Anatomy Lecture 6: Optic and Extraocular Motor Pathways

Bilateral lesions in the visual cortex will produce complete blindness

Lesions before the optic chiasm affect the same regions of the fields of both eyes

A homonymous visual field defect is caused by a lesion in one eye

The optic radiations are involved in retrochiasmatic lesions

A lesion in the medial optic radiation/Cuneus results in contralateral homonymous hemianopsia

Bilateral occipital lobe lesions can result in complete blindness

All recti muscles are abductors.

The superior oblique muscle is an elevator.

The trochlear nerve (CN IV) innervates the lateral rectus muscle.

The action of EOMs is limited to one axis of movement.

The superior rectus muscle is a depressor.

All inferior muscles are intorters.

The oculomotor nerve (CN III) innervates the lateral rectus muscle.

The oculomotor nerve nucleus is located at the level of the inferior colliculus of the midbrain.

The trochlear nerve supplies the superior oblique muscle.

The lateral nucleus of the oculomotor complex supplies the superior, medial, and inferior recti, and inferior oblique muscles.

The nucleus of Perlia is responsible for supplying the levator palpebrae superioris.

The Edinger-Westphal nuclei are responsible for vertical eye movement.

The trochlear nerve is responsible for supplying the inferior oblique muscle.

The association nuclei are responsible for convergence.

When the head turns to the left, the eye turns to the left

The frontal eye field is involved in the control of vertical saccades

The pathway for smooth pursuit eye movements involves BA 8

The vergence pathway utilizes the saccadic pathway

The optic radiations are involved in the saccadic pathway

The trochlear nucleus supplies the superior rectus muscle

The posterior commissures are involved in the decussation of some fibers in the saccadic pathway

The RiMLF is involved in the control of vertical gaze

The CN III nuclei supply the lateral rectus muscle