Infection & Infectious Diseases - Fungal & Viral Infections - PDF

 multiple protectivelayers makes harder kill andmake need to penetrate it to fungi can also grow in convonment Unicellular multicellular white multi color microscopic visible  cancer glucocortic...

 multiple protectivelayers makes harder kill andmake need to penetrate it to fungi can also grow in convonment Unicellular multicellular white multi color microscopic visible  cancer glucocorticoid organtransplant HIV AIDS agriculture systemic or topical longduration of use 6 12 months  fungalinfection dit thick cell wall moretoxic than alex  fungus cango systemic non communicable tinea versicolor usually locatedin rings valley fever dont need to know organism name  nyastatin and amphotericin B manytypes w this ending don't need to know  ONLYusedfor polyene systemicinfectie broad spectrum  wallmore cell cause make permeable Leakage frontlents thattheyuse forfunction  N F chilli HA rigor toxic kidney can'tretain kt WBC RBC platelet production relatedto cytokinerelease  gavenormal same before infusion because dehydration can cause kidney to be more sensitive to medicine andcause movekidney toxicity toxin effect on kidney Slow IV 4 6hours  need to know mechanism of action Slowprocess weeksto work G.I.SE putoinhibition hepatotoxicity hepatotoxic fungus have P45d enzymes fortheir metabolism and also affect human liver increase bloodlevel of other meds similar to polyere's azotes inhibitsynthesis of ergosterol in the cell war takes weeksto work instigation hepatoxicity r r  Usually caused dermatophyte byyeast  MTmembrane affected oral lavity infection  does NOTspreadto skill to dithft not much 6 12 months Chulation leasteffective notas effective common in women less problematic no insurance coverage risk factor moisture  MWM hwhnEff YtdmmMmnohh n common polyene Not systemic works locally less SE's does notabsorb in G I carrysgene not 60th aroundthe capsid stomachtw  intracellular pathogen use cell to prodrition BEEEPladinitiet translation  novaccine for HIV virus polio influenza COVID goal Herpes and varicella andvaricella HSV 1 oral HSV2genital shingles  damage DNA of virus by blocking DNApolymerase acyclovir pretendsto be a thymine adds to acyclovir andgetsoheorporatedin Wal DNA blockpita cant replicate thymidine kinase generalfunction adds a phosphate group to thymidine building block of DNA butacyclovir does.no tx Family of ciclovir decreased WBCinblood bone marrow suppression  absorbsthroughskin who recievesandnurses that administermeds  communicable virus NOV March goesthrough airway notessomethrea commton cold butmove symptomatic cough rainy i hand hegume wear mash needsurgical mash POBID reducerish for 5 days induce S S pregnantwomen encymenoususeto release itself frominfected all afterthat unshasspread aroundbody not superffective airborne need 1095 different strains of mthenia everyyear Tamifle reduces viralload helpsuns stuckin infectedcells to this pregnant women are often immunocompromised medication helps decrease risk needle injury IV drugs transmission increases if patient not compliant w medication or unaware are infected they transmission chanceis reduced if patient compliant w HIV meds  retyped.tn an RNAgetsinto whenuval iteration same CPU youngest convert viral orangecolor places where medscan receptyles intervene infightinginside  assertion thticles DNAproduced can byvirus getin nuchal integrase of cell and enzymehelps ans getinto orange CDU DNA boxes are where places intervene Uwstakingover medscan ODY and some of making elements of vini  Seroconversion Clinical CD4 200 latency vapidvirus production and or virus enters plasma and an opportunistic multiply inblood pathogens arise  HIV become activega platave OCCUS 2 4 week after exposure CD4 countstill normal or slightly decreased high viral load non specific symptoms can last years CD4 count gradually decimes overtime 1 and 2 take several years stage to complete alt platane andstopactivity  StephenEnough immunocompromised status causing manifestation of many infections fungus canspread upto smallintestine uncommon types of pneumonia not seen in general population  no care but tx is very successful AIDS stage advanced HIV infection and manifestation of opportunistic injections  whento use ART or HAART stage compliance underlying immone condition 2 3 anti viral viral load 3 anti viral meds more effective Usedfor degree of Uwslevel inblood willthe patient becompliant i Payphone'sde a  KÑA DNA transcribe Emtriva MTDNA UNS components accidentlypicks Vivead up this block cause incomplete DNA strand liverdisease NIV D appetiteloss diarrhea common  Block of the enzyme mostcommon don't need to med remembernames  Efavirenzspecifically can'tuse on pregnant women 6 I.SE  inhibit from assembling the virus first anti viral medication created common w the ored  increased risk of metabolicsyndrome and diabetes end w navir newer gen of HIV med prevent DNA of nwsfrom entering CD4 cells Dolutegravir Tivicay inconclusive HIV fusion inhibitor bindto 68120 viral receptor which prevents nws from attaching to CDU injectible CCR 5 antagonist receptor on CD4 that yous bindsto CCRs receptor on the Astor cardiac toxicity fusion process  one tablet increases patient compliance standard of care 2 NRTI's and I integrase inhibitors  healthy person usually thousands in HIV infection below1000count betoknsidered AIDS highest rate of multiplying and undetectable within the first few weeks and highest rate of transmission after initial exposure non compliance or usingone med at once can increase risk of resistance most anti viral meds safe to take during pregnancy makes vows in active andtransmission ratereduces becomes undetectable zero chance  I a prenatal transmission can happen if compliant w meds exposure in healthcare prophylaxis treatment what is chance of HIV transmission for patient who is NOT ontherapy 25 by any exposure aka like needle inory or sexual intercourse w treatment chance of transmission almost 0 no vaccines get mutations several host targets CDU main elements of immune system and difficult to target w o hurting the host ways of transmission med classes 5 MOC side effects G I common majority of HIV meds have diarrhea SE 2 NRTI and integrate classes are the standard of care rate of contamination is not high w one exposure