Hereditary Stone Diseases and Acute Kidney Injury Classifications

Renal calculi can lead to postrenal AKI.

Blood clots in the kidney can result in pre-renal AKI.

Benign prostatic hyperplasia is one of the causes of postrenal AKI.

Malignancies cannot cause obstruction distal to the kidney.

The initiation phase of AKI starts with diuresis.

Oliguria in AKI is characterized by a decrease in serum concentration of substances usually excreted by the kidneys.

The minimum amount of urine needed to rid the body of normal metabolic waste products is 200 mL in 24 hours.

Diuresis period in AKI is marked by a decrease in urine output.

During diuresis in AKI, laboratory values become more unstable.

Even if urinary output returns to normal levels during diuresis, renal function is usually fully recovered.

Which classification system is used by health care professionals to identify kidney injury and improve patient outcomes?

What do the grades of severity in the RIFLE classification system for AKI stand for?

What outcome in the RIFLE system for AKI requires some form of Renal Replacement Therapy (RRT)?

Which type of stones are associated with anatomic and functional urinary tract anomalies as per the text?

Why has the term 'acute kidney injury' replaced 'acute renal failure'?

What is the major cause of prerenal AKI?

Which type of AKI is characterized by actual parenchymal damage to the kidney tubules?

What is the most common type of intrinsic AKI?

Which condition can contribute to Acute Tubular Necrosis (ATN)?

What is a major risk factor for the development of Chronic Kidney Disease (CKD) according to the text?

What is the main cause of postrenal AKI?

Which phase of AKI is characterized by a gradual increase in urine output?

What is the minimum amount of urine needed to rid the body of normal metabolic waste products in 24 hours?

Which condition usually leads to a rise in pressure in the kidney tubules causing a decrease in GFR?

Which substance is NOT usually excreted by the kidneys and accumulates during the oliguria phase of AKI?