Podcast
Questions and Answers
ACE inhibitors and ARBs are not recommended in CKD due to their potential to?
ACE inhibitors and ARBs are not recommended in CKD due to their potential to?
- Increase GFR (correct)
- Promote sclerosis of the glomerular basement membrane
- Decrease proteinuria
- Raise glomerular hydrostatic pressure
What is the effect of dilation of the efferent arteriole?
What is the effect of dilation of the efferent arteriole?
- Increased glomerular hydrostatic pressure
- Increased glomerular filtration rate
- Decreased proteinuria
- Decreased glomerular hydrostatic pressure (correct)
Which statement best describes the impact of ACE inhibitors and ARBs on GFR?
Which statement best describes the impact of ACE inhibitors and ARBs on GFR?
- They reduce GFR by constricting the afferent arteriole
- They increase GFR by dilating the efferent arteriole
- They have no effect on GFR
- They decrease GFR by constricting the efferent arteriole (correct)
What role do ACE inhibitors play in preventing sclerosis of the glomerular basement membrane?
What role do ACE inhibitors play in preventing sclerosis of the glomerular basement membrane?
In CKD, ACE inhibitors are indicated for which condition?
In CKD, ACE inhibitors are indicated for which condition?
What is the mechanism by which ACE inhibitors can lead to hypotension in patients with chronic kidney disease (CKD)?
What is the mechanism by which ACE inhibitors can lead to hypotension in patients with chronic kidney disease (CKD)?
Which of the following is a mechanism by which ACE inhibitors may be nephroprotective in patients with CKD?
Which of the following is a mechanism by which ACE inhibitors may be nephroprotective in patients with CKD?
Which of the following is a key reason why ACE inhibitors (or ARBs) are considered first-line anti-hypertensive agents in patients with CKD?
Which of the following is a key reason why ACE inhibitors (or ARBs) are considered first-line anti-hypertensive agents in patients with CKD?
Which of the following is a potential adverse effect of ACE inhibitors in patients with CKD?
Which of the following is a potential adverse effect of ACE inhibitors in patients with CKD?
How do ARBs differ from ACE inhibitors in their mechanism of action in patients with CKD?
How do ARBs differ from ACE inhibitors in their mechanism of action in patients with CKD?
What is the primary rationale for using ACE inhibitors (or ARBs) as first-line anti-hypertensive agents in patients with CKD?
What is the primary rationale for using ACE inhibitors (or ARBs) as first-line anti-hypertensive agents in patients with CKD?
Which of the following is a common side effect of Angiotensin-receptor blockers (ARBs)?
Which of the following is a common side effect of Angiotensin-receptor blockers (ARBs)?
What is the primary mechanism of action of ARBs?
What is the primary mechanism of action of ARBs?
Why are ACE inhibitors or ARBs considered first-line anti-hypertensive agents in chronic kidney disease (CKD)?
Why are ACE inhibitors or ARBs considered first-line anti-hypertensive agents in chronic kidney disease (CKD)?
What is the primary adverse effect associated with ACE inhibitors?
What is the primary adverse effect associated with ACE inhibitors?
Which of the following is a contraindication for using Angiotensin-receptor blockers (ARBs)?
Which of the following is a contraindication for using Angiotensin-receptor blockers (ARBs)?
How do ARBs contribute to nephroprotection in chronic kidney disease?
How do ARBs contribute to nephroprotection in chronic kidney disease?
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