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Questions and Answers
What is the pre patent period for Ostertagia ostertagi?
What is the pre patent period for Ostertagia ostertagi?
What clinical effects are primarily associated with Ostertagia infection in calves and yearlings?
What clinical effects are primarily associated with Ostertagia infection in calves and yearlings?
Which treatment strategy is recommended for controlling Ostertagia in cattle?
Which treatment strategy is recommended for controlling Ostertagia in cattle?
What is a recommended method to prevent the transmission of endoparasites like Ostertagia?
What is a recommended method to prevent the transmission of endoparasites like Ostertagia?
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What is the lifecycle stage of Ostertagia when it becomes infective to cattle?
What is the lifecycle stage of Ostertagia when it becomes infective to cattle?
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What is a primary intermediate host for Davainea proglottina?
What is a primary intermediate host for Davainea proglottina?
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What causative agent is involved in triggering chronic gastritis in horses?
What causative agent is involved in triggering chronic gastritis in horses?
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Which diagnostic method is most effective for detecting Gasterophilus spp. infection?
Which diagnostic method is most effective for detecting Gasterophilus spp. infection?
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What is a common treatment strategy for controlling Gasterophilus spp. infestations?
What is a common treatment strategy for controlling Gasterophilus spp. infestations?
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What lifecycle stage of Habronema spp. is typically ingested by the maggots of flies?
What lifecycle stage of Habronema spp. is typically ingested by the maggots of flies?
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Which of the following is crucial for preventing transmission of Habronema spp. in horses?
Which of the following is crucial for preventing transmission of Habronema spp. in horses?
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What does the larval stage of Gasterophilus spp. develop into after leaving the mouth?
What does the larval stage of Gasterophilus spp. develop into after leaving the mouth?
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Which condition associated with Habronema spp. is particularly challenging to diagnose?
Which condition associated with Habronema spp. is particularly challenging to diagnose?
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What lifecycle stage of Dilepididae tapeworms are considered potentially fatal?
What lifecycle stage of Dilepididae tapeworms are considered potentially fatal?
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Which aspect is NOT a significant characteristic of equine gastrointestinal parasites?
Which aspect is NOT a significant characteristic of equine gastrointestinal parasites?
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What is the initial host for the lifecycle of Taenia taeniaformis?
What is the initial host for the lifecycle of Taenia taeniaformis?
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Which clinical effect is most commonly associated with endoparasitic infections in livestock?
Which clinical effect is most commonly associated with endoparasitic infections in livestock?
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How is Taenia pisiformis typically diagnosed in dogs?
How is Taenia pisiformis typically diagnosed in dogs?
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What is a common treatment strategy for endoparasites in livestock?
What is a common treatment strategy for endoparasites in livestock?
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Which method is least effective in preventing the transmission of endoparasites?
Which method is least effective in preventing the transmission of endoparasites?
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In the lifecycle of Taenia spp., what occurs after the cat consumes the infected rodent?
In the lifecycle of Taenia spp., what occurs after the cat consumes the infected rodent?
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What major health issue is often observed in sheep infected with endoparasites?
What major health issue is often observed in sheep infected with endoparasites?
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What is a key feature of the lifecycle of Taenia pisiformis related to its intermediate host?
What is a key feature of the lifecycle of Taenia pisiformis related to its intermediate host?
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Which clinical sign would most likely indicate a metacercaria infection in livestock?
Which clinical sign would most likely indicate a metacercaria infection in livestock?
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What role do wild life reservoirs play in the lifecycle of endoparasites?
What role do wild life reservoirs play in the lifecycle of endoparasites?
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Heterakis gallinarum eggs embryonate in a week before being picked up by another bird.
Heterakis gallinarum eggs embryonate in a week before being picked up by another bird.
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The primary treatment for Heterakis gallinarum is metronidazole.
The primary treatment for Heterakis gallinarum is metronidazole.
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Capillaria spp. invade different areas of chickens based on the species.
Capillaria spp. invade different areas of chickens based on the species.
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Earthworms serve as definitive hosts for Heterakis gallinarum.
Earthworms serve as definitive hosts for Heterakis gallinarum.
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Chicks can contract Heterakis gallinarum from contaminated food or worms.
Chicks can contract Heterakis gallinarum from contaminated food or worms.
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Each species of Trichostrongylus tenuis can infect various mammals but not birds.
Each species of Trichostrongylus tenuis can infect various mammals but not birds.
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After being ingested by another bird, Heterakis gallinarum mature in the intestine.
After being ingested by another bird, Heterakis gallinarum mature in the intestine.
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Infection with Heterakis gallinarum can lead to symptoms like weight gain and improved digestion.
Infection with Heterakis gallinarum can lead to symptoms like weight gain and improved digestion.
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The prepatent period for T. ovis in sheep is 6-8 weeks.
The prepatent period for T. ovis in sheep is 6-8 weeks.
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Dipylidium caninum primarily infects humans.
Dipylidium caninum primarily infects humans.
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The definitive host for Echinococcus spp. is commonly a cat.
The definitive host for Echinococcus spp. is commonly a cat.
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Toxocara canis can cause respiratory signs when ingested by mice.
Toxocara canis can cause respiratory signs when ingested by mice.
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Cysticercus is the infective stage of Dipylidium caninum.
Cysticercus is the infective stage of Dipylidium caninum.
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Proglottids of Echinococcus spp. can be found in the feces of infected sheep.
Proglottids of Echinococcus spp. can be found in the feces of infected sheep.
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The PPP for T. multiceps in sheep is longer than that for T. ovis.
The PPP for T. multiceps in sheep is longer than that for T. ovis.
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Grains of rice are a visible symptom of Dipylidium caninum infection in dogs.
Grains of rice are a visible symptom of Dipylidium caninum infection in dogs.
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Echinococcus spp. can lead to the formation of hydatid cysts in the liver and lungs.
Echinococcus spp. can lead to the formation of hydatid cysts in the liver and lungs.
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The infective stage of Dipylidium caninum is the egg stage.
The infective stage of Dipylidium caninum is the egg stage.
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Oesophagostomum species can cause severe disease in adult cattle.
Oesophagostomum species can cause severe disease in adult cattle.
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Dark diarrhea in cattle is associated with Oesophagostomum radiatum.
Dark diarrhea in cattle is associated with Oesophagostomum radiatum.
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Diarrhea and dehydration are clinical effects of Oesophagostomum infection.
Diarrhea and dehydration are clinical effects of Oesophagostomum infection.
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Anorexia, anemia, and edema can occur in cattle infected with adult Oesophagostomum.
Anorexia, anemia, and edema can occur in cattle infected with adult Oesophagostomum.
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Oesophagostomum infections commonly lead to sudden death in older cattle.
Oesophagostomum infections commonly lead to sudden death in older cattle.
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Oesophagostomum commonianum primarily affects sheep rather than cattle.
Oesophagostomum commonianum primarily affects sheep rather than cattle.
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The larval stage of Oesophagostomum is not infective to cattle.
The larval stage of Oesophagostomum is not infective to cattle.
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Weight loss and severe disease can result from an Oesophagostomum infection in calves.
Weight loss and severe disease can result from an Oesophagostomum infection in calves.
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Bacterial overgrowth in the abomasum can lead to increased albumin levels in blood.
Bacterial overgrowth in the abomasum can lead to increased albumin levels in blood.
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Increased eosinophils are indicative of a high worm burden in an infected host.
Increased eosinophils are indicative of a high worm burden in an infected host.
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Haemonchus contortus is primarily associated with causing diarrhea in livestock.
Haemonchus contortus is primarily associated with causing diarrhea in livestock.
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The presence of pepsinogen in the blood indicates healthy digestive function.
The presence of pepsinogen in the blood indicates healthy digestive function.
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Bottle jaw is a condition associated with the effects of parasitic infections.
Bottle jaw is a condition associated with the effects of parasitic infections.
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Increased levels of gastrin in blood can be harmful and contribute to anemia.
Increased levels of gastrin in blood can be harmful and contribute to anemia.
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The larval stage of Nematodirus is known for having tough and resistant eggs.
The larval stage of Nematodirus is known for having tough and resistant eggs.
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High levels of IL-4 and IL-5 in response to infections result in positive health outcomes.
High levels of IL-4 and IL-5 in response to infections result in positive health outcomes.
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Necropsy findings of red haemorrhagic surfaces in the abomasum indicate a serious condition.
Necropsy findings of red haemorrhagic surfaces in the abomasum indicate a serious condition.
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The clinical effects of worm infections include improved wool quality in sheep.
The clinical effects of worm infections include improved wool quality in sheep.
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The lifecycle of Taenia taeniaformis involves a rabbit as an intermediate host.
The lifecycle of Taenia taeniaformis involves a rabbit as an intermediate host.
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Metacercariae typically excyst in the duodenum of the host.
Metacercariae typically excyst in the duodenum of the host.
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More cattle than sheep are affected by clinical disease due to trematodes.
More cattle than sheep are affected by clinical disease due to trematodes.
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Anaemia and liver fibrosis are among the clinical signs associated with endoparasitic infections in livestock.
Anaemia and liver fibrosis are among the clinical signs associated with endoparasitic infections in livestock.
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The primary route of transmission for Taenia pisiformis is through direct contact with infected rabbits.
The primary route of transmission for Taenia pisiformis is through direct contact with infected rabbits.
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Wildlife reservoirs do not contribute to the lifecycle of endoparasites.
Wildlife reservoirs do not contribute to the lifecycle of endoparasites.
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The PBS for Taenia pisiformis is typically 6-8 weeks.
The PBS for Taenia pisiformis is typically 6-8 weeks.
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Flukicides are highly effective against all stages of metacercariae.
Flukicides are highly effective against all stages of metacercariae.
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Cats exhibit perianal irritation when infected with Taenia species.
Cats exhibit perianal irritation when infected with Taenia species.
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The ampulla of Vater is the primary site where metacercariae enter the gastrointestinal tract.
The ampulla of Vater is the primary site where metacercariae enter the gastrointestinal tract.
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The larval stage of Davainea proglottina is known as cystercoids.
The larval stage of Davainea proglottina is known as cystercoids.
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Gasterophilus spp. larvae develop in the horse's stomach for 5-7 months before pupating in feces.
Gasterophilus spp. larvae develop in the horse's stomach for 5-7 months before pupating in feces.
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Habronema spp. eggs are typically found in the environment as they are passed in feces.
Habronema spp. eggs are typically found in the environment as they are passed in feces.
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Dilepididae tapeworm infections are typically mild and do not cause serious health issues.
Dilepididae tapeworm infections are typically mild and do not cause serious health issues.
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Gastroscopy is a common method for diagnosing Habronema spp. infections.
Gastroscopy is a common method for diagnosing Habronema spp. infections.
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Equine gastrointestinal parasites can live for several months without causing noticeable symptoms.
Equine gastrointestinal parasites can live for several months without causing noticeable symptoms.
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Larval stage L1 of Habronema spp. is transmitted to horses via direct ingestion of contaminated feed.
Larval stage L1 of Habronema spp. is transmitted to horses via direct ingestion of contaminated feed.
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The initial host for Gasterophilus spp. is a horse, where they will reside in the stomach.
The initial host for Gasterophilus spp. is a horse, where they will reside in the stomach.
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Equine colic can result from chronic gastritis caused by Gasterophilus spp.
Equine colic can result from chronic gastritis caused by Gasterophilus spp.
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Maggots of stable flies do not play a role in the lifecycle of Habronema spp.
Maggots of stable flies do not play a role in the lifecycle of Habronema spp.
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Match the following parasites with their primary location in the gastrointestinal tract:
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Match the following aspects with their respective descriptions:
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Match the following clinical signs with the corresponding species of Oesophagostomum:
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Study Notes
Ruminant GI Parasites
- Pre-patent period: Time between infection and detection of the parasite
- Zoonotic: Can be transmitted between animals and humans
- Control strategies: Frequent drenching of calves, ivermectin for cows, lower stocking density, separate calves and adults, pasture rotation
- Parasite: Ostertagia → location: abomasum; Teladorsagia (Ostertagia) → location: circumcincta = sheep
Nematodirus
- Small intestine: N. battus in sheep and calves, N. fillicolis in sheep, N. Nspathiger in sheep; N. helvetianus in cattle
- Infective L3: PPP 2 weeks
- Eggs: Tough, resist freezing
- Hatching: Stimulated by cold
Haemonchus contortus
- Abomasum: Location of adult worms
- L3 infective: PPP 3 weeks
- Immunity: Increased eosinophils, mast cells, IgA, IgE, IgG, IL-4, IL-5, IL-13
Other Parasites
-
Oesophagostomum:
- O. radiatum in cattle
- O. comumbianum in cow + sheep
- O. dentatum in swine
- O. quadrispin in swine
- **Ascaris suum:**PPP = 35-40 days
-
Fasciola hepatica:
- Lifecycle includes snails
- Pathology/CS: Acute sub-acute and chronic forms depend on number of metacercariae. rate of uptake, Tx, drug resistance.
- Control/tx/dx: Dx: fecal egg count, plasma enzymes. Tx: fence off wet areas, increase soil drainage, control wildlife and anthelmintics
- Fasciola gigantica: Cattle and buffalo, acute fatal in sheep.
- Fasicolides magna: deer
Dicrocoelium dendriticum
- Intermediate host: Ants
- Snail: First intermediate host
- Doesn't penetrate: Liver or gut walls
- Metacercariae: Excyst in duodenum and travel via ampulla of Vater, then enter bile duct
- Clinical disease: Subclinical, mostly affecting sheep
Small Animal GI Parasites
-
Taenia:
- taeniaformi in rodents, cats eat infected rodent. (PPP = 6-8 weeks)
- pisiformis in rabbit, dog eat (PPP = 4-6 weeks)
- multiceps in sheep/cow, dog eat (PPP = 4-6 weeks)
- ovis in sheep/goat, dog eat (PPP=6-8 week)
- serialis in rabbit, dog eat
Dipylidium caninum
- Definitive host: SI of dog and cat
- Infective stage: Cystercoid
Echinococcus spp
- Proglottids: Shed in feces, sheep eat eggs, hyatid cyst form in liver/lung 6-12 months. Dogs may eat sheep
- PPP: 35-55 days
Toxocara canis
- Small mammals and birds: Small mammals and birds frequently a host
- Infected mom: Ingests eggs, kittens infected transmammary. L2 in egg/pratenic host, ingested (PPP=11 weeks)
- Develops in GIT only
Ancylostoma caninum
- L3 ingestion: PPP = 2-3 weeks
Trichuris vulpis
- Dog whip: Adults in large intestine, eggs in feces
- PPP: 2-3 months
Avian GI Parasites
- Ascaridia galli: Direct lifecycle - eggs embryonate in a week then can be picked up by another bird, matures in intestine. Earthworms are a paratenic host.
- Heterakis gallinarum: Chicks can get from worm or contaminated food
- **Trichostrongylus tenuis:**Infective L3, crawl onto moist vegetation
- Eggs: Hatch in environment, L1 becomes L2 and develops into L2
Capillaria and Dilepididae
- Intermediate host: slugs and snails
- Larval stage: Cystercoids
Equine GI Parasites
- Gasterophilus spp: Eggs laid on leg → L1 develop in mouth → L2 pharynx → L3 attaches to gastric mucosa; L3 survive 10-12 months before pupating.
- Habronema spp: Eggs hatch L1 pass into feces and ingested by maggots, L2-L3 develop, L3 migrate to oral part of fly.
- Parascaris equorum: (SI) PPP 6-8 weeks
- Strongylus equinus: L3 invades wall of cecum and colon, becomes L4, passes through visceral peritoneum, then to the liver, then to L5, returns to LI via pancreas
- Strongylus edentatus: L3 through intestinal wall, to live via blood, L4 migrate to peritoneum, adjacent to liver, to L5, then returns to colon and caecum
- Oxyurix equi: Eggs don't survive well outdoors (stable only)
- Strongylus vulgaris: LI, pre-patenting period = 4-5 months, L3, L4 and L5.
Other Parasites (continued)
- Strongyloides westeri: L3, free living adult worms, Indirect lifecycle (mites on veg)
- Anoplocephala perfoliate: Difficult fecal → ELISA, CS: ileal impaction
Protists
- Coccidia: Eimeria spp. or Isospora spp
- Cryptosporidium: Long-lasting, mucoid, sticky, fetid, green diarrhea
- Toxoplasmosis: Cats only definitive host; rodents are host.
- Neosporosis: Dogs only definitive host. Causes abortion in cattle. Vertical transmission.
- Giardia: Lots of species
- Trichomoniasis: Birds and humans/dogs
- Histomoniasis in turkeys and poultry;Necrosis of caecae and liver, Yellow diarrhea, listless, fatal; Carrier bird
- Balantidium: Diarrhea in reptiles;Spironucleosis in birds and hole in head in fish
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Description
This quiz addresses the various gastrointestinal parasites affecting ruminants, focusing on their life cycles, infection periods, and control strategies. Learn about specific parasites like Ostertagia and Haemonchus contortus, and understand their zoonotic implications and management practices.