Podcast
Questions and Answers
Which of the following is NOT a potential consequence of abnormally automatic cardiac fibers in the ventricles?
Which of the following is NOT a potential consequence of abnormally automatic cardiac fibers in the ventricles?
- Ventricular tachycardia (VT)
- Ventricular premature depolarizations (VPDs)
- Ventricular fibrillation (VF)
- Atrial fibrillation (AF) (correct)
Which of the following is a common form of supraventricular tachycardia?
Which of the following is a common form of supraventricular tachycardia?
- Atrial fibrillation (AF)
- Ventricular tachycardia (VT)
- Premature atrial contractions
- Atrioventricular nodal reentrant tachycardia (correct)
Which of the following conditions must be present for the occurrence of abnormal impulse conduction through reentry?
Which of the following conditions must be present for the occurrence of abnormal impulse conduction through reentry?
- Increase in the velocity of impulse conduction
- Prolonged refractoriness in only one pathway
- At least one pathway for electrical impulse travel (correct)
- Bidirectional block in both conduction pathways
What contributes to prolonged refractoriness and slowed impulse conduction velocity in cardiac tissues?
What contributes to prolonged refractoriness and slowed impulse conduction velocity in cardiac tissues?
What may result if the rate of spontaneous impulse generation of abnormally automatic tissue exceeds that of the SA node?
What may result if the rate of spontaneous impulse generation of abnormally automatic tissue exceeds that of the SA node?
What serves as the pacemaker of the heart under normal circumstances?
What serves as the pacemaker of the heart under normal circumstances?
In case the SA node fails to generate depolarizations at a rate faster than that of the AV node, which structure may take over as the pacemaker?
In case the SA node fails to generate depolarizations at a rate faster than that of the AV node, which structure may take over as the pacemaker?
What may result from abnormal increased automaticity of the SA node?
What may result from abnormal increased automaticity of the SA node?
If both the SA node and AV node fail to generate depolarizations at a rate greater than 30-40/min, what may take over as the pacemaker?
If both the SA node and AV node fail to generate depolarizations at a rate greater than 30-40/min, what may take over as the pacemaker?
What occurs as a result of electrical depolarization of the atria and ventricles?
What occurs as a result of electrical depolarization of the atria and ventricles?