Flaccid Dysarthria Etiology Quiz

What causes hypernasality?

Which condition may result in challenges controlling the soft palate and maintaining normal nasality?

What is a common characteristic of prosody in individuals with weakened laryngeal muscles?

Which component of speech is affected by inadequate subglottic air pressure in individuals with flaccid dysarthria?

What may occur if the cricothyroid muscle is weakened due to damage to the superior laryngeal branch of the vagus nerve?

Which evaluation task would be most useful for assessing speech mechanisms fatigue in individuals with flaccid dysarthria?

What type of dysarthria is caused by damage to the lower motor neurons?

Which condition can cause a person to have difficulty in elevating their jaw for speech articulation?

What is the primary symptom of Myasthenia Gravis that distinguishes it from other conditions?

Which type of nerve damage can lead to incomplete adduction of the vocal folds?

Which condition results in a paralysis that affects lower motor neurons?

What is one characteristic voice quality associated with adductor damage in flaccid dysarthria?

Which pathology can cause difficulties in maintaining strength of muscle contractions over time?

Which symptom often necessitates a stress test for diagnosis?

'Intense' hypernasality and 'breathy' voice quality together indicate a strong possibility of which type of dysarthria?

Hypernasality is caused by the lack of air in the nasal cavity when it is needed.

Flaccid dysarthria is associated with challenges in controlling the soft palate and maintaining normal nasality.

Myasthenia gravis is not linked to flaccid dysarthria.

Oromotor exercises are considered valuable for treating dysarthria.

Hypernasality can be assessed by listening for nasal airflow on any sound other than nasals.

Weakened laryngeal muscles can result in variations in pitch and loudness due to inadequate tension and stretching of the vocal folds.

Flaccid dysarthria is caused by impairment of the UMNs of the cranial or spinal nerves.

Guillain-Barre Syndrome affects sensory neurons more than motor neurons.

Poli tumors are among the etiologies of flaccid dysarthria.

Myasthenia Gravis primarily affects the connection between the LMN and muscle tissue.

Flaccid dysarthria can result in slow-labored articulation due to hypernasal resonance.

Damage to the recurrent laryngeal nerve can cause breathy vocal quality in flaccid dysarthria.

Flaccid dysarthria can impact articulation by affecting vowels requiring lip rounding.

Difficulty in elevating the jaw for speech articulation can be caused by damage to the trigeminal nerve.

Muscular Dystrophy is a form of flaccid dysarthria that results in rapid fatigue of muscular contractions.

Speech characteristics of flaccid dysarthria may include precise articulation and increased loudness.

Match the etiology with the condition that can cause flaccid dysarthria:

Match the symptom with the corresponding speech characteristic in individuals with flaccid dysarthria:

Match the nerve damage with the resulting impact on articulation in flaccid dysarthria:

Match the voice quality with the corresponding nerve damage in flaccid dysarthria:

Match the speech characteristic with the corresponding description:

Match the evaluation task with its purpose for individuals with flaccid dysarthria:

Match the muscle impairment with the resulting speech characteristic:

Match the component affected by inadequate subglottic air pressure with the corresponding speech outcome: