Chapter 16 Learning Objectives PDF
Document Details
Uploaded by ForemostAntimony5563
Passaic County Community College
Tags
Summary
This document provides learning objectives for a chapter on various aspects of immunity. The objectives include defining key terms, identifying components of the immune system, describing different aspects of the inflammatory response, and examining the functions of various molecules.
Full Transcript
Learning Objectives: Chapter 16 Learning objectives serve as a guide for your learning. After successfully completing the reading of Chapter 16 (pgs. 439-448, Fig 16.8, 452-456, Fig 16.10a) in your text, you should be able to execute the objectives listed below: 1. Define: innate immunity,...
Learning Objectives: Chapter 16 Learning objectives serve as a guide for your learning. After successfully completing the reading of Chapter 16 (pgs. 439-448, Fig 16.8, 452-456, Fig 16.10a) in your text, you should be able to execute the objectives listed below: 1. Define: innate immunity, adaptive immunity, TLRs (toll-like receptors), pathogen associated molecular patterns (PAMPs), lysozyme, probiotics, prebiotics, leukocytes, granulocytes (neutrophils, basophils, eosinophils), macrophages, inflammation, vasodilation, edema, histamine, prostaglandins, cytokines, stroma and parenchyma, complement activation, the classical pathway, the membrane attack complex (MAC) (BL1) 2. Identify 3 examples of PAMPs (BL1) 3. Describe the 1st line of defense and the 2nd line of defense in immunity (BL2) 4. Describe how the “washing action” of the eyes, mouth, lungs, urethra, vagina, and colon benefit a mammal and also identify what chemical factors are present as immune defenses in these areas (BL2) 5. Compare the function and location of the granulocytes (3 kinds) and agranulocytes (3 kinds) (BL4) 6. Summarize the signs/symptoms of inflammation (BL1) and its functions (BL2) and outline the roles of C-reactive protein and TNF-α (BL1) 7. Describe the 3 stages of inflammation using Figure 16.8 (BL2) 8. Cite an example from lecture of high, moderate and Low/No regenerative tissue repair (BL1) 9. Describe the classical complement activation pathway (starting with antibody-antigen binding and going till the creation of a transmembrane channel in the bacteria membrane )(BL2) For the mastery-oriented student: 10. Postulate the roles of CRP (C-reactive protein), fibroblasts (simulated by cytokines), and damage to the walls of the blood vessels in heart disease as part of an inflammatory condition (BL6) 11. Aspirin “thins the blood” (fibrogen) and ibuprofen interferes with prostaglandin production- hypothesize why these might help/hurt heart disease patients (BL5) and also what effect taking chronic doses of these anti-inflammatory drugs might have on the regulation or setpoint of a person’s immune system (BL6)
