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Dementia Alzheimers Vascular Lewy body FrontalTemporaldementia atrophytemp lobe SingleMultiple A...

Dementia Alzheimers Vascular Lewy body FrontalTemporaldementia atrophytemp lobe SingleMultiple A 1Ê° s.ieITsIstantiamara Atrophyfrontal temporallobe Infarcts 20401havefamilialcomponents Behavioral most variant common ae F g Damage white matter 3maintypes aphasiasemantic tiffffy.finisitors aphasianonfluent GeneticNonModifable RF family ff faffor Behavioralfrontaltemp EE Biomarker of eimpairmentcognitivefunctions www.n iiif i I fi iiiii tarten Modifable aatieastzofsm.on.fi ii ifitiiiitiiiiii amyloid protein supporting t.fiii amm.non command cognitive mean 6 Neuropsychologicalprofile f ge impairment f Probable Lonsual agnosia f ataxia gggiine i Behaviourapathy Incontinence DefinitebyFTD signssymptomsabove Differing'd ttm.Lohemipanesis Bradykinesia 3 Stages emotionalliability to due damagewhite matter 1 2 reverstsleep Differentiate btw 1 delirium dementia Aging total 5 8years Gradual stable jihadL L 4A 0 Assessment of suspecting Dementia History faffff cognitivetests Emotional function Perceptive function Geriatricdepressionscale MMS dock Montreal Hearing speech vision Functionalassessment Imaging LABtest I let thyroid MRI vitamin tea n9 ADL a s financetransportmedications Barthelindex 1 Management Hollisticview Peggy'Thedementia cognitivesymptoms monkfgnine so 2 feb Pharmacological treatment Montopharmacologial MMSE 17 Cholinesterase Inhibitor.MY fntinaentagonist

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