Aneurysm, Valve Disease, Cardiomyopathy PDF

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AstonishedBallad8020

Uploaded by AstonishedBallad8020

Saint Joseph's University

2025

DPT

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cardiomyopathy heart valve disease cardiovascular disease medical lectures

Summary

This document is a set of lecture notes on aneurysm, valve disease, and cardiomyopathy, covering topics such as risk factors, symptoms, diagnosis, treatment, and implications. The notes are organized by topic, with clear headings and sections.

Full Transcript

Sit in a different part of the room with a different seat mate ANEURYSM, VALVE DISEASE, CARDIOMYOPATHY, DPT 611 spring 2025 Learning Objectives ◦ At the end of this module, students should be able to: ◦ Describe risk factors for, presentation of, and pathophysiology of a...

Sit in a different part of the room with a different seat mate ANEURYSM, VALVE DISEASE, CARDIOMYOPATHY, DPT 611 spring 2025 Learning Objectives ◦ At the end of this module, students should be able to: ◦ Describe risk factors for, presentation of, and pathophysiology of aneurysms, valve disease, and cardiomyopathy ◦ Describe the diagnostic tests and med/surg interventions for aneurysms, valve disease, and cardiomyopathy. ◦ Describe PT implications for aneurysms, valve disease, and CM, and make decisions about treatment, referral, or emergency care ◦ Identify factors that contribute to health disparities in people with cardiovascular diseases Aneurysm ◦ Weakening in a vessel wall that causes it to bulge or balloon out ◦ Aneurysms can dissect or tear that can lead to life threatening bleeding and/or death ◦ True aneurysm: fusiform ballooning out of vessel wall on all sides of vessel or saccular where it balloons out on one side. ◦ False aneurysm enlargement of any blood vessel layers. Usually, a tear in the intima from trauma, surgery. Blood fills in between layers of blood vessel. ◦ Dissecting: tear separates the three layers of the wall Aneurysm Risk Factors Genetic conditions HTN Atherosclerosis Marfan Syndrome Untreated infections Ehlers-Danlos syndrome Turner’s syndrome Trauma Older age Smoking Bicuspid aortic valve Aneurysms ◦ Most common is in aorta ◦ Next most common is thoracic ◦ Can also occur in: ◦ Abdomen (triple A or AAA) Abdominal Aortic Aneurysm ◦ Common iliac ◦ Femoral ◦ Popliteal ◦ Brain Symptoms – depend on location ◦Asymptomatic ◦AAA – constant pain in abdomen, low back, groin ◦Cerebral – severe HA, vomiting/nausea, visual disturbances ◦Iliac: pain in abdomen, back, groin ◦Femoral or popliteal – palpation of lump that has a pulse ◦ Radiograph, CT, echocardiogram, MRI ◦ Small or slowly evolving aneurysm: watch and wait; medications for risk factor management DX and RX ◦ Median sternotomy or endovascular surgery ◦ Patch to reinforce the weakened wall ◦ Patch or hooks to close the “false lumen” PT Implications Acute care: Sternotomy precautions Mobility Exercise tolerance & medication effectiveness related to function Prescription of home exercise and lifelong behavior change Medications to lower BP and myocardial workload* *No official guidelines for activity After repair, it’s generally recommended not to lift >1/2 your weight Avoid isometrics due to elevations in BP Speak with the surgeon to get parameters Heart valve dysfunction ◦ AV (atrial ventricular ◦ Which valves are closed valves) during ventricular systole ◦ Mitral or bicuspid and why? ◦ Tricuspid ◦ What do S1 and S2 represent? ◦ Semi-lunar valves ◦ When can you feel the pulse? Before or ◦ Pulmonic or after S1? S2? In between S1 and S2? pulmonary ◦ aortic Stand Up! ◦ Student 1 – Gently grab student 2s wrist. I said gently!! ◦ Student 2 – try to pull away and break free (don’t use your other hand) ◦ Chordae Tendineae & Papillary muscles ◦ Students 1 & 2 – stand close to each other (hip to hip) without touching (stenosis) ◦ Students 1 & 2 – get in the “run away” positions again. This time, student 2 should make a little progress in getting away (prolapse) ◦ Students 1 & 2 – stand hip to hip with no spaces (valve closure) and now separate just a little (regurgitation) Types of valve disease Stenosis: narrowing Prolapse: Ballooning/Bulging Regurgitation: Leaking https://www.google.com/url?sa=i&url=https%3A%2F%2Fwww.merckmanuals.com%2Fhome%2Fmultimedia%2Ftable%2Fundersta nding-stenosis-and- regurgitation&psig=AOvVaw1Jy_S8gC2j9upb24ZIPGtm&ust=1706991838437000&source=images&cd=vfe&opi=89978449&ved=0 CBMQjRxqFwoTCIjLoqe-jYQDFQAAAAAdAAAAABAd https://www.google.com/url?sa=i&url=https%3A%2F%2Fwww.mottchildren.org%2Fconditions-treatments%2Fped- heart%2Fconditions%2Fmitral-valve- prolapse&psig=AOvVaw0k7E5zDi2uXH7rVg6R2OAx&ust=1706991917665000&source=images&cd=vfe&opi=899 78449&ved=0CBMQjRxqFwoTCPCNvs--jYQDFQAAAAAdAAAAABAE Risk Factors & Symptoms Risk Factors Symptoms ◦ HTN ◦ Asymptomatic ◦ MI ◦ Angina ◦ Older age ◦ Palpitations ◦ Ischemia to papillary muscles ◦ SOB ◦ Infective endocarditis ◦ Decreased activity tolerance ◦ Heart failure ◦ Lightheadedness/syncope ◦ Congenital malformation of valves ◦ Peripheral edema (sign of heart ◦ Radiation to chest wall – childhood failure) cancer ◦ Rheumatic fever DX & RX DX RX ◦ Auscultation ◦ Medications to reduce risk factors ◦ When there is valve dysfunction, blood ◦ Valve Repair flow is turbulent and can be heard ◦ Balloon valvuloplasty for stenosis ◦ Determine if the abnormal sound occurs during systole or diastole ◦ Valve replacement (stenosis, regurg) ◦ Many murmurs are benign, so need to ◦ Mechanical valve (lifelong anti- correlate with symptoms coagulation) ◦ Tissue valve (human or animal) ◦ Echocardiogram ◦ Transcatheter aortic valve replacement or ◦ Valve structure TAVR or TAVI (I for implantation) for ◦ Direction of blood flow Aortic valve stenosis ◦ Estimation of ejection fraction ◦ *thread up new valve and sit it on top of the old valve VIDEO: https://youtu.be/-kKKo9i3GlA Cardiomyopathy ◦ Disorder of myocardial cells ◦ Myocardial mitochondrial dysfunction → decreased aerobic capacity ◦ Ineffective pump → ↑ LVEDV & pressure → ↓ L ventricular performance ◦ Dilated, Hypertrophic, Restrictive DCM ◦ Dilated (idiopathic, ischemia, toxins, pregnancy, ETOH) ◦ increase in cardiac mass ◦ dilation of 4 chambers ◦ no wall thickening ◦ systolic dysfunction ◦ Higher volumes but lower contractility www.niaaa.nih.gov/ Hypertrophic Cardiomyopathy ◦ Heredity, HTN ◦ Increased cardiac mass ◦ Diastolic Dysfunction ◦ abnormal LV relaxation ◦ increased LV pressures Restrictive CM ◦ XRT, amyloid, DM ◦ “Stiff” ventricles ◦ Decreased V. Filling, compliance ◦ Back pressure to upstream chambers ◦ Decreased SV ◦ Less volume, normal or slightly reduced contractility Myocarditis & Pericarditis ◦ Myocarditis ◦ inflammation of myocardial wall ◦ Pericarditis ◦ inflammation of pericardium chest pain not position or activity dependent Pericardial Effusion ◦ Fluid buildup in potential space ◦ Quick accumulation vs. Slow accumulation ◦ Tamponade – Decreased venous return – Decreased ventricular filling – CO significantly drops – Shock/death – Pericardial friction rub – Pericardiocentesis Stand up for your exercise snack while we talk about simulation 2. Simulation - next class! ◦ Goals: ◦ Advance practice and proficiency with management of a patient in the acute care setting ◦ Gain practice and proficiency with environmental management ◦ Gain practice and proficiency with mobilizing the patient with sternal precautions ◦ Arrive early and stand outside of the room you were assigned ◦ Review the chart and take notes ◦ The simulation begins as soon as you step in the room. ◦ Goal is to assess cardiovascular status, mobility, cardiovascular response to activity, sternal stability, and to educate patient on sternal precautions

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