Special Populations Review PDF
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This document provides a review of special populations, focusing on obstetrics and physiological changes during pregnancy. It covers topics such as uterine changes, circulatory adaptations, and respiratory adjustments.
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Special Populations Review Obstetrics Physiologic Maternal Changes During Pregnancy Physiologic changes occurring throughout pregnancy can: Alter normal response to trauma Exacerbate or create medical conditions Physiologic Maternal Changes During Pregnancy Significant changes...
Special Populations Review Obstetrics Physiologic Maternal Changes During Pregnancy Physiologic changes occurring throughout pregnancy can: Alter normal response to trauma Exacerbate or create medical conditions Physiologic Maternal Changes During Pregnancy Significant changes occur in the uterus. Before pregnancy, the uterus: Weighs about 2.5 oz (70 gm) Has a fluid capacity of about 10 mL At the end of the pregnancy, the uterus: Weighs as much as 2.24 to 2.6 lb (1100 to 1200 g) Has the capacity to hold about 5,000 mL Physiologic Maternal Changes During Pregnancy Measurement of the fundus may indicate developmental problems: Uterine growth problems Breech position Twins © Jones & Bartlett Learning. Physiologic Maternal Changes During Pregnancy Pressure occurs on intestine and rectum Smooth muscle in the GI tract relaxes Stomach is not emptied as quickly Kidneys increase in size and volume Ureters increase in diameter Hormones cause changes to the skin, hair, and eyes Physiologic Maternal Changes During Pregnancy Circulatory changes Blood volume increases up to 50% more to: Meet fetal metabolic needs Adequately perfuse maternal organs Help compensate for blood loss in delivery Physiologic Maternal Changes During Pregnancy Circulatory changes (cont’d) The number of red blood cells increases. Clotting factors increase while fibrinolytic factors are depressed. The size of the heart increases. Physiologic Maternal Changes During Pregnancy The heart rate gradually increases by an average of 15 to 20 beats/min by term. ECG changes may include: Ectopic beats Supraventricular tachycardia Slight left axis deviation Lead II changes Physiologic Maternal Changes During Pregnancy Sensitivity to body position increases as gestation increases. Lying supine can cause compression of the inferior vena cava. If pressure is not relieved, cardiac output is decreased. Physiologic Maternal Changes During Pregnancy The birthing position may stress the cardiovascular system. The lithotomy position is standard in the United States. © Jones & Bartlett Learning. Physiologic Maternal Changes During Pregnancy Respiratory changes The diaphragm is pushed up by the uterus. Maternal oxygen demand increases. Progesterone does the following: Decreases threshold to carbon dioxide Causes the bronchi to dilate Regulates mucus production Physiologic Maternal Changes During Pregnancy Respiratory changes (cont’d) A decrease in: Expiratory reserve volume Functional residual capacity Residual volume An increase in: Tidal volume Inspiratory reserve volume Physiologic Maternal Changes During Pregnancy Maternal metabolism Weight gain averages 27 lb (12.3 kg). Increased blood volume and intracellular and extracellular fluid Uterine growth Placental and fetal growth Increased breast tissue Increased proteins and fat deposits Physiologic Maternal Changes During Pregnancy Maternal metabolism Relaxin softens collagenous tissues and relaxes the ligamentous system. The demand for carbohydrates increases. Physiologic Maternal Changes During Pregnancy Physiologic changes occurring throughout pregnancy can: Alter normal response to trauma Exacerbate or create medical conditions Physiologic Maternal Changes During Pregnancy Significant changes occur in the uterus. Before pregnancy, the uterus: Weighs about 2.5 oz (70 gm) Has a fluid capacity of about 10 mL At the end of the pregnancy, the uterus: Weighs as much as 2.24 to 2.6 lb (1100 to 1200 g) Has the capacity to hold about 5,000 mL Physiologic Maternal Changes During Pregnancy Measurement of the fundus may indicate developmental problems: Uterine growth problems Breech position Twins © Jones & Bartlett Learning. Physiologic Maternal Changes During Pregnancy Pressure occurs on intestine and rectum Smooth muscle in the GI tract relaxes Stomach is not emptied as quickly Kidneys increase in size and volume Ureters increase in diameter Hormones cause changes to the skin, hair, and eyes Physiologic Maternal Changes During Pregnancy Circulatory changes Blood volume increases up to 50% more to: Meet fetal metabolic needs Adequately perfuse maternal organs Help compensate for blood loss in delivery Physiologic Maternal Changes During Pregnancy Circulatory changes (cont’d) The number of red blood cells increases. Clotting factors increase while fibrinolytic factors are depressed. The size of the heart increases. Physiologic Maternal Changes During Pregnancy The heart rate gradually increases by an average of 15 to 20 beats/min by term. ECG changes may include: Ectopic beats Supraventricular tachycardia Slight left axis deviation Lead II changes Physiologic Maternal Changes During Pregnancy Sensitivity to body position increases as gestation increases. Lying supine can cause compression of the inferior vena cava. If pressure is not relieved, cardiac output is decreased. Physiologic Maternal Changes During Pregnancy The birthing position may stress the cardiovascular system. The lithotomy position is standard in the United States. © Jones & Bartlett Learning. Physiologic Maternal Changes During Pregnancy Respiratory changes The diaphragm is pushed up by the uterus. Maternal oxygen demand increases. Progesterone does the following: Decreases threshold to carbon dioxide Causes the bronchi to dilate Regulates mucus production Physiologic Maternal Changes During Pregnancy Respiratory changes (cont’d) A decrease in: Expiratory reserve volume Functional residual capacity Residual volume An increase in: Tidal volume Inspiratory reserve volume Physiologic Maternal Changes During Pregnancy Maternal metabolism Weight gain averages 27 lb (12.3 kg). Increased blood volume and intracellular and extracellular fluid Uterine growth Placental and fetal growth Increased breast tissue Increased proteins and fat deposits Physiologic Maternal Changes During Pregnancy Maternal metabolism Relaxin softens collagenous tissues and relaxes the ligamentous system. The demand for carbohydrates increases. Supine Hypotensive Syndrome Uterus may compress the inferior vena cava. May diminish or occlude venous blood return to the heart Can result in significant hypotension and fetal distress Supine Hypotensive Syndrome Management includes: Placing the patient in the left lateral recumbent position. Treating underlying causes. Monitoring blood pressure and other vital signs. Obtaining an ECG. Cardiac Conditions Determine the nature and treatment of any heart condition. Cardiac medications Diagnosed with dysrhythmias or heart murmurs History of rheumatic fever Born with congenital heart defect Episodes of dizziness, light-headedness Hypertensive Disorders Chronic hypertension Gestational hypertension Blood pressure equal to or greater than 140/90 mm Hg Develops after the 20th Increased risk for stroke or week of pregnancy other cardiovascular Resolves spontaneously problems Hypertensive Disorders Preeclampsia Risk factors include: First pregnancy before age 20 years Women with advanced maternal age History of multiple pregnancies Hypertension Renal disease Diabetes Hypertensive Disorders Preeclampsia (cont’d) Manifests after 20th week with a triad of symptoms including: Edema Gradual onset of hypertension Protein in the urine Hypertensive Disorders Preeclampsia (cont’d) May require emergency hypertensive medications Other conditions that may accompany preeclampsia: Liver or renal failure Cerebral hemorrhage Abruptio placenta HELLP syndrome Seizures Treatment is difficult because drugs may cause fetal distress. Magnesium sulfate is recommended. Potential complications may include: Abruptio placenta Hemorrhage Disseminated intravascular coagulation Diabetes Gestational diabetes mellitus: inability to process carbohydrates during pregnancy Treatment consists of: Diet control Oral hypoglycemic medications Diabetes Diabetes may be affected by pregnancy. Patients with a history of diabetes should have a blood glucose level test. Respiratory Disorders Shortness of breath or general dyspnea is one of the most common complaints. Often caused by hormone-related changes Asthma is a common condition that complicates pregnancy. Respiratory Disorders Maternal asthma complications: Fetal asthma complications: Premature labor Premature birth Preeclampsia Low birth rate Respiratory failure Growth retardation Vaginal hemorrhage Fetal death Eclampsia Pathophysiology of Bleeding Related to Pregnancy Abortion Expulsion of the fetus before the 20th week of gestation Classified as: Spontaneous abortion (miscarriage) Elective (intentional) abortion Pathophysiology of Bleeding Related to Pregnancy Habitual abortions: three or more consecutive miscarriages Causes include: Ovarian issues Uterine malformations Cervical conditions Infections Pathophysiology of Bleeding Related to Pregnancy Threatened abortion: abortion attempting to take place It is characterized by vaginal bleeding in the first half of pregnancy. It can progress, or it may subside. The prehospital role is transport and support. Pathophysiology of Bleeding Related to Pregnancy Imminent abortion: spontaneous abortion that cannot be prevented Signs and symptoms include: Severe abdominal pain Vaginal bleeding Cervical dilation Maintain blood pressure and prevent hypovolemia. Pathophysiology of Bleeding Related to Pregnancy Imminent abortion (cont’d) Treatment includes: Establishing an IV line of normal saline Administering 100% supplemental oxygen Obtaining an ECG Providing emotional support with rapid transport Watching for signs of shock Pathophysiology of Bleeding Related to Pregnancy Incomplete abortion: Part of the products of conception remains in the uterus. Vaginal bleeding will be continuous. Watch for signs and symptoms of shock. Start an IV line of normal saline. Consult medical control. Collect all products of conception. Pathophysiology of Bleeding Related to Pregnancy Missed abortion: The fetus dies during the first 20 weeks of gestation but remains in utero. Provide emotional support and transport. On examination: The uterus feels like a hard mass. The fetal heartbeat cannot be heard. Pathophysiology of Bleeding Related to Pregnancy Septic abortion: The uterus becomes infected following abortion. History includes fever and bad-smelling vaginal discharge after abortion. Physical examination shows fever and abdominal tenderness. Pathophysiology of Bleeding Related to Pregnancy Septic abortion (cont’d) Prehospital management includes: Establishing an IV line of normal saline Administering 100% supplemental oxygen ECG monitoring Rapid transport Fluid administration Pathophysiology of Bleeding Related to Pregnancy Third-trimester bleeding Greatest danger of hemorrhage A large volume of blood is present. Compensatory mechanisms function as a result of pregnancy. Pathophysiology of Bleeding Related to Pregnancy Ectopic pregnancy The ovum implants somewhere besides uterus. The patient usually presents with: Severe abdominal pain Hypovolemic shock Should be considered for all female patients of child-bearing age with severe lower abdominal pain. Treat for shock and provide rapid transport. Pathophysiology of Bleeding Related to Pregnancy Abruptio placenta Premature separation of the placenta from the uterine wall © Jones & Bartlett Learning. Pathophysiology of Bleeding Related to Pregnancy Abruptio placenta (cont’d) Patient will report: Sudden onset of severe abdominal pain No longer feeling the fetus moving Vaginal bleeding with dark red blood Pathophysiology of Bleeding Related to Pregnancy Abruptio placenta (cont’d) Physical examination may reveal: Signs of shock Tender abdomen and rigid uterus Absent fetal heart sounds Pathophysiology of Bleeding Related to Pregnancy Placenta previa Placenta is implanted low in the uterus and obscures the cervical canal. © Jones & Bartlett Learning. Pathophysiology of Bleeding Related to Pregnancy Placenta previa (cont’d) Painless vaginal bleeding with bright red blood. Uterus is soft and nontender. Assessment of Bleeding Related to Pregnancy Try to determine the nature of the bleeding. Use OPQRST to elaborate on the chief complaint of labor pain. Identify changes in orthostatic vital signs. Look for positive Grey Turner or Cullen sign. Management of Bleeding Related to Pregnancy Keep the woman lying on her left side. Administer 100% supplemental oxygen. Provide rapid transport. Start an IV line of normal saline. Obtain an ECG and baseline vital signs. Loosely place trauma pads over the vagina. Cephalic Presentation The newborn’s head is overly extended, creating a face presentation at birth. Brow presentation Occiput-posterior presentation Military presentation Cephalic Presentation If the newborn’s head cannot be externally rotated or the delivery cannot be completed: Support the woman and fetus. Provide rapid transport. Breech Presentations A different part of the body besides the head leads the way through the birth canal. Types: Frank Incomplete Complete © Jones & Bartlett Learning. Breech Presentations If buttocks are presenting and delivery is imminent: Position the woman with buttocks at edge of bed or stretcher, legs flexed. Allow newborn’s buttocks and trunk to deliver spontaneously. Once the legs are clear, support the body. Lower the newborn slightly. Breech Presentations Once the hairline is spotted, grasp the newborn’s ankles and lift upward. If the head does not deliver within 3 minutes, the newborn may suffocate. Do not try to forcibly pull the newborn out. Breech Presentations Other presentations are rare. Footling breech Transverse presentation In abnormal presentations, do not attempt delivery in the field. © Jones & Bartlett Learning. Shoulder Dystocia Difficulty in delivering the shoulders. If the shoulders cannot clear the birth canal, the fetus cannot breathe. A major concern for the newborn is brachial nerve plexus damage. Shoulder Dystocia McRoberts maneuver Hyperflex the woman’s legs tightly to the abdomen. You may need to apply pressure to the lower abdomen and gently pull on the fetus’s head. Nuchal Cord The umbilical cord becomes wrapped around the newborn’s neck during delivery. Slip a finder under the cord and gently attempt to slip it over the shoulder and head. If unsuccessful, cut the cord. Prolapsed Umbilical Cord The cord emerges before the fetus. Shuts off the oxygenated blood supply from the placenta Leads to fetal asphyxia © Jones & Bartlett Learning. Prolapsed Umbilical Cord Keep the woman supine with hips elevated. Administer 100% supplemental oxygen. Have the woman pant with each contraction. Gently push the presenting part back up the vagina until it no longer presses on the cord. Prolapsed Umbilical Cord Maintain pressure while another paramedic covers the exposed cord with dressings. Maintain that position throughout urgent transport. Postpartum Hemorrhage Can be either early or late hemorrhage. Early: bleeding within 24 hours of delivery Late: bleeding occurring from 24 hours to 6 weeks after delivery Blood loss exceeds 500 mL during first 24 hours after birth. Postpartum Hemorrhage Causes of postpartum hemorrhage include: Lacerations Prolonged labor or multiple baby delivery Retained products of conception Multiple pregnancy Placenta previa Full bladder Postpartum Hemorrhage Continue uterine massage. Encourage the woman to breastfeed. Notify the receiving facility of status. Transport immediately. Add a large-bore IV line en route. Do not pack dressings into the vagina. Trauma and Pregnancy Trauma is the leading cause of maternal death in the United States. Abdominal trauma occurs from the same mechanisms in pregnant women as nonpregnant women. Pathophysiology and Assessment Considerations Anatomic changes are important in trauma. Abdominal contents compress into the upper abdomen. The diaphragm elevates by about 1.5 inches. The peritoneum maximally stretches. Pathophysiology and Assessment Considerations First trimester The uterus is well protected and rarely damaged from trauma. Second and third trimesters The uterus extends into the abdomen and becomes more vulnerable to trauma. Pathophysiology and Assessment Considerations Pregnant patients will have different signs or responses to trauma. It may be more difficult to interpret tachycardia. Signs of hypovolemia may be hidden. The patient has a higher chance of bleeding to death in case of pelvic fractures. A respiratory rate less than 20 breaths/min is not adequate. Considerations for the Fetus and Trauma Fetal injury can occur from: Rapid deceleration Impaired fetal circulation If a pregnant woman has massive bleeding, maternal circulation will reroute blood from the fetus. Considerations for the Fetus and Trauma The fetal heart rate is the best indication of fetal status after trauma. A normal fetal heart rate is between 120 and 160 beats/min. A rate slower than 120 beats/min indicates fetal distress and a dire emergency. Management of the Pregnant Trauma Patient You can only treat the woman directly. Transport a pregnant woman on her left side if no spinal injury is suspected. © Jones & Bartlett Learning. Management of the Pregnant Trauma Patient Ensure an adequate airway. Administer oxygen. Assist ventilations when needed and provide a higher-than-usual minute volume. Control external bleeding and splint fractures. Management of the Pregnant Trauma Patient Start one or two IV lines of normal saline. Transport the patient in the lateral recumbent position. Management of the Pregnant Trauma Patient Maternal cardiac arrest Provide CPR and ALS as you would for any other trauma patient. CPR and ventilator support may keep the fetus viable, even if the mother is already dead. Neonate Arrival of the Newborn Obtain patient history and prepare environment. Minimum needs: Warm, dry blankets Bulb syringe Two small clamps or ties A pair of clean scissors Arrival of the Newborn If delivery in ambulance: Use blankets. Confirm ABCs. Place newborn on mother’s chest. Suction mouth, then nose. Keep newborn at level of © Jones & Bartlett Learning. mother. Arrival of the Newborn Clamp and cut the umbilical cord. Primary survey simultaneous with treatment interventions Examine skin, head, and eyes for irregularities. Inspect umbilical cord for abnormalities. Arrival of the Newborn Newborns are at risk for hypothermia. Place on prewarmed towels or radiant warmer Dry the head and body thoroughly Remove wet towels and cover with a dry towel Cover the head with a cap Arrival of the Newborn All babies are cyanotic right after birth If the newborn remains vigorous and begins to turn pink in the first 5 minutes: Maintain ongoing observation. Continue thermoregulation with direct skin-to-skin contact with mother. The Apgar Score Helps record condition at birth If score is less than 7, redo every 5 minutes until 20 minutes after birth. Algorithm for Neonatal Resuscitation Follow current neonatal resuscitation guidelines. First 60 seconds: Initial steps, reevaluation, and ventilation should occur. Additional oxygen may be necessary if the target preductal oxygen saturation value has not been reached. Algorithm for Neonatal Resuscitation If the newborn’s pulse rate is apneic or less than 100 beats/min, begin PPV. Begin chest compressions if the newborn’s pulse rate is less than 60 beats/min. If ventilation and chest compression do not improve the bradycardia, administer epinephrine preferably via IV line. Resuscitation Algorithm for Neonatal Reprinted with permission. Web-based Integrated 2015 American Heart Association Guidelines for CPR & ECC. Part 13: neonatal resuscitation. © 2015 American Heart Association, Inc. Bradycardia Most frequently occurs due to inadequate ventilation Often responds to effective PPV Morbidity and mortality are determined by: Underlying cause How quickly it is corrected Bradycardia Assessment and management Heart rate less than 100 beats/min: Provide PPV If still less than 60 beats/min: Begin chest compressions If still less than 60 beats/min: Administer epinephrine. Repeat every 3 to 5 minutes for persistent bradycardia. Meconium-Stained Amniotic Fluid Carries a high risk of morbidity If newborns inhale meconium-stained amniotic fluid, airway may become plugged. Atelectasis Persistent pulmonary hypertension Hypoxemia Aspiration pneumonitis Pneumothorax Meconium-Stained Amniotic Fluid To decrease risk of persistent pulmonary hypertension: Ensure a clear airway. Keep newborn warm. Minimize stimulation. Administer supplemental oxygen when necessary. Meconium-Stained Amniotic Fluid Assessment and management If depressed: Start PPV. Intubation and suctioning may be required to remove an obstruction. Meconium-Stained Amniotic Fluid Assessment and management (cont’d) If the newborn is not responding well to the care outlined in the neonatal resuscitation algorithm: Suspect airway occlusion or pneumothorax. Minimize hypothermia. Reassess frequently. Premature and Low Birth Weight Infants Premature: Delivered before 37 weeks of gestation Increased mortality Associated morbidities © American Academy of Orthopaedic Surgeons. Premature and Low Birth Weight Infants Low birth weight—newborns weighing less than 5.5 lb (2,500 g) Morbidity and mortality are related to degree of prematurity. Premature and Low Birth Weight Infants To determine prematurity, rely on: Physical features Information from family about gestational dating Information related to maternal or fetal complications Premature and Low Birth Weight Infants To optimize survival in the field: Provide cardiorespiratory support. Provide thermoneutral environment. Use only minimum pressure necessary to move chest when providing PPV. Premature and Low Birth Weight Infants Assessment and management Management focuses on: Clearing airway Gentle stimulation Administering supplemental oxygen and PPV if needed Providing chest compressions Maintaining a warm environment Seizures in the Newborn Suggest the presence of neurologic disorder Usually related to an underlying abnormality Prolonged seizures may cause brain injury. Seizures in the Newborn Types of seizures: Subtle seizure Tonic seizure Spasm Myoclonic Seizures in the Newborn Assessment and management Evaluate prenatal and birth history. Perform a careful physical exam. Obtain vital signs and oxygen saturation. Provide additional oxygen, assisted ventilation, blood pressure evaluation, and IV access. Seizures in the Newborn Assessment and management (cont’d) If blood glucose level is less than 40 mg/dL, give an IV bolus of 10% dextrose solution. Monitor respiratory status and oxygen saturation. Maintain normal body temperature. Keep family informed as you transport. Hypoglycemia Blood glucose level of less than 45 mg/dL May result in: Seizures Brain damage Hypoglycemia Risk factors: Disorders related to decreased glycogen stores Increased use of glucose Vomiting Common in newborns Persistent or bilious vomiting in first 24 hours may indicate: Upper digestive tract obstruction Increased intracranial pressure Vomitus aspiration may cause respiratory insufficiency or airway obstruction. Vomiting Sudden, unexpected, and forceful vomiting may occur in conjunction with: Asphyxia Meningitis Hydrocephalus Vomiting Assessment and management Stomach may be distended. May have a fever or hypothermia May also note: Temperature instability Apnea/bradycardia Abdominal tenderness/guarding Vomiting Assessment and management (cont’d) Start management with ABCs. Consider decompressing the stomach. Do not administer antiemetics. May need fluid resuscitation if dehydrated Place newborn on the side when transporting. Diarrhea Assessment and management Estimate: Number and volume of loose stools Decreased urinary output Degree of dehydration Patient management begins with ABCs. Neonatal Jaundice Considered pathologic when: Clinically visible in first 24 hours Serum bilirubin increases more than 5 mg/dL/d. Bilirubin exceeds 12 mg/dL. Conjugated bilirubin makes up greater than 20% of total serum bilirubin concentration. Clinical jaundice persists for more than 1 week in full-term infants or for more than 2 weeks in preterm infants. Neonatal Jaundice Assessment and management Transport is essential. Start on IV fluids if neonate shows significant clinical jaundice. Communicate with medical control. Thermoregulation Thermoregulation limited in newborns Average normal temperature of newborn—37°C (99.5°F) Range for neonate—36.6°C to 37.2°C (97.9°F to 99°F) © Jones & Bartlett Learning. Fever Rectal temperature greater than 38°C (100.4°F) Newborn may not always present with fever in an illness or infection. May be caused by overheating or dehydration Fever Limited ability to control their temperature. Signs and symptoms include: Irritability Somnolence Decreased feeding Warm to touch Fever Assessment and management Examine for rashes. Obtain history. Note increased respiratory rate and work of breathing. Courtesy of Centers for Disease Control and Prevention. Fever Assessment and management (cont’d) Obtain vital signs and ensure adequate oxygenation and ventilation. Cool the patient. Pathophysiology of Congenital Heart Disease Congenital heart disease (CHD) Most common birth defect Can present with varying degrees of cardiorespiratory compromise Pathophysiology of Congenital Heart Disease Noncyanotic disease (pink defects) Oxygenated blood is shunted from the left side of the heart to the right side. Called a left-to-right shunt Pathophysiology of Congenital Heart Disease Atrial septal defect Abnormal opening exists in the wall separating the atrial chambers of the heart. Ventricular septal defect: Abnormal opening exists in the wall separating the right and left ventricles. Pathophysiology of Congenital Heart Disease Patent ductus arteriosus: Ductus arteriosus does not close after birth. If left untreated, patient could develop heart failure. © Jones & Bartlett Learning. Pathophysiology of Congenital Heart Disease Cyanotic disease Deoxygenated blood from the right side of the heart mixes with the left side. Known as a right-to-left shunt Pathophysiology of Congenital Heart Disease Pulmonary stenosis Pulmonic valve near right ventricle becomes damaged. Patient will present with: Jugular vein distention Cyanosis Right ventricular hypertrophy Pathophysiology of Congenital Heart Disease Truncus arteriosus Pulmonary and aorta arteries are combined. Increases blood flow into the lungs Will require surgical intervention © Jones & Bartlett Learning. Pathophysiology of Congenital Heart Disease Tricuspid atresia Tricuspid valve is missing. Results in an undersized or absent right ventricle Will have decreased blood flow into the lungs © Jones & Bartlett Learning. Pathophysiology of Congenital Heart Disease Hypoplastic left heart syndrome Left side of heart is completely underdeveloped. Heart transplant may be needed if a surgical procedure cannot be performed or fails. Pathophysiology of Congenital Heart Disease Tetralogy of Fallot (ToF) Combination of four heart defects: Ventricular septal defect Pulmonary stenosis Right ventricular hypertrophy Overriding aorta Open heart surgery is required. Pathophysiology of Congenital Heart Disease Transposition of the great arteries (TGA) Blood goes to the lungs, then returns to the lungs. Blood from the body goes to the heart and returns to the body. © Jones & Bartlett Learning. Pathophysiology of Congenital Heart Disease Total anomalous pulmonary venous return (TAPVR) Four pulmonary veins connect to the right atrium instead of the left atrium. Results in diminished oxygen and increased load on right ventricle Assessment and Management of Congenital Heart Disease Rapid detection and transport are mandatory. Communication with medical control is critical. Pediatrics The Respiratory System Smaller tidal volume, double metabolic oxygen demand Smaller functional residual capacity Faster breathing The Cardiovascular System Children rely on pulse rate to: Compensate for decreased oxygenation Maintain cardiac output Primary Survey Use the PAT to form a general impression. Used with permission of the American Academy of Pediatrics, Pediatric Education for Prehospital Professionals, © American Academy of Pediatrics, 2000. Primary Survey Appearance Often the most important factor in determining the severity of illness, need for treatment, and response to therapy © Photos.com/Getty. Appearance (cont’d) Primary Survey life-support interventions and transport. A child with a grossly abnormal appearance requires immediate Courtesy of Health Resources and Services Administration (HRSA), Maternal and Child Health Bureau (MCHB), Emergency MedicalServices for Children (EMSC) Program. Primary Survey Work of breathing Reflects attempt to compensate for abnormalities in oxygenation, ventilation Hands-on Primary Survey Airway Determine whether airway is open and patient has adequate chest rise with breathing. If there is potential obstruction, position airway and suction as necessary. Hands-on Primary Survey Breathing Calculate the respiratory rate. Auscultate breath sounds. Check pulse oximetry. Hands-on Primary Survey Circulation Integrate information from PAT. Listen to the heart or feel pulse for 30 seconds. Double the number to get pulse rate. After checking the pulse rate, do a hands-on evaluation of skin CTC. Hands-on Primary Survey Disability Use the AVPU scale or Pediatric Glasgow Coma Scale to assess level of consciousness. Assess pupillary response. Evaluate motor activity. Combine this information with PAT to determine neurologic status. Hands-on Primary Survey Hands-on Primary Survey Exposure Perform a rapid exam of the entire body. Avoid heat loss, especially in infants. Cover child as soon as possible. Transport Decision Transport immediately for trauma with: Serious MOI Physiologic abnormality Significant anatomic abnormality Unsafe scene Attempt vascular access en route. Secondary Assessment Attempt to take the child’s blood pressure on the upper arm or thigh. Minimal systolic blood pressure = 70 + (2 × Age in years) Secondary Assessment Pediatric pain When assessing pain: Consider developmental age. Discuss child’s pain with caregivers. Use pain scales with pictures. Secondary Assessment Pediatric pain (cont’d) Consider pharmacologic methods: Acetaminophen Opiates Benzodiazepines Nitrous oxide Weigh benefits and risks of administration. Reassessment Includes the following: PAT Patient priority Vital signs Assessment of interventions Reassessment of focused exam areas Respiratory Arrest, Distress, and Failure Determining severity of illness will indicate urgency of treatment and transport. Obtain SAMPLE history on scene or during transport. Shock Compensated shock Critical abnormalities of perfusion Body is able to maintain adequate perfusion to vital organs. Intervention is needed to prevent child from decompensating. Shock Decompensated shock State of inadequate perfusion Child will be profoundly tachycardic and show signs of poor peripheral perfusion. Hypotension is a late and ominous sign. Start resuscitation on scene. Hypovolemic Shock Most common cause of shock in infants and young children Loss of volume due to illness or trauma Early signs may include: Tachycardia Pale or mottled skin Cool extremities Hypovolemic Shock Management Position of comfort Supplemental oxygen Keep the child warm. Direct pressure to stop external bleeding Volume replacement Hypovolemic Shock With compensated shock, you can attempt IV or intraosseous (IO) access en route. © Jones & Bartlett Learning. Bradydysrhythmias Reprinted with permission. Web-based Integrated 2015 American Heart Association Guidelines for CPR and ECC—Part 12: Pediatric Advanced Life Support © 2015 American Heart Association, Inc. Reprinted with permission. Web-based Integrated 2015 American Heart Association Guidelines for CPR and ECC—Part 12: Pediatric Advanced Life Support © 2015 American Heart Association, Inc. Pediatric Cardiac Arrest Algorithm Traumatic Brain Injury (TBI) Head trauma is common in childhood. Small number of children who appear to be at low risk may have an intracranial injury. Evaluate any child with head injury for signs of potential abuse. Traumatic Brain Injury (TBI) Epidural hematoma Subdural hematoma Hemorrhage into space Hemorrhage into space between the dura and skull between dura and Child abuse accounts for arachnoid membranes significant number of cases Suspect abuse until proven in infants and children. otherwise. Traumatic Brain Injury (TBI) Management includes stabilization of airway, breathing, and circulation. Perform frequent neurologic checks. Brief Resolved Unexplained Event (BRUE) Episode during which an infant: Becomes pale or cyanotic; Chokes, gags, or has an apneic spell; or Loses muscle tone Causes range from benign to serious diagnoses. Brief Resolved Unexplained Event (BRUE) Provide life support with signs of cardiorespiratory compromise or altered mental status. Transport all infants with a history of BRUE. Pediatric Trauma Emergencies Anatomy and physiology make injury patterns and responses different from those seen in adults. Developmental stage will affect response. Pathophysiology of Traumatic Injuries Blunt trauma is the MOI in most pediatric injury cases. Less muscle and fat mass leads to less protection against forces transmitted. Pathophysiology of Traumatic Injuries Falls are common. Injury will reflect child’s anatomy and height of fall. Falls from a standing position usually result in isolated long bone injuries. High-energy falls result in multisystem trauma. Injuries from bicycle handlebars typically produce intraabdominal compression injuries. Pathophysiology of Traumatic Injuries Motor vehicle crashes can result in a variety of injury patterns depending on restraints and position in car. For unrestrained passengers, assume multisystem trauma. Suspect spinal fractures with chest or abdominal bruising in a seat belt pattern. Assessment and Management of Traumatic Injuries Begin with a thorough scene size-up. Use PAT to form a general impression. If findings are grossly abnormal, move to ABCs. Initiate life support interventions. Assessment and Management of Traumatic Injuries Pneumothorax may be present with penetrating trauma of the chest or upper abdomen. Perform needle decompression. Signs and symptoms may include: Tachycardia Jugular vein distention Pulsus paradoxus Assessment and Management of Traumatic Injuries © American Academy of Orthopaedic Surgeons. © American Academy of Orthopaedic Surgeons. Assessment and Management of Traumatic Injuries Any trauma patient should be considered at risk for developing shock. Once ABCs are stabilized, continue assessment of disability with AVPU. Assessment and Management of Traumatic Injuries Place a cervical collar, and immobilize on a long backboard as indicated. Perform rapid exam to identify all injuries. Cover the child with blankets. Treat any fractures. Transport Considerations Some traumas are load-and-go because of severe injuries and unstable condition. For these situations: Perform lifesaving steps on scene or en route. Transfer quickly per local trauma protocols. Transport Considerations All trauma victims with suspected spinal injury require spinal stabilization. Do not place a collar that is too big on a child. © Mark C. Ide. Geriatrics Geriatric Anatomy and Physiology Aging process begins in the late 20s, early 30s. Organ and tissue aging may be accelerated by: Genetics Preexisting disease Diet and activity levels Toxin exposure Geriatric Anatomy and Physiology Aging rate varies from person to person. Decrease in functional capacity is normal. Courtesy of the National Cancer Institute. Changes in the Respiratory System Respiratory capacity undergoes a large reduction with age. Vital capacity decreases, and residual volume increases. Changes in blood flow distribution in the lungs result in declining PaO2. Changes in the Respiratory System Decreased sensitivity/CNS response to arterial blood gases changes Limited lung volume and maximal inspiratory pressure Limited chest expansion Changes in the Respiratory System The ability to modify respiratory rate/tidal volume in response to changes is limited. Defense mechanisms are less effective. Changes in the Cardiovascular System The cardiovascular system decreases in efficiency with age. Heart hypertrophies Changes in the Cardiovascular System Arteriosclerosis adds to systolic hypertension Diabetes Atherosclerosis Renal compromise © Jones & Bartlett Learning. Changes in the Cardiovascular System Vascular stiffening occurs as collagen and elastin production changes with age. In aortic sclerosis, the aortic valve thickens from fibrosis and calcification. Peripheral vessel walls lose elasticity. Changes in the Cardiovascular System The heart’s electrical conduction system undergoes changes over time. The number of pacemaker cells decreases with age. Bradycardia can occur. Failure of the SA node can lead to atrial dysrhythmias. Changes in the Cardiovascular System Aging makes the cardiovascular system more vulnerable to dysfunction. Potential cardiac compromises should be recognized and treated quickly. Changes in the Nervous System A neurologic exam will reflect aging-related changes in the nervous system. The brain decreases in weight and volume. Changes in the Nervous System As mental function declines, so does the regulation of: Respiratory rate and depth Pulse rate Blood pressure Hunger and thirst Temperature Sensory Changes Most sensory organs decline with age. Do not assume geriatric patients are deaf or blind. Vision changes may begin as early as 40 years. Cataracts Glaucoma Sensory Changes Visual acuity decreases even without disease Difficulty seeing at night Inability to adjust to rapid changes Presbyopia (far-sightedness) Difficulty differentiating between colors Sensory Changes Gradual hearing loss is common. Hearing aids: Consist of a microphone and an amplifier May fit in the ear canal Mainly battery operated © Maxx-Studio/Shutterstock. Sensory Changes Ménière disease: hearing-related impairment Onset in middle age Symptom cycles last several months: Vertigo Hearing loss and tinnitus Pressure in ear Sensory Changes Other sensory changes: Changes in appetite Decrease in sense of touch Sense of smell last to diminish Sensory Changes Changes may make it difficult to produce speech that is loud enough, clear, and well spaced. Sense of body position may become impaired. Changes in the Digestive System Changes may be first noted in the mouth. Fewer taste buds Reduction of saliva Dental loss Changes in the Digestive System Ill-fitting dentures may cause risk of: Choking Heartburn Abdominal pain Changes in the Digestive System Gastric secretions are reduced. Heart burn Indigestion Acid reflux Changes in the Digestive System Slight changes in small and large bowel functions from aging The rectal sphincter decreases in size and strength, causing incontinence. Slowing peristalsis causes increased constipation. Changes in the Digestive System Constipation also caused or worsened by: Some medications Diet changes Decreased physical activity Can cause straining bowel movements Changes in the Digestive System Hepatic enzyme changes: some activity declines and others’ increases. Enzyme systems that detoxify drugs decline. Taking numerous medications increases the risk for hepatic damage or drug toxicity. Changes in the Renal System Kidneys decline in weight with age. Loss of functioning nephron units Small effective filtering surface Changes in the Renal System Aging kidneys respond slowly to sodium deficiency, causing electrolyte imbalance. Rapid development of severe dehydration Exacerbated by decreased thirst mechanism Changes in the Renal System At risk of overhydration with large sodium loads Lower glomerular filtration rate. The patient is prone to serious or lethal hyperkalemia if he or she becomes acidotic or the potassium load is increased. Changes in the Endocrine System Greater risk for developing type 2 diabetes. Difficulty metabolizing carbohydrates Comorbid disorders (medications can affect glucose metabolism) Changes in the Endocrine System Increase in ADH as people age: Causes electrolyte and fluid imbalances May present as pedal or other peripheral edema Menopause: decrease in hormone secretion Changes in the Immunologic System Every immune system function is affected by aging. More prone to infection and secondary complications. Infections manifest differently in older people. Changes in the Integumentary System Wrinkling and resiliency loss in skin Thinner, drier, less elastic, more fragile skin Subcutaneous fat thinner, bruising Decreasing elastin and collagen Skin more prone to tenting in skin turgor tests Changes in the Integumentary System Sebaceous glands produce less oil, so the skin is drier. Decreased sweat gland activity Thinner or no hair Gray or white hair Changes in the Integumentary System Blood vessels are affected by atherosclerosis, providing less oxygenated blood to the skin. Production of new skin takes longer Thin and brittle fingernails and toenails Homeostatic and Other Changes Homeostatic capabilities decrease with age. Thirst mechanism Temperature regulating mechanism Blood glucose regulatory system Changes in the Musculoskeletal System Decrease in bone mass in men and women Causes brittle, easily breakable bones Joint problems Tendons and ligaments lose elasticity Synovial fluids thicken Cartilage decreases Changes in the Musculoskeletal System Height decreases, posture changes. Arthritic joints increase. Muscle mass and strength decrease. Muscles atrophy from prolonged immobility. Changes in the Musculoskeletal System More susceptible to bone fractures from falls Falls more likely to occur Difficulty with tasks requiring fine motor coordination or hand and finger strength Primary Assessment Use GEMS diamond to form a general impression. G—Geriatric patient E—Environmental assessment M—Medical assessment S—Social assessment Primary Assessment Primary Assessment Airway and breathing Geriatric patients are predisposed to airway problems. Ensure airway is not obstructed. Anatomic changes lessen effective breathing. Treat with oxygen as soon as possible. Primary Assessment Circulation If circulation is compromised, the patient has fewer reserves in a circulatory crisis. Lower heart rate Radial pulse difficult to find Heart rhythm issues and irregular pulse Treat with oxygen as soon as possible. Primary Assessment Transport decision Determine life-threatening conditions. Treat to the best of your ability. Provide transport to priority patients. Older people will easily decompensate. History Taking Use good communication skills. Thoroughly take a history if possible. Listen to the patient; wait for answers. Explain the plan. Preserve the patient’s dignity. History Taking Comprehensive history includes: Chief complaint Present illness or injury Pertinent medical history Current health care status and needs History Taking Pertinent medical history includes: Current cardiovascular health Exercise tolerance Diet history Medications Smoking and drinking habits Sleep patterns Other intrinsic and extrinsic factors History Taking Determining the chief complaint may be difficult because patients might: Believe symptoms are just part of getting old Ignore legitimate symptoms because they don’t want to be labeled a hypochondriac Underreport symptoms or report vague symptoms History Taking If the chief complaint seems trivial, use a standard list of screening questions to evaluate major organ systems functions. The list should be designed to evaluate major organ systems functions. Follow up on any positive answers. History Taking After deducing the chief complaint, conduct a history of present illness. May be difficult to separate from chronic problems How does this differ from last week? What happened today to make you call? History Taking Obtain a detailed history of medications. Have the patient list by name, dosing and frequency, and provider. Obtain permission to bring medications to the hospital. Secondary Assessment Adjust usual methods to fit older patients. Limit physical manipulation. Be aware of body temperature. Only remove clothing as necessary for inspection and palpation, and re-cover immediately. Secondary Assessment Postural BP changes vary with older people. Marked BP changes and pulse rate—possible hypovolemia or overmedication Normal BP tends to be higher. Secondary Assessment Observe respiratory rate. Tachypnea can indicate acute illness. Take lung sounds in all fields. Listen for carotid bruits and note JVD. Note any dentures. Reassessment Reassess often: conditions deteriorate quickly. Repeat primary assessment. Reassess vital signs. Reassess the patient’s complaint. Recheck interventions. Treat changes. Polypharmacy and Medication Noncompliance Polypharmacy becomes problematic when medications interact. The chance of being hospitalized increases with the number of medications. Medications may not be received because of caregiver theft. Polypharmacy and Medication Noncompliance Noncompliance includes: Failure to fill prescription Administering medication improperly Taking inappropriate medication Polypharmacy and Medication Noncompliance Other issues: Taking medication prescribed by different doctors who don’t know the full medication regimen Taking medication prescribed for someone else Difficulty understanding the drug regimen Difficulty opening medication containers Pharmacokinetics Toxic effects of drugs from aging-related alterations in pharmacokinetics Predisposed to reactions by physiologic changes in body systems and composition Medications affecting the CNS are the most common source of adverse reactions. Pharmacokinetics Reduction in nervous system response increases risk of adverse anticholinergic effects. Reduced beta-adrenergic receptor sensitivity causes most bronchodilators to be ineffective. Pharmacokinetics Diuretics, antihypertensive Cause hypotension and orthostatic changes from reduced cardiac output, total body water decrease Decreased glucose tolerance Hyperglycemic effects from diuretics and corticosteroids Pharmacokinetics Pharmacokinetics can be influenced by: Diet Smoking Alcohol consumption Other drug use Pharmacokinetics Dosage often needs to be reduced. Toxic effects present with: Psychiatric symptoms Cognitive impairment © Jones & Bartlett Learning. Geriatric Trauma Emergencies Factors that place an older person at higher risk of trauma: Slower reflexes and reduction in agility Visual and hearing deficits Equilibrium disorders Reduction in agility Geriatric Trauma Emergencies Less favorable outcomes in trauma due to: Changes in homeostatic compensatory mechanisms Aging effects on body systems Preexisting conditions Geriatric Trauma Emergencies Successful treatment when trauma-related blood loss is compensated enough for: Increased pulse rate Increased respirations Adequate vasoconstriction Geriatric Trauma Emergencies Unsuccessful recovery likely if: Decreased respiratory function Impaired renal activity Ineffective vasoconstriction More likely to sustain serious injury due to: Stiffened blood vessels Fragile tissue Demineralized bones Geriatric Trauma Emergencies Most cases involve falls or motor vehicles Falls are associated with: Higher incidence of anxiety and depression Loss of confidence Postfall syndrome Geriatric Trauma Emergencies Falls are divided into two categories: Extrinsic causes Intrinsic causes Risk increases with preexisting gait abnormalities and cognitive impairment. Geriatric Trauma Emergencies Geriatric Trauma Emergencies Home safety assessment by EMS Check for: Clear pathway to and from bathroom Handrails in bathtubs and on steps Loose rugs or other objects on floor Wheelchair ramps with grip tape Geriatric Trauma Emergencies Older adults are far more likely to be fatally injured in a motor vehicle crash. At higher risk for crashes due to: Vision impairment Errors in judgment Underlying medical conditions Geriatric Trauma Emergencies Conditions that can influence outcomes: Cirrhosis Congenital coagulopathy COPD Ischemic heart disease Diabetes mellitus Pathophysiology Head trauma: increased fragility of cerebral blood vessels, enlarged subdural space Hematoma often develops over days or weeks. As intracranial pressure increases: The patient’s state of consciousness becomes depressed. The patient becomes drowsy. Pathophysiology Spinal cord injury and compression Degenerative spinal changes cause arthritic spurs and vertebral canal narrowing. Even a sudden movement of the neck may cause spinal cord injury. Pathophysiology Chest injuries are exacerbated by rib brittleness and stiffening of the chest wall. Abdominal trauma often causes liver injury. Orthopaedic injuries are common results of falls. Pathophysiology Burns have significant risk of morbidity and mortality, especially if: Preexisting medication conditions are present Defense mechanism against infection are weakened. Fluid replacement is complicated by renal compromise. Monitor hydration status. Pathophysiology Heat gain/loss slowed by: Internal temperature Atherosclerotic vessels regulation slows. Slowed circulation Delayed ability to recognize temperature fluctuations Decreased sweat production Pathophysiology Thermoregulation affected Half of hypothermia deaths by: are in older people. Chronic disease Hyperthermia death rates Medications more than double in older Alcohol use adults. Pathophysiology Check for environmental emergencies in extreme hot and cold. You may need to keep the patient compartment at higher-than-normal temperature. Assessment and Management of Trauma Check mechanism of injury. Check for possible medical problem before the trauma. Initial management follows ABCDEs first. Assessment and Management of Trauma Check for rib fracture when assessing breathing. Obtain baseline BP. Normal blood pressure may be hypotension in an older person. Assessment and Management of Trauma Do neurologic status assessment using AVPU scale. Try to obtain complete history of the event from the patient and bystanders. © Jones & Bartlett Learning. Courtesy of MIEMSS. Assessment and Management of Trauma Obtain a list of regular medications, especially those that may affect treatment. Beta blockers Antihypertensives Diabetes medications Assessment and Management of Trauma Conduct secondary assessment, watching for signs of injury to the: Head Cervical spine Ribs and abdomen Long bones Remember the patient’s pain perception may be decreased. Assessment and Management of Trauma Additional treatment based on injuries Be cautious about isotonic solutions. Monitor cardiac rhythm throughout. Preserve temperature. Consider pain medication. Immobilize the cervical spine before transporting.