Dental Anomalies Quiz

All teeth in the mouth are smaller than normal, often seen in individuals with pituitary dwarfism.

A single tooth is significantly smaller than its counterparts, with an altered shape.

All teeth in the mouth appear larger than usual, often associated with pituitary gigantism.

One tooth is significantly larger than its counterparts, commonly seen with wisdom teeth.

Two teeth develop from a single enamel organ, resulting in two crowns sharing a single root canal.

Two developing tooth germs fuse together, creating one larger tooth with a shared root canal.

Adjacent teeth are joined by cementum, forming a single tooth.

A condition where teeth erupt outside of their normal position due to insufficient space.

Physiologic wearing of teeth due to chewing, making teeth appear worn down.

Abnormal tooth wear caused by habits, like using abrasive substances, resulting in rough and worn-down teeth.

Loss of tooth structure due to acids, not bacteria, making teeth appear worn and translucent.

Wedge-shaped defects at the gum line caused by tooth flexing under heavy loads.

Complete absence of all teeth.

Absence of one or a few teeth.

A tooth that's present but doesn't erupt, making it seem absent.

Teeth that have fallen out or been extracted.

An unusual curving or angulation of tooth roots, making extractions and root canal treatments challenging.

A rare condition where a tooth develops with an extra, inward fold, forming a 'tooth within a tooth'.

An uncommon tooth anomaly with an extra cusp or bump located in the middle of the chewing surface.

A condition where teeth have an enlarged body and pulp chamber, with the root portion abnormally short.

The presence of extra teeth in the mouth, often caused by the formation of an additional tooth germ.

Small, enamel-like deposits found on the roots of teeth, usually at the point where the roots split.

The most common site for supernumerary teeth is the anterior midline, where an extra tooth called a 'mesiodens' is found.

Extra teeth are often found in canines, premolars, and molars, especially the third molars.

A condition where the enamel of a tooth is thinner than normal, often due to disturbances during enamel formation.

A condition where the enamel is present in normal amounts but is softer or weaker than normal, due to insufficient mineral deposition.

The developmental stage of a tooth when enamel is formed is crucial for its health. If external factors disrupt the ameloblasts during this period, enamel defects can occur.

A specific type of enamel hypoplasia characterized by discoloration, irregularities, and pitting of the enamel, often affecting a permanent tooth due to infection in the overlying deciduous tooth.

Systemic factors that influence tooth development must act after birth and during early childhood (before age six) to impact the developing enamel.

Infectious diseases during childhood can significantly influence enamel formation, potentially leading to defects such as hypoplasia or hypocalcification.

A specific type of enamel defect characterized by a narrowed incisal edge and central notch, often seen in children with congenital syphilis.

Nutritional deficiencies, birth trauma, fluorosis, and idiopathic factors can also contribute to enamel defects like hypoplasia and hypocalcification.

A condition where the enamel is abnormally thin or absent due to incomplete enamel formation during tooth development.

Characterized by enamel that is present but is soft and easily chipped, often appearing opaque, yellow, or brown.

A group of inherited disorders affecting the formation of enamel in both sets of teeth. It can manifest as hypoplastic (thin enamel), hypocalcified (soft enamel), or hypermaturation (abnormal enamel structure).

An inherited condition affecting the dentin of both baby and adult teeth. It is characterized by translucent, opalescent teeth that may appear yellow to gray. Affected individuals are prone to enamel fractures, and the crowns may have a constricted appearance.

A rare inherited condition that affects the dentin. Type I, or radicular type, affects both sets of teeth and is characterized by normal-colored teeth with periapical lesions. Type II, or coronal type, affects only the permanent teeth and is characterized by opalescent primary teeth with a normal-colored permanent dentition, large coronal pulps, and few periapical lesions.

A condition caused by excessive fluoride intake during tooth development, resulting in enamel defects. The severity of fluorosis can range from mild white spots to severe pitting and discoloration.

A term used to describe teeth with a lobulated or crenated (irregularly scalloped) occlusal surface.

A type of dentinogenesis imperfecta that is associated with osteogenesis imperfecta (brittle bone disease).

A dental anomaly affecting enamel, dentine, and cementum, resulting in short roots, open apical foramina, and enlarged pulp chambers. The enamel and dentine are poorly mineralized, leading to 'ghost teeth'. Permanent teeth are more affected than primary, with maxillary anterior teeth being the most vulnerable. Eruption is delayed.

Common with aging, pulp calcifications can be microscopic or large enough to be visualized radiographically. These calcifications can be linear or nodular (pulp stones). True denticles are composed of dentine, while false denticles represent foci of dystrophic calcification.

Resorption of dentine on the pulpal walls. This can occur due to pulpal injury or without a clear trigger. It is caused by the activation of osteoclasts or dentinoclasts on the internal surfaces of the root or crown. Resorption lacunae are visible, and reversal lines indicate attempts at repair. Perforation can occur, and teeth may appear pink.

Resorption of dentine on the external surfaces of the tooth. This can be triggered by various factors like inflammation, cysts, tumors, trauma, or reimplantation. Chemical mediators, increased vascularity, and pressure contribute to this process. It typically occurs apical to the cementoenamel junction (CEJ).

Stains that are caused by external factors and can be removed by abrasive methods. These stains can originate from diet or bacterial byproducts.

Stains that arise from systemic factors during tooth development and cannot be removed by abrasive methods. An example is tetracycline staining, which appears yellow, gray, or brown.

In type I, crowns appear normal but may be lost prematurely due to short roots or periapical lesions. Radiographically, type I roots are short with obliterated pulps. In type II, pulp chambers appear enlarged. Microscopically, deeper layers of dentine exhibit atypical tubular patterns.

The deeper layer of dentine in Regional Odontodysplasia shows abnormal tubular patterns, characterized by irregular organization and a lack of proper structure. Think of the dentine tubules being misaligned and disorganized.