resp physiology Q6

it's a slight increase in Pco2>H+. it stimulates both peripheral(carotid) and central chemoreceptors.

its oxygen deficiency at tissue level caused by a low Po2 in atrial blood due to airway obstruction or fluid in the lungs. it stimulates the peripheral (carotid) chemoreceptors.

self regulatory negative feedback as inflation inhibits further inspiratory muscle activity, deflation initiates inspiratory activity.

<p>when lungs are inflated, this is detected by lung stretch receptors&gt;impulses travel via vagus nerve and large myelinated fibres to the brain&gt;slow down the breathing rate to prevent over inflation and protect the lungs.</p> Signup and view all the answers

<p>in carotid and aortic bodies.</p> Signup and view all the answers

<p>decreased arterial o2, increased co2, and H+ (rapid responding).</p> Signup and view all the answers

<p>near ventral surface of (medulla).</p> Signup and view all the answers

<p>to Co2 but not O2, they respond to the change of pH of the ECF/CSF when co2 diffuse out the cerebral capillaries.</p> Signup and view all the answers

<p>brainstem.</p> Signup and view all the answers

<p>Pre-Botzinger complex, a collection of pace-maker cells located at the upper end of Dorsal respiratory group (DRG), it discharges/transmits rhythmic resp signals.</p> Signup and view all the answers