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Inflammation Student PPT-FA2022 (4) (1) PDF

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Summary

This document presents information on various digestive system conditions, including inflammation, gallbladder disease, gallstones, cholecystitis, pancreatitis, and diverticular disorder. It covers topics like pathophysiology, clinical manifestations, assessment, diagnostics, treatments, and nursing considerations for each condition.

Full Transcript

Inflammation Acute Abdominal Conditions ALEXANDRA DEES MSN, RN This Photo by Unknown Author is licensed under CC BY-NC-ND https://create.kahoot.it/share/inflammation-fa-22/ d980510e-12c7-4895-a953-988cd6bb9637 Concept of Inflammation  Inflammation, or inflammatory response, is the body’s...

Inflammation Acute Abdominal Conditions ALEXANDRA DEES MSN, RN This Photo by Unknown Author is licensed under CC BY-NC-ND https://create.kahoot.it/share/inflammation-fa-22/ d980510e-12c7-4895-a953-988cd6bb9637 Concept of Inflammation  Inflammation, or inflammatory response, is the body’s protective response injury, allergens, or infection. With infection, this response eliminates pathogens and with injury, it allows for tissue repair (Taylor et al., 2015).  The nurse plays an important role in identifying clients at risk for inflammation as well as provide treatment for clients experiencing inflammatory responses to infection or injury. Gallbladder Disease  Pear-Shaped, hollow, saclike organ  Lies in a shallow depression on the inferior surface of the liver  Connected to the common bile duct (CBD) by the cystic duct  Function  Primary role is to store bile  Bile helps digestive system bread down fats  Bile is a mixture of mainly cholesterol, bilirubin, and bile salts  Composed of water and electrolytes( sodium, K, Ca, Chloride and Bicarb) Gallstones  Cholelithiasis  Hardened deposits of digestive fluid that forms in the gallbladder  Causes  Too much cholesterol (cholesterol gallstones)  Too much bilirubin (pigment gallstones)  Poor emptying of the gallbladder  Risk Factors  Chart 44-1 Cholelithiasis  Clinical Manifestations  Pain and Biliary Colic  Avoid Morphine  Right Abdominal pain, Radiating back and shoulder  Jaundice  Obstruction of the CBD  Bile absorbed by blood giving skin yellow appearance  Changes in Urine and Stool Color  Dark Color Urine  Stool (Grayish or Clay colored)  Vitamin Deficiency  Absorption of Vitamins A,D,E and K Cholecystitis  inflammation of the gallbladder which can be acute or chronic  Calculous  90% of cases  Stone obstructs bile flow  Acalculous  Acute gallbladder inflammation (No obstruction)  Occurs after major surgical procedure Cholecystitis  Clinical Manifestations  Pain  Tenderness  Rigidity of the upper right abdomen (radiates to the midsternal or Right shoulder)  N/V  Empyema ( gallbladder filled with purulent fluid) Gallbladder Disease  Assessment and Diagnostic Findings Table 44-1 – Studies Used in the Diagnosis of Biliary Tract and Pancreatic Disease Page 1420 ERCP Gallbladder disease  Medical Management  Nutritional and Supportive Therapy  IV Fluids, NG Suction, Analgesia, Antibiotic  Dietary Management  Low Fat Diet  Avoid Eggs, cream, pork, fried foods, cheese, rich dressings, gas-forming vegetables, and alcohol  Pharmacologic Therapy  Ursodeoxycholic acid (UDCA) and chenodeoxycholic acid (chenodiol or CDCA) Nonsurgical and Surgical  Nonsurgical Removal of Gallstones  Infusion of Solvents inserted via tube or drain (T-Tube drain)  Catheter and instrument utilizing a basket via a tube  Lithotripsy  ERCP  Surgical  Cholecystectomy  Standard of treatment Laparoscopic Cholecystectomy  Review the Nursing Process  The patient undergoing surgery for Gallblader Disease (Page 1426)  Patient Education  Chart 44-2  Figure 44-5 In laparoscopic cholecystectomy (A), the surgeon Page 1428 makes four small incisions (less than one half inch each) in the abdomen and inserts a laparoscope with a miniature camera through the umbilical incision (B). The camera apparatus displays the gallbladder and adjacent tissues on a screen, allowing the surgeon to visualize the sections of the organ for removal. Pancreas  Function  Aids in digestion of food by: excretingenzymes such as lipase, amylase, and protease secretes hormones to regulate blood sugar levels Pancreatitis  Acute or chronic  Chronic often not detected until severe exocrine function lost  Acute does not lead to chronic unless complications  Chronic can have acute episodes  Gerontologic Considerations  Mortality rate of acute pancreatitis increases with age  Multiple organ dysfunction syndrome (MODS) increases with age  Require aggressive tx to decrease mortality Pancreatitis  Clinical manifestations  Severe epigastric abdominal pain (radiate to the back)  Occurs after heavy meal  Vomiting (Bile-stained)  Assessment  Serum Levels  Diagnostic Tests Pancreatitis  Medical Management  Pain Control  NPO  Maintaining circulatory and fluid volume  Decrease production of pancreatic enzymes  IV Fluid replacement  Electrolyte replacement  NG Tube  Nursing Management-Patient Education  Alcohol Consumption  Diet  High in Carbs, Low protein and Fats  Avoid Beverages with Caffeine  Diagnostic Tests  Drug Therapy Concept Map  Enter Pathophysiology  Signs and Symptoms  Assessment  What assessment findings would support that the patient is having an inflammatory response?  Diagnostics/Labs  What diagnostics or lab values would support an inflammatory process and infection?  Treatment/Medications  Nursing Implications  Priority Nursing Diagnosis  Patient Teaching Diverticular Disorder Patho/Overview  Diverticulum  Saclike herniation of bowel lining  Most common in colon  Diverticulosis  Multiple diverticula  No inflammation or s/s  Risk Factors: low intake dietary fiber, obesity, Hx of smoking cigs, regular use of NSAIDS and acetaminophen, + family hx  Diverticulitis   Diverticula inflamed  Leads to perforation, obstruction, abscess, fistula, peritonitis, hemorrhage Clinical Manifestations  Diverticulosis  Chronic Constipation  Mild S/S  Bowel irregularity  Nausea  Decreased appetite  Bloating  Diverticulitis  Acute Onset ranges from mild to severe, pain LLQ  Change in bowel habits, nausea, fever, leukocytosis  Complications  Abscess, bleeding, peritonitis–(Beware possible Acute Abdomen)  Perforation causes pain over involved segment-local abscess, peritonitis follows  Recurrent diverticulitis-chronic complications-fistula, cramps, narrow stools, obstruction Assessment/Diagnostic Findings  Diverticulosis  Colonoscopy  CBC (Increased WBC), H&H  UA, c+s  Diverticulitis  CT with contrast  ABD Xray  Gerontology Considerations  S/S less pronounced  No pain until infection  Delay reporting out of fear Sx and Cancer Medical Management  Treatment based on complications  Hinchey stages- Table 47-3  Stage 1- treated on an outpatient basis  Acute Diverticulitis with symptoms- hospitalization  Higher stages (3&4) surgery and hospitalization  NPO  IVF  NGT if N/V and distention  Broad spectrum Abx  Opiods for pain  Increased PO intake when s/s resolve- High FIBER diet, Low fat Surgical Management  Usually resolved with medical management  Immediate SX for complications  Stage 3 and 4 diverticulitis- SX possible  2 types of surgery 1 stage- inflamed area removed, end to end anastomosis  Multistage-for complications or perforation End to end anastomosis preffered Stage 4-Hartmann procedure- leads to ostomy creation Nursing Management  Encourage PO fluid intake  Educated soft foods with increased fiber  Exercise program  Eating and defecating schedule  Daily bulk laxatives  Avoid triggers (nuts, popcorn) Peritonitis  Inflammatory process  Bacterial proliferation  Edema  Exudate  Peritoneal fluid changes  Hypermotility  Paralytic ileus Early Clinical Manifestations  Early s/s mimic original infection or inflammation process  Discomfort  Abdominal Distention/Tenderness  Temperature 37.8-38.3  Increased HR Peritonitis Progression  Anorexia-N/V  Decreased Peristalsis  Paralytic Ileus  Hypotensive  Sepsis/Septic Shock Peritonitis-Labs/Diagnostic  WBC  Hgb/Hct  Electrolytes  K, NA,Cl  Abdominal Xray  Abdominal U/S  CT scan of abdomen  U/S guided aspiration  C & S Studies Peritonitis- Management  Fluids  Electrolytes  Analgesics  Antiemetics  Oxygen therapy  Antibiotics  Surgery  CT-drainage Improvement VS Complications  Improvement  Temperature  HR  Abdomen Assessment  Peristalsis  Flatus  BM  Complications  Temp  HR  Abdomen Assessment  Emergency Sx

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