Tissue Repair and Anatomy Quiz

<p>Fibroblasts (A)</p> Signup and view all the answers

<p>It provides a mesh of elastin and collagen fibers (C)</p> Signup and view all the answers

<p>Labile tissue (A)</p> Signup and view all the answers

<p>They are the source of fibrous or scar tissue. (B)</p> Signup and view all the answers

<p>The inherent ability of a tissue to repair itself. (C)</p> Signup and view all the answers

<p>Stable tissue (A)</p> Signup and view all the answers

<p>To provide nutrients and growth factors. (D)</p> Signup and view all the answers

<p>It is made up of cells that do not proliferate. (C)</p> Signup and view all the answers

<p>Gastrointestinal epithelium (A)</p> Signup and view all the answers

<p>G0 phase (B)</p> Signup and view all the answers

<p>Neurons (B), Cardiac muscle cells (C), Smooth muscle cells (D)</p> Signup and view all the answers

<p>Cytokine priming and growth factor stimulation (A)</p> Signup and view all the answers

<p>Fibrosis occurs in response to severe tissue injury. (D)</p> Signup and view all the answers

<p>Promote cell survival and proliferation (C)</p> Signup and view all the answers

<p>They are replaced by non-functioning scar tissue. (D)</p> Signup and view all the answers

<p>Residual liver tissue must be structurally intact. (A)</p> Signup and view all the answers

<p>A chronic liver disease (A)</p> Signup and view all the answers

<p>Formation of scar tissue (C)</p> Signup and view all the answers

<p>The capacity for liver cell regeneration results in scar formation. (B)</p> Signup and view all the answers

<p>Proliferative phase (C)</p> Signup and view all the answers

<p>It typically involves well-approximated surgical incisions. (A)</p> Signup and view all the answers

<p>They degrade the extracellular matrix, including collagen. (A)</p> Signup and view all the answers

<p>Fibroblasts, thin-walled blood vessels, and extracellular matrix (D)</p> Signup and view all the answers

<p>It involves a larger wound healing response with more inflammation. (A)</p> Signup and view all the answers

<p>Secretion of cytokines and growth factors (C)</p> Signup and view all the answers

<p>The collagen structure of the scar is organized and matured. (C)</p> Signup and view all the answers

<p>Atherosclerosis of peripheral arteries (C)</p> Signup and view all the answers

<p>Keloid scar (B)</p> Signup and view all the answers

<p>Wound dehiscence (C)</p> Signup and view all the answers

<p>They arise from chronic venous hypertension. (C)</p> Signup and view all the answers

<p>More intense inflammatory response (D)</p> Signup and view all the answers

<p>3 months (D)</p> Signup and view all the answers

<p>Exuberant granulation tissue (C)</p> Signup and view all the answers

<p>When the structural framework is damaged (A)</p> Signup and view all the answers

<p>Wound dehiscence (B)</p> Signup and view all the answers

<p>Vitamin C deficiency (C)</p> Signup and view all the answers

<p>Exuberant proliferation of fibroblasts (A)</p> Signup and view all the answers

<p>Diabetic ulcers (D)</p> Signup and view all the answers

<p>Increased connective tissue deposition (D)</p> Signup and view all the answers

<p>Cultural background (B)</p> Signup and view all the answers

<p>Healing by secondary intention (C)</p> Signup and view all the answers

<p>Fibrosis (C)</p> Signup and view all the answers

Tissue repair process where surviving cells multiply to replace damaged tissue.

Tissue repair using fibrous tissue to replace injured tissue when regeneration is not enough, creating scars.

Tissue providing support, structure, and strength to other tissues.

Fibrous proteins and ground substance, the non-cellular part of connective tissue that provides a framework for cells.

The restoration of tissue architecture and function after injury.

Excess deposition of connective tissue, often referred to as scar formation.

The process of restoring damaged tissue using a combination of regeneration and connective tissue deposition.

A tissue's ability to repair itself through cell multiplication.

The process of cell growth and division, including the resting phase (G0) and active phases (G1, S, G2, M).

Tissue with a continuous capacity for cell renewal, constantly dividing and never entering G0.

Tissue with limited capacity for cell renewal, normally resting in G0 but able to enter G1 after injury.

Tissue that can't regenerate, cells are permanently in G0.

Specialized cells that can differentiate into various cell types, contributing to tissue repair.

Unlike most stable tissues, the liver has a remarkable ability to regenerate itself. It can even rebuild itself after up to 90% is removed. This is due to a combination of growth factors and cell signaling.

Fibrous connective tissue that forms in response to injury in permanent tissues. This tissue is less functional than the original tissue, leading to reduced organ function.

Chemical messengers that promote cell proliferation, survival, migration, and differentiation. They play a critical role in regeneration.

Initial phase of liver regeneration where Kupffer cells release cytokines, such as IL-6, preparing remaining hepatocytes to respond to growth signals.

The phase where growth factors, such as HGF and TGF-α, act on primed hepatocytes to stimulate cell division and regeneration.

Liver cells can regenerate after injury, but extensive damage beyond the supporting framework leads to scar formation, not just regeneration.

Immature connective tissue forming during healing. It's a mix of fibroblasts, new blood vessels, and extracellular matrix.

Process where the scar tissue is reshaped and refined by enzymes that break down and rebuild the extracellular matrix.

Wound healing where edges are closed and regeneration dominates over scar formation. This results in a small, neat scar.

Wound healing with open edges that require more connective tissue deposition, leading to a larger scar.

Wound healing where initial closure is delayed, allowing for inflammation to resolve before closing, leading to an intermediate scar.

The process where the edges of a wound are drawn together by specialized cells, reducing wound size.

Matrix metalloproteinases break down the extracellular matrix during remodeling, allowing for scar tissue reshaping and refinement.

A wound caused by poor oxygen delivery due to chronic venous hypertension.

A wound caused by poor oxygen delivery due to atherosclerosis of peripheral arteries.

A wound caused by a combination of ischemia, neuropathy, and metabolic abnormalities.

A wound caused by mechanical pressure causing local ischemia.

A raised scar that stays within the boundaries of the original wound.

A raised scar that grows beyond the boundaries of the original wound.

The separation of wound edges, often due to inadequate healing.

Healed skin is never as strong as the original tissue. Sutured wounds regain strength gradually, reaching 70-80% of normal strength after 3 months.

Complications of wound healing where the repair process fails to form the necessary components.

Infection prolongs inflammation and increases local injury, hindering healing.

Nutritional deficiencies, such as lack of vitamin C, can impair collagen synthesis and healing.

Poor blood supply reduces oxygen, nutrients, and growth factors needed for healing.