Nephropathy Patterns and Immune Complexes

Which type of cells cause glomerular injury and are involved in the progression of some glomerulonephritides?

What is the result of lymphocyte activation on exposure to antigen?

What is the effect of arachidonic acid metabolites on GFR?

What is the role of macrophages and T lymphocytes in glomerular injury?

What is the term previously used to describe peripheral layers membranoproliferative glomerulonephritis?

What is the result of complement activation in glomerular injury?

What is the role of resident glomerular cells in glomerular injury?

What is the term used to describe the activation of the alternative pathway in glomerular injury?

What is the role of coagulation proteins, such as fibrin, in glomerular disease?

What is the result of the loss of normal slit diaphragms in glomerular injury?

Which of the following is involved in the maintenance of endothelial integrity and regulation of capillary permeability?

What is the characteristic pattern of staining in anti-GBM antibody–induced glomerulonephritis?

What is the common feature of multiple types of glomerular injury, including FSGS and diabetic nephropathy?

What is the percentage of cases of human glomerulonephritis caused by anti-GBM antibody–induced glomerulonephritis?

Which cytokines are involved in inflammatory cell adhesion and recruitment?

What is the characteristic feature of membranous nephropathy?

What is the result of podocyte injury in glomerular diseases?

What is the term for the formation of immune complexes locally by antibodies reacting with intrinsic tissue antigens?

Which of the following is involved in the proliferation of mesangial cells?

What is the term for diseases with disparate etiologies whose principal manifestation is injury to podocytes?

What is the term for the deposition of immune complexes in the glomerulus from the circulation?

What is the cause of necrotizing and crescentic glomerular damage and clinical syndrome of RPGN?

What is the characteristic feature of global glomerulonephritis?

What is the mechanism underlying most forms of primary glomerulopathy and many secondary glomerular disorders?

What is the primary location of the electron-dense deposits in the EM findings of this disease?

What is the characteristic of the red cells in the urine of patients with this disease?

What is the usual time frame for the onset of symptoms after a sore throat?

What is the term for the electron-dense deposits seen in the EM findings of this disease?

What is the percentage of patients who recover renal function with conservative therapy?

What is the typical color of the urine in patients with this disease?

What is the percentage of patients who have c-ANCA or p-ANCA in their serum?

What is the term for the granular deposits seen in the IFM findings of this disease?

What is the trigger for crescent formation in glomerulonephritis?

What is the characteristic feature of glomerulonephritis in Goodpasture syndrome?

What is the effect of heavy proteinuria and increased renal catabolism of filtered albumin?

What is the cause of edema in nephrotic syndrome?

What is the characteristic feature of proteinuria in nephrotic syndrome?

What is the effect of loss of immunoglobulins in nephrotic syndrome?

What is the cause of membranous nephropathy?

What is the effect of C5b-C9 in nephrotic syndrome?