Podcast
Questions and Answers
What is the recommended daily dose of iron sulfate for infants aged 9 to 12 months for anemia treatment?
What is the recommended daily dose of iron sulfate for infants aged 9 to 12 months for anemia treatment?
- 1 mg/kg
- 6 mg/kg
- 3-6 mg/kg (correct)
- 10 mg/kg
How long should iron therapy be continued for patients with iron deficiency anemia until iron stores are replenished?
How long should iron therapy be continued for patients with iron deficiency anemia until iron stores are replenished?
- 1 month
- 3 months
- 2 months (correct)
- 2 weeks
What are the typical signs of improvement in patients with megaloblastic anemia after starting vitamin B12 or folate therapy?
What are the typical signs of improvement in patients with megaloblastic anemia after starting vitamin B12 or folate therapy?
- No change in symptoms
- Neurological symptoms worsening
- Immediate Hb increase
- Rapid reticulocytosis (correct)
When should a patient with iron deficiency anemia be reevaluated if reticulocytosis does not occur after starting oral iron therapy?
When should a patient with iron deficiency anemia be reevaluated if reticulocytosis does not occur after starting oral iron therapy?
How long should iron sulfate be administered to infants aged 9 to 12 months for the treatment of anemia?
How long should iron sulfate be administered to infants aged 9 to 12 months for the treatment of anemia?
What is the primary goal of oral iron therapy in patients with iron deficiency anemia?
What is the primary goal of oral iron therapy in patients with iron deficiency anemia?
When should reticulocytosis occur after starting vitamin B12 therapy?
When should reticulocytosis occur after starting vitamin B12 therapy?
What is the expected timeline for Hb normalization after beginning folate therapy?
What is the expected timeline for Hb normalization after beginning folate therapy?
Which enzyme deficiency can lead to chronic nonspherocytic hemolytic anemia in the presence of normal erythrocyte function?
Which enzyme deficiency can lead to chronic nonspherocytic hemolytic anemia in the presence of normal erythrocyte function?
In which population is Class II G6PD enzyme variant more common?
In which population is Class II G6PD enzyme variant more common?
How often should iron, TIBC, transferrin saturation, and ferritin levels be monitored during ESA therapy?
How often should iron, TIBC, transferrin saturation, and ferritin levels be monitored during ESA therapy?
What is the enzyme activity level of Class III G6PD enzyme variant?
What is the enzyme activity level of Class III G6PD enzyme variant?
What is the recommended monitoring interval for Hb and Hct in premature infants under iron therapy?
What is the recommended monitoring interval for Hb and Hct in premature infants under iron therapy?
Which of the following is NOT a major type of drug-induced blood dyscrasia?
Which of the following is NOT a major type of drug-induced blood dyscrasia?
What is the prevalence of Class IV G6PD enzyme variant?
What is the prevalence of Class IV G6PD enzyme variant?
In drug-induced increased hemolysis, where can destruction of red cells occur?
In drug-induced increased hemolysis, where can destruction of red cells occur?
Which region has an increased frequency of Glucose-6-Phosphate Dehydrogenase (G6PD) deficiency?
Which region has an increased frequency of Glucose-6-Phosphate Dehydrogenase (G6PD) deficiency?
What is the consequence of G6PD deficiency combined with oxidative stress on red blood cells?
What is the consequence of G6PD deficiency combined with oxidative stress on red blood cells?
