Lab 7 PDF: Oral Pathology

Acute apical periodontitis. In this early acute lesion, inflammatory cells, mainly neutrophil polymorphonuclear leucocytes, are seen clustered around the apex of a non-vital tooth. The inflammatory cells are spreading around and into bone, and there has only been time for a small amount of bone reso...

Acute apical periodontitis. In this early acute lesion, inflammatory cells, mainly neutrophil polymorphonuclear leucocytes, are seen clustered around the apex of a non-vital tooth. The inflammatory cells are spreading around and into bone, and there has only been time for a small amount of bone resorption to develop. This would be seen radiographically only as slight fuzziness of the apical lamina dura. Chronic periapical abscess. At the apex of the non-vital tooth is an abscess cavity surrounded by a thick fibrous wall densely infiltrated by inflammatory cells, predominantly neutrophils. crystals are formed in the fibrous wall. The epithelium overlying this focus has broken down, and the cholesterol has leaked into the cyst lumen. Elsewhere the lumen is lined by a flattened layer of squamous epithelium. Epithelial proliferation in an apical granuloma. Inflammation induces proliferation of odontogenic epithelium in rests of Malassez in the periodontal ligament. This change may lead to cyst formation Cellulitis. Infection spreading through the tissues is accompanied by a dense infiltrate of neutrophils, macrophages and fibrin exudate, here separating muscle bundles in a facial muscle. The muscle bundles are normally closely packed with minimal space between them