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Neuromuscular Effects of Pesticides on Insects

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Neuromuscular Effects of Pesticides on Insects

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Questions and Answers

Which mechanism is shared by DDT and permethrin in disrupting neuromuscular functioning?

Blocking neurotransmitter release

Enhancing the breakdown of acetylcholine

Inhibiting acetylcholinesterase activity

Causing Na+ ion channels to open (correct)

What is the consequence of inhibiting acetylcholinesterase activity, as described for carbamates?

Increased neurotransmitter production

Continuous stimulation of cells (correct)

Enhanced muscle relaxation

Reduced nerve cell sensitivity

How do DDT and permethrin differ from carbamates in disrupting neuromuscular functioning?

By increasing neurotransmitter breakdown

By enhancing enzyme activity

By blocking nerve impulses

By inhibiting ion channels (correct)

What effect does continuous stimulation of cells, caused by carbamates, have on the neuromuscular system?

<p>Over-excitation</p> Signup and view all the answers

Why can a high dose of carbamates lead to fatal convulsions?

<p>Resulting in continuous cell stimulation</p> Signup and view all the answers

Neuromuscular Effects of Pesticides on Insects

Choose a study mode

Podcast

Questions and Answers

Which mechanism is shared by DDT and permethrin in disrupting neuromuscular functioning?

What is the consequence of inhibiting acetylcholinesterase activity, as described for carbamates?

How do DDT and permethrin differ from carbamates in disrupting neuromuscular functioning?

What effect does continuous stimulation of cells, caused by carbamates, have on the neuromuscular system?

Why can a high dose of carbamates lead to fatal convulsions?

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