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Suffering can be studied based on Aetiology, Pathogenesis, and Morphology.
Suffering can be studied based on Aetiology, Pathogenesis, and Morphology.
True
Genetic defects, infectious agents, and hypoxia are mentioned as part of the Aetiology of suffering.
Genetic defects, infectious agents, and hypoxia are mentioned as part of the Aetiology of suffering.
True
Obesity is considered a cause of nutritional imbalance in the context of suffering.
Obesity is considered a cause of nutritional imbalance in the context of suffering.
True
ATP Depletion is mentioned as one of the mechanisms involved in suffering.
ATP Depletion is mentioned as one of the mechanisms involved in suffering.
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Cellular adaptation in the context of suffering can be both physiologic and pathologic.
Cellular adaptation in the context of suffering can be both physiologic and pathologic.
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Membrane damage due to loss of Ca homeostasis is not considered a mechanism related to suffering.
Membrane damage due to loss of Ca homeostasis is not considered a mechanism related to suffering.
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Hypertrophy is an increase in the number of cells in response to excessive hormonal stimulation.
Hypertrophy is an increase in the number of cells in response to excessive hormonal stimulation.
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Metaplasia is a reversible process where one adult cell type is replaced by another.
Metaplasia is a reversible process where one adult cell type is replaced by another.
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Hyperplasia is the replacement of normal ciliated columnar epithelial cells with stratified squamous epithelial cells.
Hyperplasia is the replacement of normal ciliated columnar epithelial cells with stratified squamous epithelial cells.
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Apoptosis is an externally controlled 'suicide' program that leads to extensive disruption of surrounding tissue.
Apoptosis is an externally controlled 'suicide' program that leads to extensive disruption of surrounding tissue.
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Atrophy can occur due to factors like restricted blood supply, aging, and excessive endocrine stimulation.
Atrophy can occur due to factors like restricted blood supply, aging, and excessive endocrine stimulation.
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Hypertrophy can lead to cell death and organellar breakdown.
Hypertrophy can lead to cell death and organellar breakdown.
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