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Questions and Answers
Adrenaline primarily acts on the heart through its interaction with the beta-2 receptors.
Adrenaline primarily acts on the heart through its interaction with the beta-2 receptors.
False
The vasoconstriction effect of high doses of adrenaline is mainly mediated by alpha-2 receptors.
The vasoconstriction effect of high doses of adrenaline is mainly mediated by alpha-2 receptors.
False
Epinephrine's effect on bronchial muscles is relaxation, primarily mediated by alpha-2 receptors.
Epinephrine's effect on bronchial muscles is relaxation, primarily mediated by alpha-2 receptors.
False
Adrenaline causes relaxation of smooth muscles in the gastrointestinal tract, primarily through beta-2 receptors.
Adrenaline causes relaxation of smooth muscles in the gastrointestinal tract, primarily through beta-2 receptors.
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Alpha-1 receptors are mainly present in the heart and kidneys.
Alpha-1 receptors are mainly present in the heart and kidneys.
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Beta-2 receptors play a role in the contraction of the iris radial muscle, leading to mydriasis.
Beta-2 receptors play a role in the contraction of the iris radial muscle, leading to mydriasis.
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Acetylcholine primarily affects beta receptors on blood vessels.
Acetylcholine primarily affects beta receptors on blood vessels.
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Epinephrine is rapidly inactivated in the liver by Monoamine Oxidase (MAO) and Catechol-o-methyltransferase (COMT).
Epinephrine is rapidly inactivated in the liver by Monoamine Oxidase (MAO) and Catechol-o-methyltransferase (COMT).
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Norepinephrine has significant effects on beta-2 receptors.
Norepinephrine has significant effects on beta-2 receptors.
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Noradrenaline is used as a vasopressor agent in treating hypotensive states.
Noradrenaline is used as a vasopressor agent in treating hypotensive states.
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Propranolol increases heart rate.
Propranolol increases heart rate.
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Atropine is an adrenergic antagonist.
Atropine is an adrenergic antagonist.
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Norepinephrine causes vasodilation.
Norepinephrine causes vasodilation.
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Acetylcholine is metabolized by MAO and COMT enzymes.
Acetylcholine is metabolized by MAO and COMT enzymes.
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Epinephrine is ineffective when administered orally.
Epinephrine is ineffective when administered orally.
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Propranolol can be used for the prophylaxis of angina pectoris.
Propranolol can be used for the prophylaxis of angina pectoris.
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Acetylcholine is a non-selective muscarinic and nicotinic agonist.
Acetylcholine is a non-selective muscarinic and nicotinic agonist.
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Atropine is a non-selective muscarinic antagonist.
Atropine is a non-selective muscarinic antagonist.
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Acetylcholine causes vasodilation through the NO pathway on vascular endothelium.
Acetylcholine causes vasodilation through the NO pathway on vascular endothelium.
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Adrenaline is commonly used as a cholinergic agonist in clinical practice.
Adrenaline is commonly used as a cholinergic agonist in clinical practice.
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Propranolol acts as a cholinergic antagonist.
Propranolol acts as a cholinergic antagonist.
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Noradrenaline mediates its effects through activation of muscarinic receptors.
Noradrenaline mediates its effects through activation of muscarinic receptors.
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