Cerebrovascular Accident PDF
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Uploaded by GleefulWatermelonTourmaline6645
Arellano University
Aira C. Gonzales, PTRP
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This document is a set of notes on cerebrovascular accident, covering topics like motor neuron pathways, blood supply to the brain, stroke syndromes, and other relevant medical details.
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Cerebrovascular Accident I. MOTOR NEURON / CORTICOSPINAL TRACT / PYRAMIDAL TRACT (4) (Loc: anteromedial side of brain stem) - Upper motor neuron (excites and inhibits LMN) - From motor cortex (BA 4, 6, 321) to SC - Corona radiata - group of motor axons; ac...
Cerebrovascular Accident I. MOTOR NEURON / CORTICOSPINAL TRACT / PYRAMIDAL TRACT (4) (Loc: anteromedial side of brain stem) - Upper motor neuron (excites and inhibits LMN) - From motor cortex (BA 4, 6, 321) to SC - Corona radiata - group of motor axons; action of neuromotor descending to lower part of CNS - When have a lesion expect weakness and paralysis - White matter - Internal capsule (post limb) - Lesion: weakness and paralysis - Midbrain - Tecum (CTS will NOT pass) - Cerebral peduncle (where CST descending-medial side) - Tegmentum - Crus cerebri AKA: Basis Pedunculi (base of the MB) - Base = front - DSE - Weber - CN 3 - Medial base of MB - (+) HEMIPLEGIA - Benedikt - CN 3 - Tegmentum of MB - (-) HEMIPLEGIA - Base of Pons - Anterior - Pyramids of medulla - Anterior - Decussate (tumawid) 90% Lat. CTS - not crossed 10% - Lateral CTS 90% - For limbs/appendicular muscle - Ant CTS 10% - For axial *Motor neuron stays in anterior/front/base front Aira C. Gonzales, PTRP Occipital lobe - vision Parietal lobe - sensory Frontal lobe - motor neuron - Level of decussation of Ant CTS is in spinal cord - The Upper motor neuron journey will end in anterior gray matter or horn cell - BELL MAGENDIE LAW ○ “SPAM” Sensory-posterior Anterios-motor ○ “VEM-DAS” ○ Ventral-efferent motor ○ Dorsal-Afferent sensory Lower motor neuron - Anterior horn cell - Start of the journey of Lower motor neuron - Where the UMN and LMN meets - LMN (loc.: Alpha, Beta, & Gamma) - Alpha - extrafusal muscle fiber - Gamma - intrafusal muscle fiber Upper motor neuron lesion Lower motor neuron lesion - Hypertonia - Hypotonia - *Recurrent inhibitory* - Flaccid - Spasticity (velocity dependent - Decrease DTR resistance) *muscle spindle - Increased DTR Disuse atrophy Denervated atrophy - Antigravity muscle - Severe muscle wasting 🌟Pathologic reflexes (Gold standard in diagnosing the UMNL) Spontaneous muscle activity - Fasciculation (can be seen in a - Babinski sign naked eye) and fibrillation (seen in electromyography) - Severe cases manifest as muscle cramping - D/t upregulation phenomenon* (sensitivity to acetylcholine) Pseudobulbar palsy Bulbar palsy Aira C. Gonzales, PTRP - Lesion of corticobulbar tract - Lesion of cranial nerves (CBS) - Tongue fasciculation - Pseudobulbar affect / emotional incontinence (uncontrollable emotion) *LMN is cranial nerves and spinal nerves ○ LMN: cranial nerve (UMN: corticobulbar tract or CBS) ○ LMN: spinal nerve (UMN: CTS) II. BLOOD SUPPLY *ICA manage the circulation in anterior *Vertebral A. Manage the circulation in posterior Internal Carotid A. ○ Ophthalmic A / OA ○ ACA ACOM A. - artery that connect the 2 ACA 🌟 Terminal Only artery that can be “asymptomatic” Medial striate / recurrent A. Of heubner *Striate = watershed areas (no water) - because they are in dulo that’s why their have a little amount of blood ○ MCA Most terminal branch “Largest” MC affected in stroke Small branches that go to innermost of the brain called Lateral striate A. / Lenticulostriate (some books says that this is the Aira C. Gonzales, PTRP terminal branch of MCA) PCA ○ Have 2 branch Central - diencephalon & midbrain Peripheral - supply temporal & occipital lobe Basilar A. ○ AICA - first branch of BA *Blood supply of cerebellum(3) = PICA, AICA, & SUCA (superior cerebellar A.) *PICA - dumaan sa lat. Medulla *Wallenberg syndrome *AICA - lat. Inf. Pons *SUCA - lat sup. Pons *Anterior spinal A. - supply medial medulla *Determine syndrome *PCA nag exit sa midbrain CIRCLE OF WILLIS 1.Ant communicating A. 2.ACA 3.ICA 4.Post communicating A. 5.PCA 6.Vestibular A. III. DEFINITION OF TERMS Stroke / Brain Attack / CVA / Apoplexia ○ Sudden onset of neurologic dysfxn secondary to the loss of blood supply to brain ○ >24hrs 🌟 ○ Non-traumatic Transient Ischemic Attack ○ “MINI STROKE” ○ Temporary 24 hrs - weeks Aira C. Gonzales, PTRP ○ Not used anymore* Deteriorating Stroke ○ “STROKE IN EVOLUTION” ○ Progression of symptoms after admission to hospital ○ EARLY = onset - 3 days ○ LATE = 3 - 7 days ○ F) i. If >85 y/o F>M Family history Previous stroke B.MODIFIABLE RISK FACTORS :) Hypertension (treatable) i. Normal 90 >120 i. 2 - 6 times increase to have stroke Smoking i. 2 - 4 increase to have stroke 1. Light smokers - 40 cid / day Diabetes i. 3-6 time increase to have stroke Diet & obesity i. *Physical activity decreased risk 35% Hypercholesterolemia i. Low density lipoprotein (LHL) 1. Bad cholesterol 2. Normal value = 60 3. Male: 10mm) (microaneurysm, pseudoaneurysm) surgical slipping, surgical px’s w/ long standing HTN coiling ○🌟 Sites: deep penetrating arteries Putamen - MC SAH secondary to saccular ○ “worst headache in my life” ○ Subcortical white matter ○ Thalamus ○ Pons ○🌟 Sites: anterior circulation ○ ICA Ant. Communicating A. - MC ○ Cerebellum - least common ○ MCA Onset: rapidly progressing w/ severe Other cause: arteriovenous headache malformation (AVM) High acute mortality ○ Tangled web of arteries & veins Those who will survive will have rapid by passing capillaries neurologic recovery within 2 - 3 Site: Ant. Circulation months. Onset: 2nd - 3rd decade ○🌟 Triad: Hemorrhage (first will show) ○ Migraine ○ Seizure Subarachnoid hemorrhage (SAH) grading - Used to know the severity of SAH Aira C. Gonzales, PTRP *Hunt and Hess Grading HUNT AND HESS SCALE 1 Awake, mild headache, slight nuchal rigidity 2 Awake, headache, nuchal rigidity, CN involvement 3 Confused & drowsy, mild focal neurologic deficit 4 Stupor, moderate to severe hemiparesis 5 Coma, herniation, posturing VI. STROKE SYNDROMES 🌟 Major blood supply ○ ACA 🌟 Generalization of the BLOOD SUPPLY of the Brain Supply: medial frontal & medial parietal ○ MCA Supply: LAHAT ng lateral except occipital ○ PCA Supply: Medial & inferior, temporal & occipital, SHET & medial midbrain ○ VBA Brain stem & cerebellum Stroke syndromes ICA Aira C. Gonzales, PTRP ○ ○ ○ 🌟 First branch ophthalmic artery (OA) 🌟 OA - amaurosis fugax MCA - aphasia ○ UE > LE ACA ○ Asymptomatic (rare) ○ LE > UE & face ○ (+) Perseverance ○ Hemiplegia & hemiparesthesia ○ Gait Apraxia (new name) - astasia (x - stand) & abasia (x - walk) (old name) AKA: Disconnection apraxia ○ Akinetic mutism - “tahimik/ lack the ability to move or speak” (severe) ○ Abulian - partial response ○ (+) Grasp reflex, groping reflex (dakma) ○ (+) Palmomental reflex ○ (+) Sucking reflex ○ Urinary incontinence Neurogenic bladder ○ Paratonia - force dependent mov’t Px is unaware that his hand moving “alien hand syndrome Types: gegenhalten (resist) & mitgehen (assist) PCA Peripheral Central ○ 🌟 Photo (visual problem) Hemianopsia w/ macular sparing Central post stroke syndrome ○ “thalamic pain syndrome” / determine - roussy after 3-6 ○ Visual agnosia months Dyschromatopsia C/L hemiballismus Prosopagnosia ○ Subthalamus (inability to recognize C/L hemiplegia face) CN 3 palsy Alexia w/o agaraphia ○ Weber syndrome (c/l hemi & (“pure word CN 3 plasy) blindness”) Simultanagnosia (inability to perceive more than one object at a time) Past (memory) ○ Immediate recall ○ Short term memory loss ○ Long term memory loss Aira C. Gonzales, PTRP ○ Topographic amnesia MCA ○ UE & face > LE ○ Dysphagia ○ Hemianopsia Left AKA: Analytical / categorical 🌟 Right AKA: representational hemisphere 🌟 🌟 Aware Aphasias “MGBUWL” ○ Broca - upper stem of MCA Unaware Neglect syndrome Denial of impairment ○ Wernicke - Lower Dressing apraxia (pseudo) ○ Global - Main Constructional apraxia (pseudo) Slow & cautious ○ D/t Visuospatial deficit Apraxias ○ Ideational apraxia ○ Ideomotor apraxia 🌟 Body Image / body scheme deficit Affective agnosia (inability to perceive emotions) Sad / depressed (d/t difficulty in Quick & impulsive expression of positive emotion) Happy (d/t difficulty in expression (+) Perseverance negative emotion) 🌟 Disorganized 🌟 Memory loss (language related) (+) Gerstmann syndrome(BA area Rigidity of thought Poor judgment & poor insights Memory loss (Visuospatial related) 39) Inability to self correct Irritable VBA ○ Brainstem stroke syndromes Brain stem stroke Location Affection Manifestation syndrome Weber Motor Medial basal midbrain 🌟CNCST3 C/L hemiplegia I/L CN 3 palsy Benedikt Sensory Tegmentum of the midbrain tract - CN 3 🌟 - Spinothalamic (STT) C/L hemisensory I/L CN 3 palsy C/L Ataxias - Superior C/L Loss of PVT Cerebellar C/L chorea peduncles - Medial lemniscus Aira C. Gonzales, PTRP Locked in Bilateral basal/base - All in the pons - Severe cases: syndrome Motor of pons & medulla 🌟 oblongata (CN 5 - 12) upward gaze 🌟 - CST & CBT - Okay and CN 1-4 Millard-gubler Motor Lateral pons 🌟 CST CN 6-7 C/L hemiplegia I/L CN 6-7 palsy Wallenberg Lateral medulla - STT C/L *Crossed oblongata - PICA - CN 5 hemisensory hemianesthesia* - Inferior (body) Sensory cerebellar I/L peduncle hemisensory - Sympathetic (face) -🌟tract Vestibular Nucleus - CN I/L Ataxias I/L horner syndrome 8 I/L nystagmus - ambiguus I/L CN 9,10,11 - 9,10,11 🌟 nucleus - CN palsy Determine Medial medulla CST C/L hemiplegia syndrome oblongata - Anterior CN 12 I/L CN 12 palsy Motor spinal A. Medial lemniscus C/L PTV loss *Cerebellar peduncles - Loc: Lateral of brain stem - Connection of the brain stem to cerebellum - 1 pair each area (2) - Spinocerebellar tract - Inferior cerebellar peduncles - Middle cerebellar peduncles - Superior cerebellar peduncles - The only C/L ataxia in cerebellar *Medial lemniscus - Fasciculus gracilis & fasciculus cutaneous (dorsal Aira C. Gonzales, PTRP column) - Proprioception - Two-point discrimination - Vibration - Dorsal column, medial lemniscus system “DCML sys” - Level of Decussation of dorsal column = medulla oblongata SUCA AICA (Inferior Pons) PICA (lateral MO) (superior Pons) Lateral superior Lateral inferior pontine syndrome Wallenberg syndrome pontine syndrome Middle Middle cerebellar peduncles Inferior cerebellar peduncles cerebellar peduncles I/L Ataxias I/L Ataxias I/L Ataxias STT - C/L STT - C/L hemisensory STT - C/L hemisensory hemisensory Sympathetic tract: I/L horner's syndrome Sympathetic tract: I/L Sympathetic horner's syndrome tract: I/L horner's syndrome Medial lemniscus - C/L PVT loss CN 8 🌟 CN 5,7,8 🌟 CN 5,8,9,10,11 🌟 VII. Others A.Aphasias Name Naming Comprehe Fluency Repetition Reading Writing Lesion nsion comprehe site nsion Aira C. Gonzales, PTRP Anomia (MC Poor Good Good Good G G Angular residue) gyrus Conduction Poor Good Good Poor G P Supramarg inal gyrus, arcuate fasciculus (parietal operculum ) Transcortical Poor Good Poor Good G P Medial motor frontal border zone Broca Poor Good Poor Poor G P 3rd frontal (shh)(motor) convolutio n Transcortical Poor Poor Good Good G P Medial sensory parietal border zone Wernicke's Poor Poor Good Poor P P Post sup. (sensory) Temporal gyrus Isolation of Poor Poor Poor Good N/A N/A N/A language (Parang parrot) Global Poor Poor Poor P P P Broca & Wernicke/s area Pure word G P G P G G Heschl’s deafness gyrus Pure word P G P P G G AKA: mutism aphemia Pure word G G G G P G PCA: blindness Alexia w/o agraphia B.Synergy Patterns Spared (hindi kasali sa pagbalik) “FLATS EDEMA” ○ Finger Edema ○ Lats Dorsi ○ Ankle Evertors ○ Teres Major Aira C. Gonzales, PTRP ○ Serratus Anterior FLEXION SYNERGY EXTENSION SYNERGY UE Scapular Retraction/Elevation or Scapular Protraction Hyperextension Shoulder Abduction and ER Shoulder Adduction,* IR Elbow Flexion* Elbow Extension Forearm Supination Forearm Pronation* Wrist and Finger Flexion Wrist and Finger Flexion LE Hip Flexion,* Abduction, ER Hip Extension, Adduction.* IR Knee Flexion Knee Extension* Ankle Dorsiflexion, Inversion Ankle plantarflexion,* Inversion Toe Dorsiflexion Toe Plantar Flexion C.Brunnstrom’s stages of Recovery Stage Tone Movement 1 Flaccidity No movement Aira C. Gonzales, PTRP 2 Spasticity begins to appear Minimal voluntary movement 3 Peak of spasticity, severe Mastered mov’t of synergy 4 Spasticity begins to decline Some movement outside the synergy are mastered (Pron-supi, elbow flexion) (Forward arm raise) (Put hand behind) 5 Spasticity continue to decline More difficult mov’t combination are mastered (Pron-supi, elbow extension) (Horizontal arm raise) (Overhead activity) 6 Disappear Individual jt. mov't are possible 7 Normal Normal D.Functional Walking Categories I. Physiologic walker - Px stands only during exercise or within the parallel bars II. Household walker A.Limited household walker 1. Px's can do household chores with assistance B.Unlimited HHW 1. Px's can do household chores w/o assistance 2. Px can not go out of the house III. Community walker A.Most limited CW 1. Up & down curbs 2. Manage stairs to some degrees B.Least limited CW 1. Up & down stairs completely 2. Can go to uncrowded places C.Unlimited CW 1. Can go to crowded places a) Such as shopping center Aira C. Gonzales, PTRP