What effect does mitochondrial injury have on intracellular levels?
Understand the Problem
The question is asking about the effects of mitochondrial injury on the levels of certain intracellular components or processes. Specifically, it is inquiring into how mitochondrial damage impacts ATP production, glycolysis, reactive oxygen species, and the function of the electron transport chain.
Answer
Increased intracellular calcium, decreased ATP synthesis, oxidative stress.
Mitochondrial injury can result in increased intracellular calcium levels, decreased ATP synthesis, and contribute to oxidative stress.
Answer for screen readers
Mitochondrial injury can result in increased intracellular calcium levels, decreased ATP synthesis, and contribute to oxidative stress.
More Information
Mitochondria are vital for energy production in cells. When injured, they can't produce ATP efficiently, leading to energy deficiencies. High intracellular calcium can disrupt cellular processes and contribute to cell injury.
Tips
A common mistake is failing to consider how mitochondrial injury can create a cascade of effects on overall cellular health.
Sources
- Drug-induced toxicity on mitochondria and lipid metabolism - sciencedirect.com
- General anesthetics cause mitochondrial dysfunction and reduction - journals.plos.org
- Mitochondrial Dysfunction: Pathophysiology and Mitochondria - pmc.ncbi.nlm.nih.gov
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