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Questions and Answers
What vascular disorder is associated with hypertension and is characterized by 'onion skin,' concentric, laminated thickening of arteriolar walls and luminal narrowing?
What vascular disorder is associated with hypertension and is characterized by 'onion skin,' concentric, laminated thickening of arteriolar walls and luminal narrowing?
- Dissecting aneurysm
- Hyaline arteriolosclerosis (correct)
- Atherosclerosis
- Malignant hypertension
What is the consequence of increased demands placed on the heart by hypertension?
What is the consequence of increased demands placed on the heart by hypertension?
- Ventricular atrophy
- Cardiac dilation
- Myocardial ischemia
- Ventricular hypertrophy (correct)
What percentage of cerebrovascular disease is attributed to inadequate management of hypertension?
What percentage of cerebrovascular disease is attributed to inadequate management of hypertension?
- 49%
- 62% (correct)
- 32%
- 75%
What type of changes are particularly prominent in the kidney in malignant hypertension?
What type of changes are particularly prominent in the kidney in malignant hypertension?
What adaptive response involves adding sarcomeres in parallel to the original sarcomeres in myocardial hypertrophy due to pressure overload?
What adaptive response involves adding sarcomeres in parallel to the original sarcomeres in myocardial hypertrophy due to pressure overload?
What is the most common physiologically definable cause of secondary hypertension?
What is the most common physiologically definable cause of secondary hypertension?
What are the pathological consequences of raised arterial pressure on large arterial blood vessels?
What are the pathological consequences of raised arterial pressure on large arterial blood vessels?
What characterizes hyaline arteriolosclerosis associated with benign hypertension?
What characterizes hyaline arteriolosclerosis associated with benign hypertension?
How does hyaline arteriolosclerosis contribute to nephrosclerosis in the kidneys?
How does hyaline arteriolosclerosis contribute to nephrosclerosis in the kidneys?
What are the selective responses to hypertension in vascular walls?
What are the selective responses to hypertension in vascular walls?
What is the mechanism primarily associated with all forms of established hypertension?
What is the mechanism primarily associated with all forms of established hypertension?
Which factor is not a cause of essential (idiopathic) hypertension?
Which factor is not a cause of essential (idiopathic) hypertension?
What are systolic pressures between 140 and 159 mm Hg suggestive of?
What are systolic pressures between 140 and 159 mm Hg suggestive of?
In the group with uncontrolled hypertension and high blood pressures, what is the immediate action recommended?
In the group with uncontrolled hypertension and high blood pressures, what is the immediate action recommended?
Which factor is vital to consider in patients with systolic pressures between 140 and 159 mm Hg or diastolic pressures between 90 and 99 mm Hg before deciding on treatment?
Which factor is vital to consider in patients with systolic pressures between 140 and 159 mm Hg or diastolic pressures between 90 and 99 mm Hg before deciding on treatment?
What is the pathological consequence of raised arterial pressure in systemic hypertensive heart disease?
What is the pathological consequence of raised arterial pressure in systemic hypertensive heart disease?
Which condition is characterized by marked thickening of the left ventricular wall leading to reduced lumen size?
Which condition is characterized by marked thickening of the left ventricular wall leading to reduced lumen size?
What is the primary cause of left atrial dilation in systemic hypertensive heart disease?
What is the primary cause of left atrial dilation in systemic hypertensive heart disease?
In chronic cor pulmonale, what is primarily observed in the right ventricle?
In chronic cor pulmonale, what is primarily observed in the right ventricle?
What is the consequence of increased left ventricular wall thickness over time in systemic hypertensive heart disease?
What is the consequence of increased left ventricular wall thickness over time in systemic hypertensive heart disease?
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