Podcast
Questions and Answers
What is the most common cause of camphor-related poisonings?
What is the most common cause of camphor-related poisonings?
- Ingesting camphorated oil with acne products
- Using it as a rubefacient
- Mistaking it for castor oil (correct)
- Using camphor as a repellent
How quickly can signs and symptoms of camphor poisoning appear after ingestion?
How quickly can signs and symptoms of camphor poisoning appear after ingestion?
- 5 to 15 minutes (correct)
- 30 to 45 minutes
- 1 to 2 hours
- 3 to 4 hours
What is the predominant effect of camphor on the brain?
What is the predominant effect of camphor on the brain?
- Stimulation at all levels (correct)
- Depression of brain activity
- Selective stimulation of specific brain areas
- No effect on brain activity
Which of the following is NOT a sign or symptom of camphor poisoning?
Which of the following is NOT a sign or symptom of camphor poisoning?
What is the predominant effect on the skin in camphor poisoning?
What is the predominant effect on the skin in camphor poisoning?
What is the approximate lethal dose of camphorated oil for adults?
What is the approximate lethal dose of camphorated oil for adults?
How does camphor enter the central nervous system (CNS)?
How does camphor enter the central nervous system (CNS)?
Which organ may experience transient damage due to camphor poisoning?
Which organ may experience transient damage due to camphor poisoning?
What is the primary mechanism for inducing toxic symptoms in camphor poisoning?
What is the primary mechanism for inducing toxic symptoms in camphor poisoning?
What is the purpose of monitoring pulse oximetry and/or arterial blood gases in patients with moth repellent exposure?
What is the purpose of monitoring pulse oximetry and/or arterial blood gases in patients with moth repellent exposure?
Why is emesis not recommended in most cases of mothball ingestion?
Why is emesis not recommended in most cases of mothball ingestion?
What is a characteristic of paradichlorobenzene mothballs compared to naphthalene mothballs?
What is a characteristic of paradichlorobenzene mothballs compared to naphthalene mothballs?
How does naphthalene absorption through the skin increase?
How does naphthalene absorption through the skin increase?
In individuals with glucose-6-phosphate dehydrogenase (G-6-PD) deficiency, which metabolites of naphthalene are especially toxic?
In individuals with glucose-6-phosphate dehydrogenase (G-6-PD) deficiency, which metabolites of naphthalene are especially toxic?
What clinical sign is specifically associated with inhalation of paradichlorobenzene?
What clinical sign is specifically associated with inhalation of paradichlorobenzene?
What is the recommended treatment for hemolysis caused by naphthalene exposure?
What is the recommended treatment for hemolysis caused by naphthalene exposure?
What should be administered after inducing emesis in asymptomatic patients who have ingested mothballs?
What should be administered after inducing emesis in asymptomatic patients who have ingested mothballs?
What test can be performed to distinguish between naphthalene and paradichlorobenzene mothballs?
What test can be performed to distinguish between naphthalene and paradichlorobenzene mothballs?
How does paradichlorobenzene absorption increase when ingested?
How does paradichlorobenzene absorption increase when ingested?
What is the most common cause of death in barbiturate poisoning?
What is the most common cause of death in barbiturate poisoning?
Which drug is generally preferred over barbiturates to control convulsions in camphor poisoning?
Which drug is generally preferred over barbiturates to control convulsions in camphor poisoning?
What is the suggested treatment when the quantity of camphor ingested is unknown?
What is the suggested treatment when the quantity of camphor ingested is unknown?
What is the major toxic event that follows barbiturate ingestion?
What is the major toxic event that follows barbiturate ingestion?
What is the effect of barbiturates on the GABA-Cl- ionophore complex in the CNS?
What is the effect of barbiturates on the GABA-Cl- ionophore complex in the CNS?
What is a potentially serious problem that follows toxic ingestions of barbiturates?
What is a potentially serious problem that follows toxic ingestions of barbiturates?
What is the characteristic effect of shorter-acting barbiturates compared to phenobarbital?
What is the characteristic effect of shorter-acting barbiturates compared to phenobarbital?
What is a common contributing factor to barbiturate poisoning?
What is a common contributing factor to barbiturate poisoning?
What is a potential effect of chronic barbiturate (ab)use?
What is a potential effect of chronic barbiturate (ab)use?
What is the suggested immediate treatment for camphor poisoning?
What is the suggested immediate treatment for camphor poisoning?
Which of the following is a common sign of acute benzodiazepine toxicity?
Which of the following is a common sign of acute benzodiazepine toxicity?
What is the primary mechanism of action of benzodiazepines in the nervous system?
What is the primary mechanism of action of benzodiazepines in the nervous system?
What is the main toxic effect of chloral hydrate and its metabolites in the CNS?
What is the main toxic effect of chloral hydrate and its metabolites in the CNS?
What is the recommended management for acute overdose of chloral hydrate?
What is the recommended management for acute overdose of chloral hydrate?
Which drug is considered a specific antidote for benzodiazepine overdose?
Which drug is considered a specific antidote for benzodiazepine overdose?
What is a common indication of barbiturate toxicity in moderate cases?
What is a common indication of barbiturate toxicity in moderate cases?
What is the effect of benzodiazepine overdose on motor performance?
What is the effect of benzodiazepine overdose on motor performance?
Which intervention is recommended for hypotension in acute barbiturate overdose?
Which intervention is recommended for hypotension in acute barbiturate overdose?
What is the most reliable indicator of clinical toxicity in barbiturate poisoning?
What is the most reliable indicator of clinical toxicity in barbiturate poisoning?
Which treatment has been shown to greatly increase phenobarbitone elimination?
Which treatment has been shown to greatly increase phenobarbitone elimination?