Bitemporal c tea Hemianor NasalHemia Else'sParisiantreatisestakeotihm Homonymous Hemianopsia a Cerebral A how would a pt present w a CVA onecan i.fiitiiiiiiiimohasint Ilfutralateral LE weakness sensory loss in morethan u pipcontralateral UE facial weakness sensorylossCanlead to Homonymous Hemianopsia Visual Hallucination Memory deficits PIEpsilatfacial sensory loss Hornerssyndrome constriction eyeliddroop DifficultySwallowing Nystagmus Aidftigo Ipsilat facial weakness Ataxia scalystagmus Ipsilat ataxia dysmetria Difficult eye movement Truestatements of VOR Helps tomaintainstablevisiongazestability duringrapidheadmotion NormalVORspeedof eyemovement is equaltoheadmovement eyesto reflexivelymove in theopposite direction VORhelpskeepobjects infocus bycausing of head movement Lobes its functional areas Parietal iiiiiii.mn ei siiienanonusonalite Function Movement or sensations PrimarySomatosensoryCortex Tempting Function Heaving Balance WernickesAreaAuditoryCortex Infis FunctionSensory decisionmaking motorcontrol processing ottiiit.be FunctionVision How piiiiii.in ilalciataiiisiit ititiia CVA in L CerebralHemisphere Sudden onset of Bsidedweakness numbness L Sideremainsintact What is gate controltheory Waypain isableto be perceived and sent tobrain Components include SGNociceptor largenew T cell eventuallybrain signalnotinhibited before reaching T cell painwillbe Nociceptor if transmitted Largeorderneuronsends signal to inhibitory interneron nociceptive signal wouldbeinhibited not allowing twice fire I SF Icell Largefiber painallows forpainto Examplerubbing Circle of willis IE i Eii All of these are true regarding AVM It is a cluster of abnormally formed bloodvesselsin brain SC It involvesveinsthatstrechenlarge andorbreak Congenital present in 1 of population Damage tothecerebellum causes deficits ipsilaturally or contralaterally decousation Why Double W.hn istahinPoPihityli9htsprefs intatont if notcorrect AlsoCN involved epet Profict thepupil in response to light T.EE itihtantiiuntht.Pi it tw constricts Explain Stereocilia Kinocilia cilia allowforactivation makingyou aware toheadturing Stereociliamoving tokino Oneside becomes activewhileother inhibited IIiiti.tt iititamato li Cerebellarnuclei location Function Dentale location LatHemisphere Function Control of voluntarymovement cog language Sensory function Emboliform Location IntermediateHemispha Function Limb percision Fastigal Location Vermis Flocculonodule Function Motor coordination motor function non eyemovement Globose Location Intermediate Hemisphere FunctionFinetunemovements of arms legs all coordinationandaccuracy What peripheralsensitization responsiveness threshold or nociceptive stim of receptive feilds Thisexplains Hyperalgesia ble it explains pain to nociceptivestim Centralsensitization is Receptive Feild response threshold explaningallodynia True statements of basal ganglia Direct pathwaypromotesmovement whiletheindirect pathway inhibitsmodulates movement GPe andSNprhaveaninbitoryeffect on thethalamus SNpe exerts itsinfluences on striatum Person wstrokelimited toonly B parietal cortex willshow the followingclinicalpresentation Lhemineglect sensory loss more in face andUE hemi If Unaffected Spech Sudden in ICP AccordingtoMonroeKellie Doctrinewhat is themostlikely explanation of response Thenwillbe an increase in CSF drainage via ventricles Differences in the supporting cells of the PNSVSCNS CNS Oligodendrocytes myelintomultipleaxonsIhibition axon regeneration Astrocytes maintain homeostasisregulation of BBBAttachto A Epindymal linefluidfilledareas Regulates Blood CSFBarrier Microglia Phagocitic promotetissuerepair ofaxons regeneration PNS Schwan Myelerationtoonlylaxonpromoteelongation regeneration Satellite Maintainoptimalenvironment fornever by communicating wothers how it Explain the reasoning why a pt maynotbe able to speak butcan sing and would differ if L temporal lobe is affected vs R temporal lobe Spech is controlled on leftside of brain Singing is on Rightside of brain controlled So if someone has a BCVA they can speak butcannotsing So if someonehas a LCVA they can sing butcannot speak Exam 2 am MuscleSpindles Detect changes muscletension in TO Detectchanges in muscle length Diffent SC injuries howthey wouldpresent Brownsequard Hemisection of SC bc of traum or weakness Contralateral pain temp loss Aniopilatshiprealysis AllbutDCMLaffected bc of vascularissuestumorantwedge fracture posthpgal'spineless of pain tempsensationBUTcanstillsensevibration proprioception DCMLaffected É fascierissuesFit antwedge fracture caidutofquinffynd.si proprioception vibrationDCML Total Bparalysis LCST Lumber Sacral N dysfunction bc of tumer or trauma C.inaYlg ininE Example86 yo ptpresents w hyperextension toCSpine injury motenate consistent fifteen.EE ii Damage or trauma to Tiztz Beststatement E spinitiistties Provids info abt unconsiousproprioception Partsof thebrain asidefrom lobes function Diencephalon Autonomic NSSenorymotorintegration Thalamus Central processing of sensory stem Hypothalamus Homeostasis PEMBsensory perception reflexive eyemovementmotor coordination PonsBreathingCircadianrythmfacialsensation Medulla Autonomicfunction HR BPRM Amygdala limbic BasalGanglia Moterplanning dopamine Hippocampalformation Memory Facial recognition Typesof Planes Direct Transverse Horizontal b bottom SagitallongitudinalSide toSide FrontalCoronalFronttoback caudal Headto Rostro tail Brain SC RostrodorsalFronttoback of brain Top Bottom  Types of mechanoreceptors Getless exact w Mitten s p an Function Fine touchVibration ÉÉath superficial Function light touch localization as the1ˢᵗ 19th b ftp fcmnot Pt sis Function Vibration tingleproprioception ÉÉiti it pocepto What is wallerion degeneration distal Occurs tosite ofinjury Cascade of events postinjury causing break down of affected n What are thetypes of nervedamage Neuropraxia DegenNumb tingle Axtontagged ftp.nggpessien separation of axon butNOTsheethslow recovery Completeseverusuallyno recoverypotential Why is a deep reflex what would you expect to see in a tendon pt documented 4 vs It Hyperactive responseHyperreflexia clonus 15 Briskslightresponse Meningial layers superficial to deep 3 meningial spaces LYBtaiett.es Arachne matter Piamortter Spaces ubarachnoid v A CSF what dursiijnimb.sidupiienflTmp Difference in electricpotentin inside outsideG of a nerve cellmembrane rest Threshold is 55mV Resting is 70mV What are the 4 ventricles location flow 2 lateralventure's Cfozialying hemispherecontainschoroidplexus inbodyproducingCSF i.int atticiephalon locatedbtwn medulla pons cerebellum leads tothespinal canal Types of CVAandruptures EDH Btwndura Skull Location CauseRuptureofmiddlemeningialA Headacheconfusion dizziness Nausea gifymptoms Btwn dura arachnoid Location CauseRupture ofbridgingveins AcuteVelocitydependent traume Acuteon chronicfall ofAVM causingRopter sajhii.is fi diicin I nauseaslurred speechenlargedpupil Location Btwnarachnoid pia C Evaumatic Contusion Non traumatic Rupture of AVMor aneyorism clapHeadache confusionnausea Mood personalitychanges Inflight ifeng.de t.in Tissues of CCCBBSSC Traumatic Coup Contrecoup Contusion NontraumaticHTNHem pressure or AVM Rupture SymptomsHeadachedizzinessnauseavomitingnumbness on 1sidevisual orhearingdifficulty Differentiate CNS from PNS CNS PNS BS SC Somatic Autonomic NS SomaticNS Actionsmovements Autobiety's Involuntary Parasympathetic Vs Sympathetic NS Sympathetic vs Parasympathetic Parasympathetic Sympathetic Long pre ganglionic Activation causes HR Shortpost ganglionic Pairedvertebralganglia Craniosacralorigin thoraiolumber origin Slowdown Rest digest Preganglionicneuron releasesAch Pre Postganglionicneurons andpost releases NE use Ach as NT Autonomic tone is when both systems are allowing a balancebtwn bc they areconstantly active What specificFallgory of cholinergic reeptors are found on cardiac musclesmooth muscleandglands Muscarinic Receptor 2 types of cholinergic receptors to Muscarinic Cardiat Sutton 2 Nicotinic PostganglionictoAutonomicAdrenalMedulla skeletalmuscle toallowformuscularcontraction Communicating VSNon Communicating Hydrocephalus Communicating Reabsorbtionissue NonCommunicatingBlockage What is themonroeKelliedoctorine Volume of CSFblood braintisse Pt post AVMruptue affecting PMC PSSC on Lside Weexpect loss of Voluntary movement on Rcontralateral side Finetouch proprioception Vibration on Rcontralateral side Pain temperaturesensation on Rcontralateral side Herniation's locations wherethey occur TIPEns.PE foramen magnum Geathrough lobe compression sub.fiiPeral cinfiaibtianstentoi.at DiencephalonMB shiftdown What is themonosynaptic vs polysynaptic strich reflex YE.isit.mhhonTm iimeitii.isiitiinmiat muscle Poly Pickupfoot bc yousteppedonsum Slow b c 3neuronchain Multiplemuscles contract bc of stim LMN US UMN Ulocation Brain SC DeficitPresents as reflexiaBabinski Spastic ParalysisHypertonia Hyper Muscle Sign up groups Location Nerves exitingSCtomuscle PFitid8aF.is HypotoniaHyporeflexia DenervationAtrophyIndividualSpecificmuscle