Altitude Illness and Submersion Injuries
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Questions and Answers

What is the typical onset time for symptoms of Acute Mountain Sickness (AMS) after arriving at altitudes over 8000 feet?

  • 24-36 hours
  • 12-24 hours
  • 1-2 hours
  • 6-10 hours (correct)
  • Which treatment is recommended to reduce symptoms of AMS if acclimatization fails after 24 hours?

  • Continue ascent quietly
  • Administer acetazolamide (correct)
  • Rest without any intervention
  • Increase sleeping altitude
  • How long after ascent will most patients with AMS typically resolve without treatment?

  • 1 week
  • 24 hours
  • 72 hours (correct)
  • 48 hours
  • What is the hallmark sign of High-Altitude Cerebral Edema (HACE)?

    <p>Ataxic gait</p> Signup and view all the answers

    What is the appropriate action to take if a patient exhibits altered consciousness due to HACE?

    <p>Immediately descend at least 500 to 1000 meters</p> Signup and view all the answers

    What symptoms are typically associated with High-Altitude Pulmonary Edema (HAPE)?

    <p>Dyspnea on exertion and tachypnea</p> Signup and view all the answers

    Which medication can be administered to treat nausea and vomiting in a patient with AMS?

    <p>Prochlorperazine</p> Signup and view all the answers

    What should be maintained in terms of oxygen saturation for a patient experiencing HAPE during treatment?

    <p>Greater than 90%</p> Signup and view all the answers

    At what altitude does acute altitude illness commonly occur in travelers?

    <p>Above 2500 m</p> Signup and view all the answers

    Which medication is specifically contraindicated during the treatment of HAPE?

    <p>Beta-blockers</p> Signup and view all the answers

    What factor is unique to HACE compared to AMS and HAPE?

    <p>It manifests with ataxia and altered consciousness.</p> Signup and view all the answers

    Which condition is most accurately associated with very high altitudes ranging from 3500 to 5500 m?

    <p>Extreme hypoxia</p> Signup and view all the answers

    What physiological change often occurs at high altitudes to compensate for decreased oxygen levels?

    <p>Increased ventilation</p> Signup and view all the answers

    What is the primary risk factor for developing high-altitude illness?

    <p>Prior altitude sickness</p> Signup and view all the answers

    What symptom is often the first indication of altitude exposure?

    <p>Headache</p> Signup and view all the answers

    Which altitude is defined as extreme altitude and poses significant health risks without supplemental oxygen?

    <p>Above 5500 m</p> Signup and view all the answers

    What percentage of travelers may experience symptoms of acute altitude illness when rapidly ascending above 2500 m?

    <p>25%</p> Signup and view all the answers

    Which of the following is NOT one of the three main illnesses associated with high-altitude exposure?

    <p>Chronic Mountain Sickness (CMS)</p> Signup and view all the answers

    What characterizes wet non-fatal drowning?

    <p>Survival after aspiration of liquid into the lungs</p> Signup and view all the answers

    Which of the following is a common physical sign observed after a person is rescued from drowning?

    <p>Altered level of consciousness (LOC)</p> Signup and view all the answers

    What is the primary physiological response that begins drowning episodes?

    <p>Breath-holding due to panic</p> Signup and view all the answers

    What is the major component of morbidity and mortality associated with drowning?

    <p>Cerebral hypoxia</p> Signup and view all the answers

    Which group is considered at higher risk for drowning incidents?

    <p>Males of all ages</p> Signup and view all the answers

    What typically occurs during the drowning process leading to hypoxemia?

    <p>Breath-holding followed by inspiratory efforts</p> Signup and view all the answers

    What is a potential late-onset symptom of drowning injuries?

    <p>Respiratory distress</p> Signup and view all the answers

    Which condition may arise due to the aspiration of water during a drowning event?

    <p>Noncardiogenic pulmonary edema</p> Signup and view all the answers

    What is a common cardiovascular complication observed in nonfatal drowning victims?

    <p>Atrial fibrillation</p> Signup and view all the answers

    Which threshold volume of water aspiration is cited as significant for blood volume changes in drowning victims?

    <p>11 mL/kg</p> Signup and view all the answers

    What is considered the most critical initial treatment for victims of submersion injury?

    <p>Rescue breathing</p> Signup and view all the answers

    Which factor significantly influences patient outcomes in drowning incidents?

    <p>Presence of contaminants</p> Signup and view all the answers

    What is the recommended action if a hypothermic drowning patient has weak pulses?

    <p>Assess for pulses for at least one minute</p> Signup and view all the answers

    What condition must be monitored during the management of drowning victims?

    <p>Core body temperature</p> Signup and view all the answers

    What is the primary purpose of continuous pulse oximetry in managing drowning victims?

    <p>To monitor oxygen saturation levels</p> Signup and view all the answers

    What is one potential reason for neurologic deterioration in drowning victims?

    <p>Ischemia due to hypoxemia</p> Signup and view all the answers

    In an asymptomatic drowning patient, what is the advised observation time frame before discharge?

    <p>8 hours</p> Signup and view all the answers

    What indicates a need for intubation in a drowning victim?

    <p>Inability to protect the airway</p> Signup and view all the answers

    What electrolyte imbalance is typically associated with freshwater drowning?

    <p>Hyponatremia</p> Signup and view all the answers

    Which is a possible cause of chest discomfort in drowning victims?

    <p>Takotsubo cardiomyopathy</p> Signup and view all the answers

    Which management phase involves immediate resuscitation efforts following a drowning incident?

    <p>Prehospital care</p> Signup and view all the answers

    What characteristic changes in ECG might suggest myocardial ischemia after drowning?

    <p>ST depression and T wave inversion</p> Signup and view all the answers

    Study Notes

    High-Altitude Definitions

    • High altitude: 1500 to 3500 m (4,950 to 11,500 ft); causes decreased exercise performance and increased ventilation at rest.
    • Altitude illness commonly occurs with rapid ascent above 2500 m (8,200 ft).
    • Very high altitude: 3500 to 5500 m (11,500 to 18,050 ft); PaO2 falls below 60 mm Hg, SaO2 drops below 90%.
    • Extreme altitude: above 5500 m (roughly 18,000 ft); marked hypoxemia, hypocapnia, and impossible successful acclimatization.
    • Acute altitude illness occurs at elevations >2500 m, affecting 25% of rapid ascenders.
    • Symptoms include hypoxia and hypothermia, influenced by rapid ascent, exertion, prior illness, and sea level residency.
    • Compensatory responses: increased ventilation, respiratory rate (RR), heart rate (HR), and cardiac output (CO).
    • Main altitude illnesses: Acute Mountain Sickness (AMS), High-Altitude Cerebral Edema (HACE), High-Altitude Pulmonary Edema (HAPE).

    High-Altitude Headache

    • Often the first and sometimes only symptom following altitude exposure.
    • Treatment: Oxygen (if available), NSAIDs, or Acetaminophen.

    Acute Mountain Sickness (AMS)

    • Onset occurs 6-10 hours after reaching altitudes >8000 ft; symptoms may resolve within 72 hours without treatment.
    • Symptoms: Headache (HA), fatigue, anorexia, nausea/vomiting (N/V), persistent dizziness, frequent awakening during sleep.
    • Treatment involves halting ascent, monitoring, and acclimatization; descent is necessary if symptoms worsen.
    • Acetazolamide, Ginkgo biloba, and symptom relief medications (NSAIDs, prochlorperazine) recommended.

    High-Altitude Cerebral Edema (HACE)

    • Rare condition occurs due to rapid ascent.
    • Signs and symptoms: ataxic gait, altered consciousness, headache, N/V, hallucinations, hypoxemia, seizures, cranial nerve palsy.
    • Diagnosis typically requires imaging (CT/MRI) to rule out other causes.
    • Treatment includes immediate descent, dexamethasone, oxygen therapy, and possibly hyperbaric treatment.

    High-Altitude Pulmonary Edema (HAPE)

    • Develops within 1-4 days after rapid ascent and may follow AMS.
    • Signs and symptoms: decreased exercise tolerance, dyspnea on exertion, cough, tachycardia/tachypnea, fatigue, low-grade fever, cyanosis, audible rales, possible pink/blood-tinged sputum.
    • Vital signs indicate hypoxemia and may show patchy lung edema on chest X-ray.
    • Treatment necessitates descent, oxygen therapy, nifedipine, inhaled beta-agonists, and potentially hyperbaric therapy.

    Terminology

    • Drowning: Respiratory impairment caused by submersion in liquid.
    • Fatal Drowning: Death due to drowning with no recovery.
    • Non-Fatal Drowning: Survival after submersion, which may lead to temporary or permanent effects.
    • Wet Non-Fatal Drowning: Survival following liquid aspiration into lungs.
    • Dry Non-Fatal Drowning: Survival following asphyxia from reflex laryngospasm.

    Risk Factors

    • High vulnerability in children and males.
    • Substance use (ETOH/drug) increases risk.
    • Seizure disorders can lead to complications.
    • Hypoxia can result in respiratory and cardiac arrest.

    Signs and Symptoms

    • Post-rescue: anxiety, vomiting, wheezing, altered level of consciousness (LOC).
    • Physical symptoms: tachypnea, retractions, and cyanosis.
    • Respiratory signs can be delayed, appearing up to 6 hours after incident.
    • Assess for potential additional injuries, including head trauma and spinal injuries.

    Submersion Pathophysiology

    • Drowning initiates panic and loss of normal breathing, leading to breath-holding and struggles for air.
    • Reflex inspiratory efforts can cause hypoxemia either through aspiration or laryngospasm.
    • Cerebral hypoxia is a major contributor to morbidity and mortality.

    Pulmonary Pathophysiology

    • Fluid aspiration leads to varying degrees of hypoxemia.
    • Fresh and salt water can both wash out surfactant, leading to conditions like noncardiogenic pulmonary edema and ARDS.
    • Symptoms may include shortness of breath, crackles, and wheezing; radiographs may show variable pulmonary edema.

    Neuro Pathophysiology

    • Hypoxemia and ischemia can result in neuronal damage, cerebral edema, and increased intracranial pressure.
    • 20% of nonfatal drowning victims may sustain neurological damage despite successful cardiopulmonary resuscitation (CPR).

    Cardiac Pathophysiology

    • Potential arrhythmias (sinus tachycardia, bradycardia, atrial fibrillation) often arise from hypothermia and hypoxemia.
    • Diving and swimming can provoke fatal arrhythmias in those with congenital long QT syndrome.
    • ECG changes may indicate myocardial ischemia linked to factors like takotsubo cardiomyopathy or hypothermia.

    Salt Water vs Fresh Water Drowning

    • Earlier views suggested significant differences between saltwater and freshwater drowning impacts.
    • Critical threshold for blood volume changes is approximately 11 mL/kg aspirated; nonfatal victims rarely exceed 3-4 mL/kg.
    • Water temperature and contaminants can influence patient outcomes.

    Management of Submersion Injuries

    • Management phases: prehospital care, emergency department (ED) care, and inpatient care.
    • Immediate resuscitation by bystanders significantly improves outcomes.
    • Priority is generally given to ventilation; rescue breathing should start quickly upon reaching stable surfaces.
    • CPR should be initiated after confirming absence of breath and assessing for weak pulses in hypothermic victims.
    • Intubation is indicated for neurologically deteriorating patients or those with inadequate oxygenation.

    Trauma Evaluation and Care Protocols

    • Remove wet clothing, conduct imaging for other injuries.
    • Continuous monitoring of pulse oximetry and capnography is crucial.
    • Administer inhaled beta-adrenergic agonists for bronchospasm and monitor cardiac activity through EKG.
    • Rule out hypothermia and evaluate underlying disorders through relevant lab tests (ECG, CBC, CMP, UDS, cardiac enzymes).

    Prognosis and Outcomes

    • Survival rates vary, reported as high as 75%; neurologic deficits occur in about 6% of survivors.
    • Some studies indicate a mortality rate of 74%, with 4% surviving with severe disabilities.
    • Poor prognosis factors include:
      • Submersion duration exceeding 5 minutes.
      • Basic life support initiation taking longer than 10 minutes.
      • Total resuscitation efforts lasting more than 25 minutes.
      • Age over 14 years.
      • Low Glasgow Coma Scale scores.

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    Description

    This quiz covers the definitions and effects of altitude on health, specifically focusing on altitude illness and the complications of high-altitude exposure. Understand the classifications of high altitude and the physiological responses to low oxygen environments. Ideal for students studying environmental science or physiology.

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