3.4 Quiz 1

Which reflex prevents overexpansion of the lungs by inhibiting respiratory muscles?

What is the primary influence of visceral pain on respiration?

Which center in the medulla and pons is responsible for forced exhalation?

Which body temperature change stimulates ventilation for heat elimination?

What effect does intense stimulation have on the respiratory rate?

Which of the following is NOT true about the main respiratory control neurons?

What is the primary reason behind an increase in ventilation during exercise?

Which condition is most likely to stimulate peripheral chemoreceptors and lead to increased ventilation?

What is a characteristic feature of hyperpnea during exercise?

How do peripheral chemoreceptors respond to an increase in non-volatile acids in arterial blood?

Which component is responsible for generating the spontaneous rhythmicity of breathing?

How do alterations in carbon dioxide concentration affect the control of breathing?

Which nuclei in the medulla are responsible for controlling inspiration and expiration?

What is the main role of the pontine respiratory centers in breathing control?

Which respiratory pattern involves cycles of gradual increase in tidal volume followed by a gradual decrease in tidal volume and then a period of apnea?

How does the brain cortex influence the normal pattern of inspiration and expiration temporarily?

What type of breathing pattern is characterized by maximal brief inspiratory efforts separated by long periods of expiration, typically seen in severe anoxia and cardiac arrest?

In what condition would you most likely observe Kussmaul breathing, characterized by extremely deep and rapid breaths?

Which respiratory pattern involves prolonged inspirations separated by brief expirations, a pattern rarely observed in humans?

What type of breathing involves slow, deep inspirations caused by an interruption of vagal input to the brainstem?

Which center, located in the caudal pons, promotes inspiration by stimulating the Dorsal Respiratory Group?

What effect does an increase in pneumotaxic output have on respiration?

What happens to breathing rate and depth when the vagus nerve is cut above the pons?

Which conclusion can be drawn from a mid-pons transection with vagus nerve intact?

In a spinomedullary death scenario, what is the likely impact on initiating respiration?

Which area of the brain contains receptors responsible for the effect of intravenous lactic acid on increasing ventilation?

What physiological event typically occurs first when affecting respiration in vivo among decreased CSF pH, increased arterial PCO2, increased CSF PCO2, stimulation of medullary chemoreceptors, and increased alveolar PCO2?

When would spontaneous respiration cease according to the options provided?

If a drug that stimulates the carotid bodies is injected, what change would be expected in arterial blood?

During quiet respiration, what happens when regular bursts of impulses are sent to inspiratory muscles?

Which receptors are stimulated by lung expansion and prevent overexpansion of the lungs during forced breathing?

Where are irritant receptors located and what stimulates them?

Which receptors are described as 'juxta-capillary' and respond quickly to irritants and pulmonary vascular congestion?

Which receptors, when stimulated, can result in rapid, shallow breathing?

What is the main function of irritant receptors when stimulated?

Which type of respiratory receptor is involved in preventing overexpansion of the lungs?

Which receptor type is associated with feelings of dyspnea when activated?

Which nerve pathway is involved in transmitting signals from irritant receptors?

What is the role of J receptors in the respiratory system?

Which receptor type is involved in inducing bronchoconstriction upon stimulation?