7 Questions
Which neurotransmitter is the primary excitatory neurotransmitter?
Glutamate
What can happen if there is an excessive amount of glutamate in the brain?
Seizures
Which receptors are responsible for controlling the sodium channel in response to glutamate?
AMPA receptors
Which mechanism is responsible for the strengthening of synaptic connections?
Long-Term Potentiation
What happens when there is a high amount of glutamate released into the synaptic cleft?
NMDA channels open
What is the role of NMDA receptors in increasing synaptic connection strength?
They build new AMPA receptors
What is the purpose of Long-Term Depression?
To weaken synaptic strength
Study Notes
Neurotransmitters
- Glutamate is the primary excitatory neurotransmitter.
Glutamate's Effects
- Excessive amount of glutamate in the brain can lead to excitotoxicity, causing damage to neurons.
Glutamate Receptors
- NMDA (N-Methyl-D-Aspartate) and AMPA (Alpha-Amino-3-Hydroxy-5-Methyl-4-Isoxazole Propionic Acid) receptors are responsible for controlling the sodium channel in response to glutamate.
Strengthening Synaptic Connections
- Long-Term Potentiation (LTP) is the mechanism responsible for the strengthening of synaptic connections.
Glutamate Release
- High amount of glutamate released into the synaptic cleft can activate NMDA receptors, leading to an influx of calcium ions, which in turn strengthens synaptic connections.
NMDA Receptors' Role
- NMDA receptors play a crucial role in increasing synaptic connection strength by allowing an influx of calcium ions, leading to LTP.
Long-Term Depression
- The purpose of Long-Term Depression (LTD) is to weaken synaptic connections, counterbalancing the strengthening effects of LTP.
Test your knowledge on glutamate, long-term potentiation (LTP), and long-term depression (LTD) with this quiz! Explore the role of glutamate as the primary excitatory neurotransmitter and learn about the effects of excess glutamate and GABA. Discover the different glutamate receptors and their functions. #neurotransmitters
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