Summary

This document provides general information on the mechanism of injury, red flags for fractures and the assessment of trauma, including Primary and Secondary Surveys. It also covers the pathophysiology and clinical manifestations of fractures and basic bone healing stages.

Full Transcript

**[Week 11 -- Fractures ]** **[Mechanism of Injury & Red Flags ]** **Mechanism of Injury (MOI)** - "Mechanism of destiny" - MOI: how energy is transferred from the environment onto the body - Provides insight into the potential injuries the team can expect to find - Knowing the pat...

**[Week 11 -- Fractures ]** **[Mechanism of Injury & Red Flags ]** **Mechanism of Injury (MOI)** - "Mechanism of destiny" - MOI: how energy is transferred from the environment onto the body - Provides insight into the potential injuries the team can expect to find - Knowing the pattern of injury can guide the team in predicting and preparing for certain injuries **MOI Red Flags** - - MVCs: - High-speed collisions or rollover accidents - Impact at high velocities (e.g. head-on collision, rear-end, side impact) - Ejection from vehicle (unrestrained driver) - Significant intrusion into the vehicle department - Vehicle occupant death - Motorcyclist/cyclist/pedestrian struck by vehicle - Falls from heigh (\>3m, especially onto a hard surface) - Penetrating injuries (e.g. stab wounds) with unknown trajectory or depth - Gunshot wounds to the torso, head or neck **E-Scooters** - Inc prevalence and severity of injury - Injuries more common in those \< 18 - Injuries more frequent in winter months - Increased risk of long bone fractures, and paralysis compared to cyclists **[Trauma Assessment ]** - To recognize life threatening conditions, identify injuries & set priorities for care - Includes a [PRIMARY & SECONDARY] survey in a team approach - Organized and systematic ***Primary Survey*** - A -- Airway (w simultaneous c-spine protection) - B -- Breathing - C -- Circulation - D -- Disability (neuro status) - E -- Expose/environmental controls (remove clothing to inspect entire body for injuries; keep pt warm) ***Secondary Survey*** - F -- Full set of vital signs, focused adjuncts (incl cardiac monitor, urinary catheter, & G tube), family presence - G -- Give comfort measures (verbal, touch, pharmacologic & non-pharmacologic management of pain) - H -- History & Head-to-toe assessment - I -- Inspect posterior surfaces **Trauma Panel & Diagnostics** - - CBC - Lytes & BUN - Creatinine, Amylase, Lipase - PT, PTT - Blood Type and cross match - Ethanol level - Urinalysis - Urine toxicology screen - ABGs - Lactate - ECG - Imaging (e.g. X-ray, CT, US, FAST) **[Fractures]** - Disruption/break in continuity of the structure of bone - Result from mechanical overload of the bone, when more stress is placed on the bone than it can absorb - Account for high % of traumatic injuries - Some fractures are secondary to disease process (e.g. cancer or osteoporosis weaken bone structure) **Fractures: Classification** - *Open or closed* - **Open:** skin broken, and bone and soft tissue exposed (risk of infection) - **Closed:** skin intact - *Complete or incomplete* - **Complete:** break is completely through bone - **Incomplete:** bone does not completely break (may crack/bend); bone is nondisplaced (e.g. greenstick fracture -- common in kids) - Direction of fracture line (e.g. spiral, transverse, oblique) - *Displaced or nondisplaced* - **Displaced:** two ends separated from one another (two ends are far apart) - **Nondisplaced:** bone is aligned and periosteum is intact **Fractures: Pathophysiology** - When a fracture occurs muscles attached to bone ends are disrupted muscles can undergo spasm and pull the fragments out of position - The periosteum and blood vessels in the cortex and marrow of the fractured bone are disrupted - Soft tissue damage frequently occurs - Bleeding occurs from both the soft tissue and from the damaged ends of the bone - Bone tissue surrounding the fracture site dies creating an intense inflammatory response **Fractures: Clinical manifestations** - - Immediate localized pain; tenderness; muscle spasm - Decreased/loss of function - Deformity/unnatural position - Edema -- inflammatory response - Ecchymosis -- bruising - Crepitation -- bone on bone grinding - Guarding ![](media/image2.png)**Fractures: Bone healing** **Stages of Bone Healing** ---------------------------- ------------------------------------------------------------------------------------------------------------------------------------------------------ **Fracture hematoma** Bleeding creates a hematoma around fracture ends, forming a semisolid clot within 72 hours. **Granulation tissue** Phagocytosis absorbs necrosis, converting the hematoma to granulation tissue, forming the basis for new bone (osteoid) within 3-14 days **Callus formation** Minerals and new bone matrix form an unorganized network of bone (callus) around the fracture, appearing by the end of the second week **Ossification** Callus ossifies from 3 wks to 6 mths, stabilizing the fracture site. Limited mobility may be allowed (some weight bearing) **Consolidation** Callus development continues, closing the gap between bone fragments, which can take up to a year (depending on bone and fracture type) **Remodeling** Excess bone is reabsorbed, restoring the bone's OG strength and shape. Bone remodels in response to stress, with weight bearing gradually introduced **Bone Healing: Factors influencing healing** - - Displacement and site of the fracture - Blood supply to the area - Immobilization and internal fixation devices (e.g., screws, pins) - Inadequate reduction and immobilization - Excessive movement of fracture fragments - Infection, poor nutrition, and systemic disease - Age (e.g.. newborn: 3 wks for a midshaft femur fracture; adult: 20 wks) - Smoking (increases healing time) -- causes inflammation and dec collagen synthesis - **Adequate circulation to the fracture site and adequate fragment immobilization are crucial for effective bone healing** **Fractures: Assessment & Management** *Initial nursing assessment* - Subjective (symptoms, health hx, meds, previous sx) - Objective (signs; system-based assessment -- integumentary, CV (blood flow), neuro, MSK (movement and reflexes), etc.) *Initial Nursing Management* - Treat life-threatening injuries first - Control external bleeding ASAP with direct pressure or sterile pressure dressings and elevation of extremity - Check neurovascular status distal to injury (CHECK PULSES) before and after splinting (or any other interventions) - Elevate injured limb if possible (reduce blood flow mitigating blood loss) - Do not attempt to straighten fractured or dislocated joint (can cause more injury) - Open fractures: do not manipulate protruding bone ends; administer tetanus/antibiotic prophylaxis - Closed fractures: apply ice to area to reduce swelling - Obtain imaging - Mark location of distal pulses to facilitate repeat assessment - Immobilize/splint fracture site (above and below) - Unnecessary movement inc soft tissue damage (may convert a closed fracture to open) ***Neurovascular Assessment*** - As part of initial and on-going assessment; q30mins for first 4H of casting/splinting/traction or other intervention, and then Q3-4H - Peripheral vascular assessment -- colour, temp, cap refill, peripheral pulses, edema - Peripheral neurological assessment -- sensation, motor function and pain (wiggle their toes, fingers, etc.) - Compare bilaterally ***Collaborative Management goals of fracture treatment*** - Anatomical realignment of bone fragments - Immobilization to maintain realignment - Restoration of normal or near-normal function of injured parts *Management**:*** **Open reduction** - Correction of bone alignment through surgery - Often used for compound fractures that are comminuted or accompanied by severe neurovascular injury - Includes internal fixation (and/or external fixation) with use of pins, rods, etc. - Main disadvantages -- risk of infection + complications assoc w anesthesia *Management**:*** **Closed reduction** - Nonsurgical, manual realignment of bone fragments to previous anatomical position - Traction and countertraction manually applied to bone fragments to restore position, length, and alignment *Management**:*** **Traction** - Application of a pulling force to an injured or diseased part of body or extremity, while countertraction pulls in the opposite direction - Must be maintained constantly - **Purpose of traction:** - Prevent or reduce muscle spasm - Immobilize joint or part of body - Reduce a fracture - Two most common types of traction - **Skin traction** - Used for short term tx until skeletal fraction or sx is possible - Tape, boots, or splints, applied directly to skin to maintain alignment, assist in reduction, and help diminish muscle spasms in injured extremity (ex. Buck traction) - Skin traction weight range 2.3-4.5 kg - **Skeletal traction** - In place for longer periods - Used to align injured bones and joints or to tx joint contractures and congenital hip dysplasia - Provides a long-term pull that keeps injured bones and joints aligned - Physician inserts pin or wire into bone, either partially or completely, to align and immobilize injured body part - Skeletal traction weight range: 2-20 kg *Management**:*** **Casts** +-----------------------------------+-----------------------------------+ | **Cast Care for Patients** | | +===================================+===================================+ | **Do's** | **Don'ts** | +-----------------------------------+-----------------------------------+ | - Apply ice directly over | - Get plaster cast wet | | fracture site for first 24 | | | hrs (leave time for swelling) | - Remove padding | | | | | - Elevate extremity above level | - Insert objects inside cast | | of heart for first 48 hrs | | | | - Bear weight on new cast for | | - Move joints above and below | 48 hrs (not all casts are | | cast regularly | weight bearing) | | | | | - Reports signs of possible | - Cover cast with plastic for | | problems to HCPs | prolonged periods | | | | | | - High impact activities | +-----------------------------------+-----------------------------------+ - Temporary circumferential immobilization device - Allows the pt to perform many normal ADLs - Application incorporated joints above and below fracture - Restricts tendon and ligament movement -- assisting w joint stabilization while fracture heals *Management:* **Pharmacological Therapy** - Pain management is key - Analgesics (NSAIDs to reduce inflammation) - Muscle relaxants - Open fractures: tetanus prophylaxis, and antibiotics **[Complications of Fractures ]** - Direct complications - **Problems with bone infection\*** (common in open fractures) - Bone union - Avascular necrosis - Indirect complications - **Compartment syndrome\*** - **Rhabdomyolysis (breakdown of skeletal muscle)\*** - **Venous thromboembolism\*** - **Fat embolism syndrome\*** - Hypovolemic shock ***Infection*** - High incidence in open fractures and soft tissue injuries - Massive or blunt soft tissue injury often has more serious consequences than fracture - Treatment is costly in terms of - Extended nursing and medical care - Time for tx - Loss of patient income - Osteomyelitis may become chronic - Management/prophylaxis antibiotics, wound cleaning and debridement, wound care ***Compartment Syndrome*** - Elevated intra-compartmental pressure within a confined myofascial compartment compromises neurovascular function of tissues within that space - Causes cap perfusion to be reduced below the level necessary for tissue viability - 38 compartments are located in the upper and lower extremities - Swelling compressed blood vessels decrease circulation to that part of the body - Most often occurs in the legs (specific the anterior compartment of the leg) or forearms - Two causes: - Decreased compartment size (resulting from restrictive dressing, splints, casts, etc.) - Increased compartment volume (related to bleeding, edema, etc.) - Ischemia can occur with 4-8 hours after onset - Depends on elevation and pressure - Prolonged ischemia limb no longer viable - If untreated loss of nerve and muscle function, infection, myoglobinuria and renal failure; amputation may be necessary *Compartment syndrome:* **clinical manifestations** - May occur initially or may be delayed for several days - Six Ps are characteristics of impending compartment syndrome - **Paresthesia:** numbness and tingling d/t compression on nerves - **Pain:** distal to injury that is not relieved by opioid analgesics and pain on passive stretch of muscle travelling through compartment - **Pressure:** inc in compartment -- tense skin over area of injury - **Pallor:** coolness and loss of colour of extremity - **Paralysis:** loss of function - **Pulselessness:** diminished/absent peripheral pulses - Red flags: - throbbing pain out of proportion to the initial injury - no relief of pain with opioids - decreased or absent pulse - tense skin over area of injury *Compartment Syndrome: **Management*** - quick identification - Extremity should not be elevated above heart level - Elevation may raise venous pressure and slow arterial perfusion - Application of cold compresses may result in vasoconstriction and rebound vasodilation that may exacerbate compartment syndrome - May be necessary to remove or loosen bandage or split cast - Surgical decompression may be necessary -- fasciotomy (emergency tx) - position limb at level of heart, call MD, prepare for fasciotomy - Fasciotomy: surgically go in and cut across fascia to relieve pressure off nerves, restore circulation ***Rhabdomyolysis*** - Potentially life-threatening syndrome resulting from the breakdown of skeletal muscle fibers w leakage of muscle contents into the circulation - Common cause: crush injuries - Clinical features: nonspecific, elevated CK (helpful to indicate muscle pain), dark reddish0brown urine (first sign; indicates myoglobinuria) - Early complications include severe hyperkalemia that causes cardiac arrhythmia and arrest - The most serious late complication is **acute renal failure** - Myoglobin released from damaged muscle cells precipitates as a gel-like substance which causes obstruction in renal tubules - Large amts of myoglobin acute tubular necrosis acute renal failure - Management: fluid resuscitation and/or dialysis ***Venous Thromboembolism*** - Veins of lower extremities and pelvis are highly susceptible to thrombosis - Precipitating factors - Incorrectly applied cast or traction - Local pressure on a vein - Immobility - Age - Underlying health conditions (e.g. cancer) - Meds (e.g. estrogen-containing oral contraceptives) - Instruct pt to wear compression stockings (if indicated) - Prophylactic anticoagulant drugs may be ordered (e.g. LMW heparin) - Management may include anticoagulants and thrombolytics ***Fat embolism syndrome*** - Presence of systemic fat globules from fracture that are distributed into tissues and organs after a traumatic skeletal injury - Causes: fractures of long bones, ribs, tibia, & pelvis (most common); total joint replacement, spinal fusion, liposuction, crush injury, and bone marrow transplantation - Fatal in 5-15% of patients - 2 theories related to etiology: - Mechanical theory -- fat is released from the marrow of injured bone enters systemic circulation - Biochemical theory -- hormonal changes caused by trauma/sepsis stimulate release of free fatty acids which form into emboli *Fat Embolism Syndrome:* **Clinical Manifestations** - Usually 24-48 hrs after injury - Clinical course may be rapid and acute - Hypoxemia, dyspnea, tachypnea (early findings) - Changes in LOC - Petechial (pinpoint) rash (intravascular thromboses caused by hypoxemia) - Fat globules transported to lungs cause s&s of acute respiratory distress syndrome (ARDS) - Chest pain, tachypnea, dyspnea, tachycardia, decreased PaO2 *Fat Embolism Syndrome:* **Diagnosis & Management** - Diagnosis is made clinically - Diagnostic abnormalities: fat cells in blood; decreased PaO2, dec plt count; CXR may reveal areas of lung infiltration - Treatment = prevention - Careful immobilization of a long bone fracture is most important factor in prevention - Management is mainly supportive -- fluid resuscitation, oxygenation, encouraging coughing and deep breathing **[Pelvic Fractures ]** - MOI is usually trauma (e.g. MVCs, falls, etc.); 2/3rds of all pelvic fractures occur in pedestrians struck by vehicles - Pelvic fractures co-exist in about 30% of all multiple trauma injuries - Classified as stable or unstable (disruption of ring = unstable fracture) - Clinical manifestations: bruising around flank, groin or perineum (may indicate internal bleeding); leg length discrepancy - Up to 50% mortality (hemorrhage is significant cause) - Bind an unstable pelvic fracture: pelvic binders, sheets (whatever is initially available) - Helps to control hemorrhage -- prevent disruption of vessels and mass hemorrhage *Pelvic Fractures:* **Collaborative Management** - Thorough trauma nursing assessment - High flow O2 - Frequent monitoring of vital signs - 2 large bore IV sites, fluid boluses, cross match - Splint pelvis, immobilize spine & legs - **No foley** catheter if blood at urinary meatus **[Hip Fractures ]** - Leading cause of morbidity and mortality among old adults - Numerous studies have associated low bone mass and increasing age with an increased risk of hip fracture - Lower bone density contributes to osteoporosis - More than 90% of hip fractures are caused by falling - Described based on their location (e.g. intertrochanteric, femoral neck, subtrochanteric) - Femoral neck is a common place for fractures especially in older adults - Clinical manifestations: external rotation, muscle spasm, shortening of the affected extremity; severe pain and tenderness at fracture site *Hip Fractures:* **Collaborative Management** - Initial immobilization until surgery - Assess for neurovascular complications (assess extremity for colour, temp, cap refill, distal pulses, edema, sensation, motor function and pain) - Surgery - Pain management -- analgesics, muscle relaxants, traction - Post op care: monitor VS, neurovascular status, ins and outs, respiratory interventions (e.g. deep breathing and coughing), pain management, observe the dressing and incisions for signs of bleeding and infection - Implement measures to avoid dislocation (e.g. avoid extremes in flexion, elevate toilet seats and chairs) - Provide patient education - Physiotherapy & rehabilitation **[Trauma Tips ]** - - Assessments: - Primary & secondary survey - Reassess after each intervention - Continuous HTT reassessment will provide early detection of changes - Accurate ins and outs - Interventions: - Focus on stabilizing A,B, C, D - OXYGEN - GCS \< 8 = intubate - No foley if blood at urinary meatus - No NG tube with facial trauma or suspected basal skull \$ - Tetanus prophylaxis - Antibiotics for compound fractures, CSF leak or penetrating injury (prevent secondary osteomyelitis, meningitis, peritonitis) - Blood products - Splints - Wound/burn dressings - Pain management - Other - Succinct report/documents - Transfer early (to trauma centre if indicated) - Provide family support - Provide travel directions family

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