Disturbances in Digestion, Absorption, and Elimination
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Questions and Answers

Which of the following physiological processes is directly disrupted in the pathophysiology of gastritis?

  • The production of intrinsic factor for vitamin B12 absorption.
  • The secretion of bile acids into the duodenum.
  • The peristaltic movement of the small intestine.
  • The integrity of the gastric mucosal barrier. (correct)

A patient with a history of chronic NSAID use is diagnosed with gastritis. Which mechanism is most likely contributing to the development of gastritis in this patient?

  • Increased production of protective prostaglandins in the stomach lining.
  • Direct toxic effect of NSAIDs on the liver, leading to altered bile production.
  • Stimulation of gastric acid secretion by NSAIDs.
  • Inhibition of prostaglandin synthesis, leading to decreased gastric mucosal protection. (correct)

A patient presents with epigastric pain, dyspepsia and nausea. The patient reports regular alcohol use. Which condition is most likely responsible for these symptoms?

  • Celiac Disease
  • Gastritis (correct)
  • Ulcerative Colitis
  • Achalasia

A patient is diagnosed with gastritis following a severe burn injury. What is the most likely mechanism contributing to the development of gastritis in this scenario?

<p>Reduced gastric blood flow and increased acid production due to the stress response. (A)</p> Signup and view all the answers

A patient with gastritis is prescribed a medication to reduce gastric acid secretion. Which type of medication is most likely prescribed?

<p>A proton pump inhibitor. (D)</p> Signup and view all the answers

A patient who underwent an esophagogastroduodenoscopy (EGD) was found to have erosion of the gastric mucosa. Which of the following conditions is most likely the cause of this finding?

<p>Gastritis (D)</p> Signup and view all the answers

Following an ingestion of strong acid, a patient develops acute gastritis. Besides immediate supportive care, what is the priority in managing this patient?

<p>Protecting the airway and preventing further damage to the gastrointestinal tract. (A)</p> Signup and view all the answers

A patient presents with signs of upper gastrointestinal bleeding, including melena. Which of the following conditions would most likely be considered as a possible cause?

<p>Gastritis (B)</p> Signup and view all the answers

Which of the following is the least likely component of medical management for acute gastritis?

<p>Encouraging immediate resumption of a regular diet. (C)</p> Signup and view all the answers

A patient with a duodenal ulcer reports experiencing pain. When is this pain most likely to occur?

<p>2-3 hours after eating a meal. (B)</p> Signup and view all the answers

Which of the following factors is least likely to be directly implicated in the development of peptic ulcer disease (PUD)?

<p>Following a diet high in fiber and alkaline foods. (A)</p> Signup and view all the answers

The pathophysiology of peptic ulcer disease (PUD) primarily involves an imbalance between aggressive and protective factors in the gastrointestinal tract. Which of the following best describes this imbalance?

<p>Increased acid/pepsin concentration and decreased resistance of the mucosal barrier (A)</p> Signup and view all the answers

A patient is diagnosed with a gastric ulcer. Based on the typical symptoms of this condition, what characteristics would you expect their pain to exhibit?

<p>Dull, gnawing pain immediately after eating. (B)</p> Signup and view all the answers

A patient with a history of peptic ulcer disease (PUD) presents with symptoms of dumping syndrome following recent surgery. Which dietary modification is least appropriate for managing this condition?

<p>Drinking plenty of fluids with meals to aid digestion. (D)</p> Signup and view all the answers

A nurse is caring for a patient with a peptic ulcer. What assessment finding would be most indicative of a potential gastrointestinal bleed?

<p>The patient's stool is dark and tarry. (A)</p> Signup and view all the answers

A patient who has been taking NSAIDs daily develops a peptic ulcer. Besides discontinuing NSAIDs, what is another appropriate treatment?

<p>Start a proton pump inhibitor (PPI) to reduce stomach acid. (D)</p> Signup and view all the answers

Flashcards

Pyrosis

A burning sensation in the chest caused by stomach acid refluxing into the esophagus.

Gastritis

Inflammation of the gastric mucosa (stomach lining).

Peptic Ulcer Disease (PUD)

Erosion in the GI tract lining, like the stomach, duodenum, or esophagus.

PUD Causes

Most common causes are NSAID use and H. pylori infection.

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PUD Pathophysiology

Acid/pepsin overwhelms mucosal defenses, damaging tissue.

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Gastric Ulcer Symptoms

Pain is immediate after eating, weight loss and vomiting occur.

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Duodenal Ulcer Symptoms

Pain occurs 2-3 hours after eating and improves with food, dark stool.

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Dumping Syndrome

Lie down after eating, avoid fluids with meals and eat frequent small meals.

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Gastritis Causes

Aspirin, NSAIDs, alcohol, H. pylori, ingestion of strong acids/alkalis, major injuries, or stress.

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Gastritis Pathophysiology

Disruption of the mucosal barrier leads to corrosive agents contacting the gastric mucosa, causing inflammation.

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Gastritis Symptom: Pain

Epigastric pain.

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Gastritis Symptom: Dyspepsia

Indigestion.

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Gastritis Symptom: Anorexia

Loss of appetite.

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Gastritis Symptom: Melena

Black, tarry stools (digested blood).

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Gastritis Symptom: Hematochezia

The presence of fresh blood in the stool.

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Study Notes

  • Module 1 discusses responses to altered nutrition and gastrointestinal tract conditions
  • The module covers assessment, analysis/nursing diagnosis, planning, implementation of care, client education, evaluation of outcomes, and reporting/documentation
  • The module also addresses disturbances in ingestion, digestion, absorption, and elimination

Disturbances in Ingestion

  • Achalasia
  • Esophageal spasm
  • Hiatal hernia
  • Diverticulum
  • Gastroesophageal reflux

Disturbances in Digestion

  • Gastritis
  • Peptic ulcer disease

Disturbances in Absorption and Elimination

  • Celiac disease
  • Intestinal obstruction
  • Crohn's disease
  • Ulcerative colitis

Gastritis

  • Inflammation of the gastric mucosa
  • Causes include local irritants like aspirin, NSAIDs, alcohol, H. pylori infection, ingestion of strong acids or bases, major traumatic injuries like burns or infections, and stress

Pathophysiology of Gastritis

  • Disruption of the mucosal barrier allows corrosive agents like hydrochloric acid, pepsin, and alcohol to contact the gastric mucosa, causing inflammation

Symptoms of Gastritis

  • Epigastric pain
  • Dyspepsia (indigestion)
  • Anorexia
  • Hiccups
  • Nausea and vomiting
  • Melena or hematochezia
  • Pyrosis

Medical Management of Gastritis

  • The gastric mucosa is capable of repairing itself
  • Refrain from alcohol and food until symptoms subside
  • Antacids, PPIs, and IV fluids can be administered

Nursing Management of Gastritis

  • Reducing anxiety
  • Promoting optimal nutrition
  • Relieving pain

Peptic Ulcer Disease (PUD)

  • Ulceration formed in the GI tract, dependent on location as gastric, duodenal, or esophageal ulcer
  • Causes include NSAID use, H. pylori infections, smoking and alcohol consumption, and genetics

Pathophysiology of PUD

  • Increased concentration of acid/pepsin or decreased resistance of the protective mucosal barrier
  • Exposure of damaged mucosa to irritants leads to inflammation, injury, subsequent erosion, and formation of ulcers

PUD Symptons

  • Gastric Ulcer: pain immediately follows eating, made worse with food, marked by dull, gnawing pain, weight loss, and coffee ground/beef red discharge
  • Duodenal Ulcer: Pain 2-3 hours after eating, improved with food, with pain waking patient up, normal weight, and dark, tarry stools

Nursing Management for PUD

  • Assess bowel movement and bowel sounds
  • Monitor GI for bleeding (stool/vomitus)
  • Implement strategies for dumping syndrome: lie down 30 minutes after eating, no fluids with meals, small frequent meals
  • Avoid spicy, acidic, and fried foods; limit caffeine and encourage low fiber intake

Treatment for PUD

  • Surgical repair if severe
  • Medications: PPIs, antibiotics, H2 blockers, and antacids

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Description

This module explores responses to altered nutrition and gastrointestinal conditions, covering assessment, diagnosis, planning, and implementation of care. It addresses disturbances in ingestion like achalasia and hiatal hernia, digestion including gastritis and peptic ulcers, and absorption/elimination such as celiac disease and colitis.

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