Termination of Anesthesia

The termination of anesthesia is primarily caused by elimination of the drug from the body.

Anesthesia termination can occur due to the drug redistributing from nervous tissue to muscles, viscera, and adipose tissue.

Redistribution of the drug during anesthesia termination is limited only to nervous tissue.

The redistribution process during anesthesia termination involves moving the drug from muscles to nervous tissue.

Muscles play no role in the termination of anesthesia through drug redistribution.

Saturation of muscle tissue can lead to prolongation of anesthesia.

Short-term administration can prevent the slowing of the redistribution phase.

Prolonged administration may result in acceleration of the redistribution phase.

Saturation of adipose tissue is a potential consequence of prolonged administration.

Redistribution phase occurs more rapidly with prolonged administration of anesthesia.

Fospropofol is an oil-soluble derivative that is less painful.

Fospropofol is slowly converted to propofol in the body.

Fospropofol is more painful than propofol when administered.

Fospropofol is a water-soluble derivative that is rapidly metabolized in the body.

Propofol is derived from fospropofol.

Thiopental is a hydrophilic drug.

After IV injection, thiopental causes unconsciousness within about 10 seconds.

The duration of unconsciousness caused by thiopental is around 15-20 minutes.

Thiopental redistributes to fatty tissues after causing unconsciousness.

Thiopental is a water-soluble drug.

Adverse effects of anesthesia are more pronounced in children compared to adults.

The termination of anesthesia is primarily caused by the redistribution of the drug from nervous tissue to other body tissues.

During the termination of anesthesia, the drug moves from muscles to nervous tissue.

The redistribution of the drug during anesthesia termination is limited to only a few body tissues.

The drug's elimination from the body plays no role in the termination of anesthesia.