Biology Past Paper Questions PDF

252. Indicate the reaction, that catalyzed by glycerol kinase: (lyserodan-glycerol-3phosphate) 253. Name the substrate of the mitochondrial stage of gluconeogenesis:( Pyruvate\Krbes) 254. Specify, a normal level of glucose in human serum: (: 3.5 - 5.5 mol) 255. An increase in blood glucose levels ab...

252. Indicate the reaction, that catalyzed by glycerol kinase: (lyserodan-glycerol-3phosphate) 253. Name the substrate of the mitochondrial stage of gluconeogenesis:( Pyruvate\Krbes) 254. Specify, a normal level of glucose in human serum: (: 3.5 - 5.5 mol) 255. An increase in blood glucose levels above normal is called: (Hyperglycemia) 256. Choose a hormone that causes hyperglycemia: (Adrenaline) 257. Choose a hormone, that causes hypoglycemia(insulin) 258. Specify the organ most sensitive to hypoglycemia during prolonged fasting: (MI)(brain) 259. With hyperglycemia caused by a decrease in insulin levels, a violation of the flow of glucose into cells, the process is activated in the cells: (synthesis of glucose from non-carbohydrate precursors metabolic pathway – gluconeogenesis) 260. Indicate the enzyme, the congenital deficiency of which leads to hypoglycemia in glycogenosis: (glycogen phosphorylase) 261. A liver enzyme needed to maintain blood sugar levels: (Glucose6phosphatase) 262. The formation of ribulose-5-phosphate at the oxidative stage of the pentose phosphate pathway occurs as a result of the reaction: (decarboxylden) 263. Indicate, a reaction leading to the formation of erythroso-4-phosphate and fructose-6- phosphate from sedoheptulose-7-phosphate and glyceroaldehyde-3-phosphate: (Transaldose) 264. An enzyme that catalyzes the conversion of ribulose-5-phosphate to ribose-5-phosphate: (Isomerase) 265. Name the method for modifying glycogen phosphorylase and glycogen synthase: (removal of phosphate groups from a biological compound As a result of dephosphorylation, energy-rich ATP is found adenosine triphosphatase across macroergic membranes ion transport\ phosphorylation and dephosphorylation) 266. Muscle glycogen not involved in maintaining blood glucose levels because: (: due to lack of glucose 6-phosphatase.) 267. To identify ways, which stimulates glycogen degradation by glucagon hormone. (glycogenolysis) 268. Indicate the biological role of glycogen mobilization in the liver: (blood glucose concentration saver) 269. Indicate the organ-specific enzyme of liver: (glucose 6 phosphatase) 270. A hormone, that stimulates the mobilization of glycogen in the liver: (Adrenaline) 271. A hormone that activates glycogen phosphorylase: (Adrenaline) 272. Indicate the final product of the breakdown of glycogen in muscles: (glucose 6 phosphate ) 273.Indicate the chemical bond in the linear regions of the glycogen molecule:( alpha-1\4- glycoside) 274.The donor of apoproteins is: (density high lipoproteins) 275.Removal of excess cholesterol from cells is carried out: (high density Lipoproteins) 276. The reason for the increased content of chylomicrons and VLDL in the blood is a genetic defect of: (LPL\LPL genetic (lipoprotein lipase) 277. Indicate, an effect of insulin on beta-oxidation of fatty acids: (: inhibition) 278. The synthesis of ketone bodies are regulated by:( hydroxymethylglutaryl-CoA-synthase \ HMG-CoA- synthase) 279. During fasting, physical activity under the influence of the hormones glucagon and epinephrine, increases the speed: (B oxidation (in fat cells) 280. Beta-oxidation of fatty acid is inhibited by:( malonyl-CoA metabolite) 281.. The transport of dietary fats from the intestine to the tissue is carried out: (Chylomicrons/ HM \ecosenic lipid) 282.. In the post-absorptive period, the content of chylomicrons and VLDL in the blood is increased with a genetic defect (enzyme): (: LPL/ Lipoprotein lipase) 283. Of all the complexes that transport lipids, atherogenic ones are: (low-density lp) 284. Calculate the number of ATP, that is formed during the completely beta-oxidation of stearic acid: (147) 285. Specify a product of the first reaction of fatty acid synthesis: (Malonyl-CoA metabolite) 286. The regulatory enzyme of synthesis of fatty acids acetyl-CoA-carboxylase is activated: (dephosphorylated in a way) 287. The regulation of the synthesis of ketone bodies is carried out by an enzyme: (hydroxymethylglutaryl-CoA-synthase) 288. Name the products of intracellular hydrolysis TAG: (fatty acid ) 289. Synthesis of cholesterol in the liver is regulated according to the feedback principle. Indicate an inhibitor of HMG-CoA reductase: (LIPID/FAT/cholesterol) 290. Name the enzyme, that catalyzes the formation of cholesterol esters: (HLD) 291. Choose a key reaction in the biosynthesis of cholesterol from acetyl-CoA: (: 3,5- dihydroxy-3- methylvaleric acid (mevalon) 292.Indicate the type of lipoproteins containing the highest concentration of cholesterol: (HLD) 293. What enzyme is involved in the formation of cholesterol esters: (Acyl—S— CoA+CarnitineÿAcylcarnitine+CoA-SH (acyltransferase) 294. Excess intake of cholesterol from food leads to: (reduction of HMG-CoA reductase) 295. Risk factors for the development of atherosclerosis are: (increased TTLP) 296. With a lack of the coenzyme tetrahydrobiopterin, the metabolism of amino acids is disrupted: (aromatic ak\phen, tyr, tri) 297. The regulatory enzyme that determines the rate of catecholamine synthesis: (tyrosine-3- monooxygenase\TG) 298.Indicate a substance capable of transferring the formyl group to other compounds: (serine- oxymethyltransferase\H4folate) 299.The methyl group of this amino acid is used in transmethylation reactions: (methionine- oxytocin\ -amino acid) 300. Name an enzymes, which necessary for the inactivation of biogenic amines: (DNMT (methyltransferase)