Podcast
Questions and Answers
What is the most common bacterial cause of otitis externa?
What is the most common bacterial cause of otitis externa?
Which of the following is NOT a risk factor for developing otitis externa?
Which of the following is NOT a risk factor for developing otitis externa?
Severe infections of otitis externa can lead to which of the following complications?
Severe infections of otitis externa can lead to which of the following complications?
Which variant of otitis externa is characterized by chronic drainage from a middle ear infection?
Which variant of otitis externa is characterized by chronic drainage from a middle ear infection?
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What is the primary organism responsible for otomycosis?
What is the primary organism responsible for otomycosis?
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Malignant otitis externa is particularly concerning for which of the following outcomes?
Malignant otitis externa is particularly concerning for which of the following outcomes?
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What typically leads to the recurrent episodes of acute otitis media in children?
What typically leads to the recurrent episodes of acute otitis media in children?
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What physical change occurs in the middle ear due to obstruction of the auditory tube in AOM?
What physical change occurs in the middle ear due to obstruction of the auditory tube in AOM?
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In which patient population is otomycosis most likely to occur?
In which patient population is otomycosis most likely to occur?
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Which bacteria are commonly implicated in acute otitis media?
Which bacteria are commonly implicated in acute otitis media?
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What is a hallmark clinical feature of acute otitis media (AOM)?
What is a hallmark clinical feature of acute otitis media (AOM)?
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Which factor is most associated with the development of otitis media with effusion (OME)?
Which factor is most associated with the development of otitis media with effusion (OME)?
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Which type of otitis media is characterized as 'dry' with no active infection?
Which type of otitis media is characterized as 'dry' with no active infection?
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What is a common cause of tympanic membrane perforation?
What is a common cause of tympanic membrane perforation?
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Which of the following describes a cholesteatoma?
Which of the following describes a cholesteatoma?
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What is a typical clinical feature associated with chronic otitis media?
What is a typical clinical feature associated with chronic otitis media?
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Which common complication of tympanic membrane perforation needs urgent referral?
Which common complication of tympanic membrane perforation needs urgent referral?
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What characterizes acute otitis media (AOM) on otoscopy?
What characterizes acute otitis media (AOM) on otoscopy?
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Which of the following is NOT a common clinical feature of otitis media with effusion (OME)?
Which of the following is NOT a common clinical feature of otitis media with effusion (OME)?
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What is a typical characteristic of Benign Paroxysmal Positional Vertigo (BPPV)?
What is a typical characteristic of Benign Paroxysmal Positional Vertigo (BPPV)?
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Which diagnostic method is used to confirm Benign Paroxysmal Positional Vertigo (BPPV)?
Which diagnostic method is used to confirm Benign Paroxysmal Positional Vertigo (BPPV)?
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What is a significant risk associated with otitis media, especially in children?
What is a significant risk associated with otitis media, especially in children?
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What triad of symptoms is critical for the diagnosis of Meniere’s disease?
What triad of symptoms is critical for the diagnosis of Meniere’s disease?
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Which factor is NOT known to trigger Meniere’s disease episodes?
Which factor is NOT known to trigger Meniere’s disease episodes?
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Which statement best describes Vestibular Neuronitis?
Which statement best describes Vestibular Neuronitis?
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What defines labyrinthitis in the context of inner ear health?
What defines labyrinthitis in the context of inner ear health?
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How is the acute phase of Vestibular Neuronitis characterized?
How is the acute phase of Vestibular Neuronitis characterized?
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What is the predominant symptom experienced during an episode of Meniere’s disease?
What is the predominant symptom experienced during an episode of Meniere’s disease?
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What is the primary characteristic of conductive hearing loss?
What is the primary characteristic of conductive hearing loss?
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Which condition is considered a type of sensorineural hearing loss?
Which condition is considered a type of sensorineural hearing loss?
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What is a common cause of presbycusis?
What is a common cause of presbycusis?
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What is the normal response for a tuning fork test using the Rinne method?
What is the normal response for a tuning fork test using the Rinne method?
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Which statement accurately describes ototoxicity?
Which statement accurately describes ototoxicity?
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What distinguishes the audiometry testing method from tuning fork tests?
What distinguishes the audiometry testing method from tuning fork tests?
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What phenomenon occurs in conductive hearing loss during the Weber test?
What phenomenon occurs in conductive hearing loss during the Weber test?
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Which frequency range does the human ear primarily excel at detecting?
Which frequency range does the human ear primarily excel at detecting?
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Which pathological condition is characterized by a collection of skin cells and other debris in the middle ear?
Which pathological condition is characterized by a collection of skin cells and other debris in the middle ear?
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What is the main effect of otosclerosis on hearing?
What is the main effect of otosclerosis on hearing?
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What is the primary complication associated with cholesteatoma?
What is the primary complication associated with cholesteatoma?
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Which cells primarily undergo implantation leading to primary acquired cholesteatoma?
Which cells primarily undergo implantation leading to primary acquired cholesteatoma?
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What is one potential severe consequence of cholesteatomas if left untreated?
What is one potential severe consequence of cholesteatomas if left untreated?
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Where do cholesteatomas most commonly develop in the tympanic membrane?
Where do cholesteatomas most commonly develop in the tympanic membrane?
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Which of the following contributes to the growth of a cholesteatoma?
Which of the following contributes to the growth of a cholesteatoma?
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What is the hallmark clinical feature of cholesteatoma?
What is the hallmark clinical feature of cholesteatoma?
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What structural change occurs to the tympanic membrane in primary acquired cholesteatoma?
What structural change occurs to the tympanic membrane in primary acquired cholesteatoma?
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Which of the following is NOT typically associated with cholesteatoma?
Which of the following is NOT typically associated with cholesteatoma?
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What is often implicated in the bacterial infection associated with cholesteatomas?
What is often implicated in the bacterial infection associated with cholesteatomas?
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What is a key characteristic of primary acquired cholesteatoma regarding age onset?
What is a key characteristic of primary acquired cholesteatoma regarding age onset?
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Study Notes
HEENT 2 - Overview
- Topics covered include ear and vestibular apparatus pathologies, general mouth and sinus anatomy, and the neurology of olfactory and gustatory pathways.
- This is a BMS 200 course.
Ear Pathologies
- Conductive vs. sensorineural hearing loss: Distinguishing between types of hearing loss.
- Otitis externa & otomycosis: Ear canal infections.
-
Otitis media: Inflammation of the middle ear.
- Acute otitis media (AOM): Rapid onset of symptoms.
- Otitis media with effusion (OME): Fluid buildup in the middle ear.
- Chronic otitis media: Long-term inflammation.
- Cholesteatoma: A non-cancerous cyst with keratin debris.
- Tympanic membrane perforations: A hole in the eardrum.
Hearing Loss - Generalities
- Incidence of hearing loss: About 15% of people between 20 and 69 years old experience some degree of high-frequency hearing loss, often due to noise.
- Outer hair cell vulnerability: Outer hair cells are more susceptible to noise damage than inner hair cells.
- Presbycusis: Age-related hearing loss caused by a combination of neuronal and hair cell loss.
- Ototoxic substances: Antibiotics and other drugs can damage the inner ear.
Hearing Loss - Generalities (Conductive and Sensorineural)
- Conductive hearing loss: Impaired sound transmission in the outer or middle ear. Impacted by trauma, infection, plugging of the ear canal, otosclerosis, or cholesteatoma. This affects all frequencies.
- Sensorineural hearing loss: Loss of higher frequencies more than lower ones. This is often caused by presbycusis, ototoxic agents, or noise. Also involves endolymph problems, labyrinth or CN 8 infections/tumors.
Tuning Fork Investigation of Hearing
- Weber test: Tuning fork placed on the head to assess bone conduction hearing. If sound is louder in one ear, it might suggest conduction hearing loss in that ear. Sound is heard equally in both ears if normal.
- Rinne test: Tuning fork placed on mastoid then near ear to assess both air and bone conduction. Air conducted sound is heard after bone conduction stops if normal.
Audiometry
- Frequency range: Human hearing range from 20 to 20,000 Hz, though best between 1000-4000 Hz for speech (500-2000Hz).
- Audiometry assessment: Measures hearing at specific frequencies to characterize hearing loss better than tuning forks.
- Different forms of audiometry: Includes speech audiometry.
Otitis Externa
- Causative agent: Bacterial (90%) - mainly staphylococcal, Pseudomonas aeruginosa, or E. coli.
- Risk factors: Humidity, cerumen loss (trauma/Q-tip use), heat, increased pH, ear canal obstruction, and exposure to water (especially colonized).
- Clinical features: Otalgia (pain in ear), movement of pinna can elicit pain, otorrhea (ear discharge) - can be purulent, itching of external canal, edema/occlusion of ear canal, conductive hearing loss.
- Treatment: Topical antibiotics.
Otitis Externa - Variations
- Furunculosis: Staphylococcal infection in outer 1/3 ear canal. Often painful.
- Chronic otitis externa: Repetitive trauma cause, more itchy, chronic drainage.
- Malignant/necrotizing otitis externa: Progressive, serious, often damaging temporal bone/cranial nerves.
- Otomycosis: Fungal infection (usually 80% Aspergillus, remaining common species Candida).
Otitis Media
- Acute otitis media (AOM): Rapid onset of symptoms (fever, otalgia), typically lasting less than 6 months, recurrent is ≥ 3 episodes within 6 months with complete resolution in between episodes or ≥ 4 in 12 months.
- Causes: Auditory tube dysfunction, blocked tube due to adenoid swelling/URTI. Bacteria often from upper airway/oral flora.
- Symptoms: Rapid onset and severity of otalgia, fever, and sometimes conductive hearing loss. Possible otorrhoea (possible).
- Clinical features: Bulging, redness in tympanic membrane, often opaque, loss of bony landmarks, limited mobility (especially with effusion behind membrane) on pneumatoscopy.
- OME (Serous Otitis Media): Unresolved AOM, persistent effusion in affected ear. 3 months of persistent effusion.
- Clinical features: Conductive hearing loss, fullness in ear (possible tinnitus or low-grade fever).
- Otoscopic features: Translucent/gray TM, possibly fluid behind TM visible as air/fluid levels or bubbles. Loss of light reflex/reduced mobility.
Tympanic Membrane Perforations
- Causes: Middle ear infections, trauma (barotrauma or physical injury).
- Clinical features: Sudden onset of pain, hearing loss, might be bloody otorrhoea, dizziness, or tinnitus.
- Healing: Often self-heals spontaneously.
- Importance of location: Posterio-superior damage to TM more likely to damage ossicles, needing urgent referral.
Chronic Otitis Media
- Causes: Recurrent or chronic ear infections, Eustachian tube dysfunction (approximately 70% of patients observed after middle ear surgery).
- Types: Suppurative or serous, described by chronic drainage through perforated TM, Benign - no active infection "dry". Inflammatory effect, both middle ear and affected mastoid.
- Clinical features: Otorrhoea. Possible conductive hearing loss, tinnitus and fullness in ear.
Cholesteatomas
- Definition: Non-neoplastic cysts lined with keratinizing epithelium. Filled with debris.
- Location: Mostly in the posterior-superior region (attic) of the middle ear.
- Size: Typically 1-4 cm.
- Types: Congenital (rare). Primary acquired, Secondary acquired.
- Complications: Conductive hearing loss (most minor), bony erosion, temporal bone infections, meningitis, intracranial involvement, deep neck infections, possible rupture resulting in life-threatening problems.
Dizziness
- Classification: Vertiginous (vertigo-environment moving) or Non-vertiginous (organic or functional).
- Vertiginous/vertigo: Caused by issues with inner ear or brainstem-cerebellar disorders (peripheral/central).
- Non-vertiginous: Usually organic disorders involving vision compromise or low blood pressure, or functional disorders related to mood.
Benign Paroxysmal Positional Vertigo (BPPV)
- Description: Short-lasting episodes of vertigo triggered by head position changes (especially when getting out of bed, turning head), typically not involving spontaneous nystagmus.
- Presentation: Quick onset, usually <1 minute, of rotatory nystagmus (movement of eyeball), often accompanied by nausea or vomiting.
- Cause: Free-floating otoliths in semicircular canals causing sensory disruption.
- Diagnosis: Dix-Hallpike test, showing nystagmus when turning head in specific positions.
Meniere's Disease
- Symptoms: Episodic attacks of tinnitus, hearing loss, and vertigo lasting minutes to hours.
- Mechanism: Endolymphatic over-accumulation in the membranous labyrinth may result in distorting inner ear.
- Triggers: High salt intake, caffeine, stress, nicotine, alcohol
- Diagnostics: Two spontaneous episodes of rotational vertigo lasting > 20 minutes. Associated to sensorineural hearing loss.
Vestibular Neuronitis
- Description: Sudden onset of disabling vertigo, often accompanied by nausea/vomiting, and imbalance. Hearing loss is usually absent.
- Presentation: Acute phase: severe vertigo, nausea, vomiting, and imbalance lasting 1-5 days, nystagmus. Convalescent phase: imbalance, motion sickness lasting days to weeks, gradual vestibular adaptation for weeks-months.
Labyrinthitis
- Description: Acute infection of the inner ear resulting in vertigo and hearing loss. Infection can be viral (serous) or purulent/bacterial (often a complication of acute or chronic otitis media).
- Presentation: Sudden onset of vertigo, nausea/vomiting, tinnitus, hearing loss (unilateral). May be associated with fever/pain(uncommon), bacterial may involve meningitis.
- Causes: Bacterial (S. pneumoniae, H. influenzae, M. catarrhalis, etc.), viral (rubella, CMV, measles, mumps, varicella zoster), and possible autoimmune cause; meningitis could be complication.
Acoustic Neuroma
- Description: Intracranial tumor that develops from Schwann cells in vestibular and/or cochlear nerve. Often observed in the cerebellopontine angle with ~ 80% of tumors in the area being related to acoustic.
- Presentation: Imparting hearing, sometimes facial nerve symptoms via tumor expansion. May have elevated intracranial pressure if tumor progresses enough.
- Diagnosis: MRI imaging of the relevant area of the brain.
- Treatment: Surgical removal/radiation.
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Description
Test your knowledge on the anatomy and pathologies of the ear, mouth, and sinus as covered in the BMS 200 course. This quiz focuses on various types of hearing loss, infections, and the neurological pathways related to taste and smell. Dive into the details of otitis and other key topics.