Podcast
Questions and Answers
In the context of echocardiography, which of the following best describes the underlying biophysical principle that enables image formation?
In the context of echocardiography, which of the following best describes the underlying biophysical principle that enables image formation?
- The thermoelectric effect, where temperature gradients within the heart induce electrical currents convertible to images.
- Nuclear magnetic resonance, exciting hydrogen atoms within cardiac tissues and detecting emitted radiofrequency signals.
- Differential absorption of ionizing radiation by cardiac tissues, creating a density map.
- The Doppler effect, specifically the measurable frequency shift of reflected sound waves from moving cardiac structures. (correct)
What is the most critical limitation of transthoracic echocardiography (TTE) in the context of advanced cardiovascular imaging?
What is the most critical limitation of transthoracic echocardiography (TTE) in the context of advanced cardiovascular imaging?
- Lack of real-time three-dimensional imaging capabilities, hindering comprehensive ventricular volume assessment.
- Suboptimal acoustic windows due to body habitus, leading to compromised image resolution and diagnostic accuracy. (correct)
- Inability to quantify myocardial strain with sufficient precision for early detection of subtle cardiomyopathies.
- Failure to provide adequate visualization of coronary artery anatomy for the detection of non-calcified plaques.
In patients undergoing transesophageal echocardiography (TEE) for suspected aortic dissection, what pharmacological intervention is MOST critical to manage a potential adverse reaction during the procedure?
In patients undergoing transesophageal echocardiography (TEE) for suspected aortic dissection, what pharmacological intervention is MOST critical to manage a potential adverse reaction during the procedure?
- Preemptive administration of a loop diuretic to prevent pulmonary edema from increased venous return during sedation.
- Administration of a non-selective beta-blocker to counteract reflex tachycardia and hypertension from esophageal manipulation. (correct)
- Prophylactic use of a glycoprotein IIb/IIIa inhibitor to mitigate the risk of thromboembolic events secondary to probe insertion.
- Routine use of a vasopressor to counteract hypotension induced by the mechanical pressure of the TEE probe on the heart.
A patient undergoing cardiac catheterization via the femoral approach develops sudden-onset, severe lower back pain radiating to the groin, accompanied by hypotension and tachycardia. What immediate intervention is MOST crucial, beyond standard bleeding precautions?
A patient undergoing cardiac catheterization via the femoral approach develops sudden-onset, severe lower back pain radiating to the groin, accompanied by hypotension and tachycardia. What immediate intervention is MOST crucial, beyond standard bleeding precautions?
Following percutaneous coronary intervention (PCI) with stent placement, a patient exhibits ST-segment elevation in previously normal leads along with hemodynamic instability. Which of the following represents the MOST likely underlying mechanism?
Following percutaneous coronary intervention (PCI) with stent placement, a patient exhibits ST-segment elevation in previously normal leads along with hemodynamic instability. Which of the following represents the MOST likely underlying mechanism?
In the management of a patient post-cardiac catheterization who develops a large hematoma at the femoral access site, what is the MOST appropriate next step after applying manual pressure?
In the management of a patient post-cardiac catheterization who develops a large hematoma at the femoral access site, what is the MOST appropriate next step after applying manual pressure?
A patient with known heart failure presents with worsening dyspnea and lower extremity edema. Laboratory results reveal a significantly elevated B-type natriuretic peptide (BNP). What pathophysiological mechanism MOST directly accounts for the increased BNP level?
A patient with known heart failure presents with worsening dyspnea and lower extremity edema. Laboratory results reveal a significantly elevated B-type natriuretic peptide (BNP). What pathophysiological mechanism MOST directly accounts for the increased BNP level?
A patient with chronic heart failure is prescribed digoxin. Which of the following electrolyte imbalances MOST significantly increases the risk of digoxin toxicity and associated arrhythmias?
A patient with chronic heart failure is prescribed digoxin. Which of the following electrolyte imbalances MOST significantly increases the risk of digoxin toxicity and associated arrhythmias?
In a patient with acute pulmonary edema secondary to left ventricular failure, what is the primary rationale for administering intravenous morphine beyond its analgesic properties?
In a patient with acute pulmonary edema secondary to left ventricular failure, what is the primary rationale for administering intravenous morphine beyond its analgesic properties?
Which of the following best describes the mechanism by which beta-blockers improve outcomes in patients with heart failure with reduced ejection fraction (HFrEF)?
Which of the following best describes the mechanism by which beta-blockers improve outcomes in patients with heart failure with reduced ejection fraction (HFrEF)?
In the context of managing a patient with an acute ST-segment elevation myocardial infarction (STEMI), which of the following represents the MOST critical rationale for rapid percutaneous coronary intervention (PCI)?
In the context of managing a patient with an acute ST-segment elevation myocardial infarction (STEMI), which of the following represents the MOST critical rationale for rapid percutaneous coronary intervention (PCI)?
Following an acute myocardial infarction (MI), a patient develops Dressler's syndrome. What underlying immunological mechanism is PRIMARY in the pathogenesis of this condition?
Following an acute myocardial infarction (MI), a patient develops Dressler's syndrome. What underlying immunological mechanism is PRIMARY in the pathogenesis of this condition?
A patient presents to the emergency department with chest pain suggestive of angina. Initial ECG is non-diagnostic. Which biomarker is the MOST sensitive and specific for detecting myocardial injury in this setting within the first few hours of symptom onset?
A patient presents to the emergency department with chest pain suggestive of angina. Initial ECG is non-diagnostic. Which biomarker is the MOST sensitive and specific for detecting myocardial injury in this setting within the first few hours of symptom onset?
A patient with stable angina is prescribed nitroglycerin for sublingual use. What is the MOST accurate explanation of nitroglycerin's mechanism of action in relieving anginal pain?
A patient with stable angina is prescribed nitroglycerin for sublingual use. What is the MOST accurate explanation of nitroglycerin's mechanism of action in relieving anginal pain?
A patient with peripheral artery disease (PAD) reports experiencing claudication. Which of the following pathophysiological mechanisms BEST explains the cause of this symptom?
A patient with peripheral artery disease (PAD) reports experiencing claudication. Which of the following pathophysiological mechanisms BEST explains the cause of this symptom?
What is the MOST critical consideration when educating a patient with peripheral artery disease (PAD) about lower extremity care?
What is the MOST critical consideration when educating a patient with peripheral artery disease (PAD) about lower extremity care?
A patient with chronic venous insufficiency develops a venous stasis ulcer on their lower leg. What is the PRIMARY underlying pathophysiological mechanism contributing to the development of this ulcer?
A patient with chronic venous insufficiency develops a venous stasis ulcer on their lower leg. What is the PRIMARY underlying pathophysiological mechanism contributing to the development of this ulcer?
In a patient with a deep vein thrombosis (DVT), what role does Virchow's triad play in the pathogenesis of the thrombus formation?
In a patient with a deep vein thrombosis (DVT), what role does Virchow's triad play in the pathogenesis of the thrombus formation?
What pharmacological intervention is MOST critical in the acute management of a patient diagnosed with a deep vein thrombosis (DVT)?
What pharmacological intervention is MOST critical in the acute management of a patient diagnosed with a deep vein thrombosis (DVT)?
Which of the following represents the MOST critical long-term complication associated with chronic kidney disease (CKD)?
Which of the following represents the MOST critical long-term complication associated with chronic kidney disease (CKD)?
What is the PRIMARY mechanism by which uncontrolled hypertension accelerates the progression of chronic kidney disease (CKD)?
What is the PRIMARY mechanism by which uncontrolled hypertension accelerates the progression of chronic kidney disease (CKD)?
Which dietary modification is MOST critical for patients with chronic kidney disease (CKD) to slow disease progression and manage complications?
Which dietary modification is MOST critical for patients with chronic kidney disease (CKD) to slow disease progression and manage complications?
Why is erythropoietin (EPO) replacement therapy often necessary in patients with chronic kidney disease (CKD)?
Why is erythropoietin (EPO) replacement therapy often necessary in patients with chronic kidney disease (CKD)?
Which of the following represents the MOST critical consideration when caring for a patient who has just undergone creation of an arteriovenous fistula (AVF) for hemodialysis access?
Which of the following represents the MOST critical consideration when caring for a patient who has just undergone creation of an arteriovenous fistula (AVF) for hemodialysis access?
What underlying pathophysiological mechanism PRIMARY explains the increased risk of bladder cancer in smokers?
What underlying pathophysiological mechanism PRIMARY explains the increased risk of bladder cancer in smokers?
Following a radical cystectomy with ileal conduit formation, what is the MOST critical long-term complication that patients should be monitored for?
Following a radical cystectomy with ileal conduit formation, what is the MOST critical long-term complication that patients should be monitored for?
Which urinalysis finding is MOST specific for identifying a urinary tract infection (UTI)?
Which urinalysis finding is MOST specific for identifying a urinary tract infection (UTI)?
In a patient experiencing urinary retention due to benign prostatic hyperplasia (BPH), what pharmacological mechanism does tamsulosin (Flomax) employ to improve urinary flow?
In a patient experiencing urinary retention due to benign prostatic hyperplasia (BPH), what pharmacological mechanism does tamsulosin (Flomax) employ to improve urinary flow?
What is the MOST critical post-operative nursing intervention following a transurethral resection of the prostate (TURP)?
What is the MOST critical post-operative nursing intervention following a transurethral resection of the prostate (TURP)?
What underlying mechanism explains why beta-blockers are a risk factor for erectile dysfunction?
What underlying mechanism explains why beta-blockers are a risk factor for erectile dysfunction?
In the management of acute bacterial prostatitis, which antibiotic class is generally considered FIRST-LINE due to its excellent penetration into prostatic tissue?
In the management of acute bacterial prostatitis, which antibiotic class is generally considered FIRST-LINE due to its excellent penetration into prostatic tissue?
A patient with chronic prostatitis is being educated on self-care strategies. What is the rationale behind recommending regular ejaculation as part of their management?
A patient with chronic prostatitis is being educated on self-care strategies. What is the rationale behind recommending regular ejaculation as part of their management?
In a patient with hypertensive crisis exhibiting signs of end-organ damage, such as encephalopathy, acute kidney injury, or myocardial ischemia, which intravenous antihypertensive agent is GENERALLY preferred?
In a patient with hypertensive crisis exhibiting signs of end-organ damage, such as encephalopathy, acute kidney injury, or myocardial ischemia, which intravenous antihypertensive agent is GENERALLY preferred?
Flashcards
Echocardiogram
Echocardiogram
Noninvasive ultrasound to evaluate heart structure and function.
Transthoracic Echocardiogram (TTE)
Transthoracic Echocardiogram (TTE)
Echocardiogram from the outside, non-invasive; for valve disorders and cardiomyopathy.
Transesophageal Echocardiogram (TEE)
Transesophageal Echocardiogram (TEE)
Invasive echo via esophagus to have superior image quality because it is closer to the heart.
Cath Lab
Cath Lab
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Percutaneous Coronary Intervention (PCI)
Percutaneous Coronary Intervention (PCI)
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B-type Natriuretic Peptide (BNP)
B-type Natriuretic Peptide (BNP)
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CHF
CHF
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Left (Ventricular) Failure
Left (Ventricular) Failure
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Right Sided Heart Failure
Right Sided Heart Failure
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ACE inhibitors & ARBS
ACE inhibitors & ARBS
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Diuretics
Diuretics
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Digoxin
Digoxin
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Beta-blockers
Beta-blockers
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Continuous Positive Airway Pressure (CPAP)
Continuous Positive Airway Pressure (CPAP)
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Cardiac Resynchronization Therapy (CRT)
Cardiac Resynchronization Therapy (CRT)
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Pulm Edema
Pulm Edema
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Atherosclerosis
Atherosclerosis
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Angina Pectoris
Angina Pectoris
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Myocardial Infarction
Myocardial Infarction
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HTN
HTN
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Peripheral Vascular Resistance
Peripheral Vascular Resistance
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RAAS
RAAS
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HTN nursing Interventions
HTN nursing Interventions
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Medication side effects for HTN medications
Medication side effects for HTN medications
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PAD & PVD
PAD & PVD
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Arteriosclerosis
Arteriosclerosis
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Atherosclerosis
Atherosclerosis
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PAD
PAD
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Collateral circulation
Collateral circulation
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Classic signs & symptoms of DVT
Classic signs & symptoms of DVT
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CKD
CKD
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Prevention of cystitis
Prevention of cystitis
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Genitourinary
Genitourinary
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Urinary Incontinence
Urinary Incontinence
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TURP (Transurethral Resection of the Prostate)
TURP (Transurethral Resection of the Prostate)
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Prostatitis
Prostatitis
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Acute bacterial prostatitis
Acute bacterial prostatitis
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Chronic prostatitis
Chronic prostatitis
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Study Notes
Echocardiogram
- A noninvasive ultrasound assesses heart structure and function
- Diagnoses valve disorders and cardiomyopathy like wall motion abnormalities, valve problems, and ejection fraction
- Detects fluid around the heart
Transthoracic Echocardiogram (TTE)
- A non-invasive external ultrasound of the heart
- Good image quality that can be limited by body habitus
- Sedation and routine prep are unneeded
- It is used to diagnose valve disorders and cardiomyopathy
- Evaluates heart structure size, shape, and motion
- Measures ejection fraction to determine heart health; a low ejection fraction is bad
Ejection Fraction
- It is non-reversible but its progression can be slowed
How Echocardiograms are Used
- Uses sound waves (no radiation)
- Gel is applied to help waves pass through the skin
- A probe is moved over the chest area
- The gel is wiped off after the procedure
Patient Education for Echocardiograms
- Explain the test's purpose
- Explain that the test takes 30-60 minutes
- Reassure that the procedure is painless and requires no sedation or recovery time
Transesophageal Echocardiogram (TEE)
- An invasive procedure where a scope is passed into the esophagus
- Offers superior image quality due to its proximity to the heart
- Produces clear ultrasonic images because waves pass through less tissue
- Detects clots, valve issues, endocarditis, and congenital defects
- More convenient for obese patients or women with large breasts; it bypasses extra tissue for a better heart view
How TEEs are Used
- Involves passing a small transducer through the mouth into the esophagus
- Sedation is used to prevent the gag reflex
- Provides images of the heart
Patient Education for TEEs
- Explain the test purpose
- The patient must be NPO for at least 6 hours prior to the test
- Explain to the patient how the procedure will be performed
Cath Lab
- Used for coronary angiography and stent placement
- Done in the cath lab or OR
- Invasive procedure to evaluate the presence and degree of coronary artery blockage
- Goal is to find and fix blockages, opening up the artery
Catheter Insertion
- A catheter is inserted into one of three types of vessels and threaded into either side of the heart
- The vessels are femoral, brachial, and radial
Percutaneous Coronary Intervention (PCI)
- Performed when cardiologists can access the blockage via a catheter
- Involves procedures like percutaneous transluminal coronary angioplasty (PTCA), arthrectomy, and stenting
Percutaneous Transluminal Coronary Angioplasty (PTCA)
- Stent goes around a balloon, which is inflated on the blockage to expand
- The stent is placed and left there
Arthrectomy
- Procedure to clean out blockages
Stenting
- Involves inserting expandable tubes into a narrowed or blocked artery
Rheolytic Thrombectomy
- Involves aspirating clots out of the vessel
Post Cath Care
- Frequently check vital signs and hemodynamic status
- Enforce bed rest per protocol, due to blood thinners and fall risk
Catheter Placement in the Groin
- Keep the patient on bedrest and lay them back for 1-2 hours
- Avoid bending and bleeding at the puncture/incision site
- The head of the bed must not be more than 15 degrees and bending the hip can cause bleeding
Assessing for Complications
- Look for bleeding at the perforation, heart, and insertion site
- Position is important because they will hold pressure on the cath site for 15 minutes
- For wrist bleeding; draw air out of the balloon slowly
- For groin bleeding; assess the site and look for bleeding beneath
Other Complications
- Hematoma formation involves bleeding inside the tissues
- Assess the site for color, swelling, and hardness
- Thrombosis has a risk of clot from bedrest, assess circulation via capillary refill, pedal pulse, and skin tone
- Cardiac dysrhythmias
Kidney Damage
- Radioactive dye is hard on the kidneys, an IV helps remove the dye
Discharge Instructions
- Avoid heavy activity for 24-48 hours and lifting over 5-10 pounds until cleared by a cardiologist
- Keep the site clean with soap and water, and pat it dry
- Watch for infection and report abnormalities or hematomas
- Medication teaching
Complications
- Bleeding/hematoma requires holding pressure and calling the provider
- Infection, dysrhythmias, contrast-induced nephropathy, thrombosis, allergic reaction to contrast dye, and arterial dissection are all complications
Cardiomyopathy
- Disease that affects the heart muscle, stretching of the heart
CHF (Heart Failure)
- Can be thought of as pump failure
Types of Heart Failure
- Left (Ventricular) failure causes a backup into pulmonary circulation
- Right-sided heart (ventricular) failure causes systemic circulation failure
- High output failure is due to increased metabolic need
- Systolic heart failure means the heart cannot contract or push forcefully enough to reduce injection fraction
- Diastolic heart failure means the heart doesn't relax or refill as it should
Left-Sided Heart Failure
- Involves the inability of the left ventricle to fill or eject enough blood out
- Think lungs since blood gets backed up in the lungs
Effects of Left Sided Heart Failure
- Decreased cardiac output leads to impaired tissue perfusion (especially toes and fingers)
- There’s increased venous pressure
- Backing up of blood (pulmonary congestion) leads to breathing issues
- Weakness, fatigue, dyspnea, orthopnea, pulmonary congestion, cough, and pink-frothy sputum are signs/symptoms
- Chest discomfort and irregular heart rhythm
Right-Sided Heart Failure
- Involves the inability of the right ventricle to fill or eject enough blood into the pulmonary circulation
- The right side of the heart normally pumps deoxygenated blood to the lungs
Other Symptoms
- Peripheral edema, ascites, and enlarged liver
- Diuresis at rest occurs because lying down puts less workload and the body does its job
- Jugular distention and dependent edema are also seen
- Fluid retention and high blood pressure are symptoms
Diagnostics and Lab Testing
- B-type natriuretic peptide (BNP) is a hormone that promotes vasodilation & diuresis, produced when the ventricles have fluid overload; used to determine heart failure
- CMP indicates electrolytes
- CBCs look at blood supply or anemia
- Chest XRAY sees the heart and fluid presence
- Echocardiography visualizes the heart structure with sound waves
Nursing Interventions
- Take medications as prescribed
- Stay active, but don’t overdo it and take breaks
- Limit salt intake to avoid fluid overload
- Report symptoms to the provider
- Elevate the head of the bed and use extra pillows
- Avoid NSAIDs
Medications
- ACE inhibitors & ARBs which block aldosterone and prevents water and sodium retention which means a cough is normal and harmless
- Diuretics get rid of excess fluid and reduce workload
- Digoxin increases contractility while slowing the heart rate and SNS
- Beta-blockers reduce the workload and vasodilate
More Interventions
- Continuous Positive Airway Pressure (CPAP) is used for sleep apnea
- Cardiac Resynchronization Therapy (CRT) uses a pacemaker
Other Lifestyle Changes and Interventions
- Encourage dietary modifications, such as a low sodium diet
Pulm Edema
- A large amount of fluid backing up in the lungs (LHF)
- A complication of LHF related to fluid backup
Nursing Assessment & Interventions
- Assess for shortness of breath at rest, anxiety, restlessness, and confusion
- Monitor increased BP, reduced urinary output, pink-frothy sputum, and dysrhythmias
- If not hypotensive, sit the patient up and drop legs to reduce venous return
- Provide high flow oxygen therapy, nitroglycerin, diuretics, and IV morphine
Dietary & Lifestyle Changes
- Focus on sodium reduction
CAD (Coronary Artery Disease)
- Also known as heart disease
- CAD is a broad term including stable angina and acute coronary syndromes
- One of the most prevalent cardiovascular diseases in adults
Atherosclerosis
- Blocked/hardened/narrowed coronary arteries
- Results in myocardial infarction through plaque buildup in arterial walls
- High cholesterol attaches to arterial walls where it becomes hardened resulting in a deficient blood supply.
Risk Factors
- Non-modifiable risk factors are age, gender, ethnicity, and family history
- Modifiable risk factors include smoking, diet, activity, and obesity
Diagnostics and Lab Testing
- Look at lipid panels, LDL (bad cholesterol) <100, HDL (good cholesterol) >60, triglycerides >= 175 mg/dL
- Elevated BP
- ECG or EKG
Other Testing and Interventions
- Stress test and cardiac cath
- Check troponin T levels
Nursing Interventions
- Medication adherence and monitor for chest pain
Patient Teaching
- Stop smoking
Diet Modifications
- Follow a low fat, low cholesterol, high fiber/Mediterranean diet
- Patients should limit their intake of sweets and sugar-sweetened beverages
- Refer to a dietitian
Physical Activity
- Regular, moderate physical activity increases HDL levels and reduces triglyceride levels
- Reduces the incidence of coronary events and overall mortality risk
- Aim for 150 minutes of moderate or 75 minutes of vigorous aerobic activity per week
Medication Education
- If diet alone cannot normalize cholesterol levels, use meds alongside diet
Further Info
- Bile acid sequestrants, HMG-CoA (statins), and cholesterol absorption inhibitors can be used
Angina Pectoris
- An imbalance between oxygen supply and the cardiac muscle's demand
- It causes chest pain and can lead to ischemia
Chronic Stable Angina
- Patient has had angina before
- Easily relieved with medication (nitroglycerin)
Signs and Symptoms
- Chest pain, may be poorly localized and may radiate
- Feeling of weakness or numbness in the arms, wrists, and hands
- Shortness of breath, pallor, diaphoresis, dizziness or lightheadedness, nausea & vomiting are symptoms
Maintained with Meds
- Nitroglycerin is the main treatment
- Dissolve under the tongue, one at a time, 5 minutes apart (max of 3 doses)
- If no improvement after 3 doses, seek immediate medical attention
Myocardial Infarction
- Heart attack caused by plaque holding on to platelets and causing blood clots, preventing oxygen from reaching the heart
- Described as feeling like an elephant is sitting on the chest
Stemi
- EKG shows how the hearts beating, STEMI EKG can look like a tombstone with ST elevation
Nonstemi
- Shows a normal sinus rhythm
Nursing Assessment
- Assess airway, vital signs, breathing, and circulation
- Prep the patient for cath lab to open the blockage
- Obtain 12-lead ECG for STEMI
- Ask about pain location, radiation, how the patient would describe the pain, and if it is similar to previous pain
Other Questions for Patients
- "Can you rate the pain on a 0-10 scale, with 10 being the most pain?"
- "When did the pain begin?"
- "How long does it last?"
- "What brings on the pain?"
- “What helps the pain go away?"
- “Do you have any other symptoms with the pain?”
Medical and Nursing Interventions
- Morphine sulfate for pain
- Oxygen administration
- Nitroglycerin and Aspirin/Plavix
Other Interventions
- Beta blocker for vasodilation and to reduce HR & BP
- Have patient stop activities and sit and rest in bed in a semi-Fowler's position
- Monitor troponin T, CKMB, and lipid profiles
- Possible stress test or thallium, microalbuminuria, blood coagulation studies, check electrolytes, and possible chest XRAY
Types of Angina and Patient Teaching
- Unstable angina: new onset, increasing frequency and severity, not relieved with rest or nitroglycerin
- Intractable or refractory angina: severe incapacitating chest pain
- Variant angina/Prinzmetal’s angina: pain at rest with reversible ST-segment elevation; caused by coronary artery vasospasm
- Silent ischemia: objective evidence of ischemia (such as electrocardiographic changes with a stress test), patient reports no pain
Complications After MI
- Death, arrhythmia, rupture, tamponade, heart failure, valve disease, aneurysm of ventricle, Dressler’s syndrome, embolism, recurrence/ mitral regurgitation
Vascular Information
- “Peripheral” means away from the heart (fingers, toes, brain, ears, nose, etc!)
HTN (Hypertension)
- The pressure in a cardiovascular system determined by cardiac output (how much the heart can pump out, HR x Stroke Volume = CO)
High BP Impacts
- High BP can affect the heart, kidneys, brain, peripherals, etc
Peripheral Vascular Resistance
- Small arterioles; smallest diameter; responsive to fight or flight
Diagnosis of HTN
- Must be based on 2+ accurate readings taken 1-4 weeks apart or 2-3 week period
Arterial Baroreceptors
- Take action when a change is sensed
- Can lead to vasoconstriction and vasodilation
- Sense changes in body position, such as orthostatic hypotension
Regulation of Body Fluid Volume
- Kidneys regulate BP by retaining or excreting fluid
RAAS (Renin-Angiotensin-Aldosterone System)
- Converts angiotensin 1 to angiotensin II and controls aldosterone release
- Causes sodium to be reabsorbed and retain fluid
Vascular Autoregulation
- Maintains tissue perfusion
HTN Classifications (for Patients <60)
- Normal: both systolic and diastolic must be 120-139 mmHg & 80-89 mm Hg
- Stage 1 HTN: systolic or diastolic 140-159 mm Hg & 90-99 mm Hg
- Stage 2 HTN: systolic or diastolic >/= 160 mm Hg & >/=100 mm Hg
HTN Classifications (Age Considerations)
- Ages 60-65 ~ 150/90
- Ages 150 mm Hg; diastolic >130-150 mm Hg
- Common symptoms include morning HA, blurred vision, and dyspnea
- May lead to kidney failure, left ventricular heart failure or stroke if untreated
Modifiable Risk Factors
- Maintain healthy weight; exercise regularly (30-60 minutes/day, 5 days/week); DASH meal plan; reduce daily sodium intake (2300 mg or less a day, 1500 mg a day for people 50+); limit daily alcohol intake; avoid tobacco/ nicotine; decrease caffeine intake; maintain potassium and calcium intake; reduce stress; and monitor BP daily.
Nonmodifiable Risk Factors
- Age, family hx, gender, race
Signs and Symptoms
- Headaches, facial flushing, diaphoresis, nosebleeds, dizziness, fainting, vision changes, nocturia are common
CMS
- Often asymptomatic “silent killer” and targets organs with retinal damage, cardiac changes, renal changes, and neurological changes
Other Interventions
- Lifestyle modification, limit alcohol, quit smoking, monitor BP regularly, and exercise
Medical Management
- Diuretics (first prescribed) and antihypertensives as a means of medical management
Medication Side Effects
- Too low BP (hypotension); dizziness; bradycardia; orthostatic hypotension; erectile dysfunction; electrolyte imbalance; cough
Patient Teaching
- Stand up slowly, and encourage medication adherence
- Encourage regular BP checks
Complications of Prolonged Force
- Prolonged arterial muscle thickening increases work
- Increased force damages arterial inner lining, high cholesterol forming a plaque, and hardened plaque in the artery
Other Complications
- Aneurysm; accelerated atherosclerosis; narrowed vessel lumen; ventricular hypertrophy; myocardial infarction; heart failure; cerebrovascular disease; renal failure; and retinopathy
Hypertensive Crises
- Hypertensive Crisis: occurs rapidly; may be urgent or emergent; REQUIRES IMMEDIATE INTERVENTION
More On Hypertensive Crisis
- CM: extremely elevated diastolic/systolic pressure; severe headache; dizziness, blurred vision; shortness of breath; anxiety
- Administer oxygen; position in semi-Fowler’s position, or reverse Trelleborg, or higher, to take pressure off the heart; start IV access; administer IV antihypertensive therapy
- IV HYPERTENSIVE THERAPY: sodium nitroprusside- Nipride: for emergency hypertension crisis; fast, potent vasodilator; causes venous and arterial vasodilation; infusion pump; protect from light;
- Monitor BP every 5-15 minutes until diastolic
Prostate-Specific Antigen (PSA) Levels
- 0-4 ng/mL=normal
- 4-10 ng/mL=prostatitis
-
10ng/mL = cancer
Primary Prostate Symptoms
- Urgency, frequency, and nocturia
- After it progresses: urinary retention, hematuria, enlarged prostate, back pain, secondary to metastasis (bone pain, because cancer can spread to lymph nose and bones); painful ejaculation; sexual dysfunction
Symptoms d/t Metastasis
- Backache, hip pain, perineal and rectal discomfort, enemia, weight loss, weakness, nausea, oliguria (decreased urine output)
Radical Prostatectomy Nursing Interventions
- For Radical Prostatectomy: maintain hydration with IV therapy; care for wound drains; analgesics ; prevent emboli; prevent pulmonary
TURP (Transurethral Resection of the Prostate)
- Surgical removal of prostate tissue through the urethra
- Closed procedure where patients given anesthesia; removes part of the enlarged prostates
- A 3-way catheter is inserted to irrigate the bladder to prevent blood clots
Post Op Care For TURP
- CHECK FOR BLADDER OCCLUSION FIRST! - Irrigation to remove clots; insure drainage bag is not full
- 30-45 mL retention balloon through the urethra of the bladder
- Taped to patient’s abdomen or thigh
- Monitor decrease in hematuria, first day red urine is normal but after 24 hours should be pinkish, clots are normal, bright-red urine after 24 hours report
Other Aspects of TURP
- Check B NO suppository, and vital signs
- Closed continuous bladder irrigation and possible serial urines after cathether removal
- Ambulate early and no prolonged sitting
- Stool softeners
Post-op Teaching
- Avoid alcohol, kegal exercises and increase fluids
- Resume sexual function after 6-8 weeks
Other Factors
- Erectile Dysfunction is also called impotence and it has risk factors like beta-blockers and antidepressants
- Offer reassurance
Prostatitis
- Inflammation or infection of the prostate and organisms reaching the prostate through the urethra or the blood stream
- Extremely painful, especially in acute cases
Acute Bacterial Prostatitis
- Low back pain, deep pain between the anus & scrotum; chills & fever; dysuria, frequency, urgency, nocturia; discomfort when sitting; anal penetration=bad hygiene
Medications For Acute
- Antipyretics, antispasmodics that may have UTI, and stool softeners
- Encourage fluid intake
Chronic Prostatitis
- Involves chronic UTI with weak stream, difficulty starting urination, pain and urethral discharge
Reduce Fluid Intake
- Avoid retention of prostatic fluid by providing reduced fluid intake
Nursing Care
- Increase fluids and avoid foods/fluids that promote a diuretic effect
- Sitz bath and donut pillows are indicated
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