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Questions and Answers
What does a respiratory rate of 28 bpm indicate?
What does a respiratory rate of 28 bpm indicate?
Which ventilation method is characterized by an inverse ratio of inspiration to expiration?
Which ventilation method is characterized by an inverse ratio of inspiration to expiration?
What is a primary goal of APRV?
What is a primary goal of APRV?
What distinguishes SIMV from AC ventilation?
What distinguishes SIMV from AC ventilation?
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Which complication can result from APRV due to its method of ventilation?
Which complication can result from APRV due to its method of ventilation?
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What is the most common risk factor for developing a pulmonary embolism?
What is the most common risk factor for developing a pulmonary embolism?
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Which of the following symptoms is NOT typically associated with pulmonary embolism?
Which of the following symptoms is NOT typically associated with pulmonary embolism?
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Which diagnostic test is considered the most definitive for diagnosing pulmonary embolism?
Which diagnostic test is considered the most definitive for diagnosing pulmonary embolism?
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What is a common complication associated with Assist Control (AC) ventilation mode?
What is a common complication associated with Assist Control (AC) ventilation mode?
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Which of the following treatments would be most appropriate to reduce the risk of bleeding in a patient with pulmonary embolism?
Which of the following treatments would be most appropriate to reduce the risk of bleeding in a patient with pulmonary embolism?
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What immediate action should be taken for ongoing verification after intubation?
What immediate action should be taken for ongoing verification after intubation?
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What should be monitored in a patient receiving assisted ventilation to prevent respiratory alkalosis?
What should be monitored in a patient receiving assisted ventilation to prevent respiratory alkalosis?
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Which of the following is a significant nursing consideration when managing a patient with pulmonary embolism?
Which of the following is a significant nursing consideration when managing a patient with pulmonary embolism?
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What is the primary purpose of the cuff in a tracheostomy tube?
What is the primary purpose of the cuff in a tracheostomy tube?
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Which of the following symptoms indicates airway obstruction?
Which of the following symptoms indicates airway obstruction?
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What is the appropriate initial intervention for a patient showing signs of airway obstruction?
What is the appropriate initial intervention for a patient showing signs of airway obstruction?
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What does a fully inflated pilot balloon indicate about the tracheostomy cuff?
What does a fully inflated pilot balloon indicate about the tracheostomy cuff?
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What should be done if a tracheostomy tube becomes dislodged?
What should be done if a tracheostomy tube becomes dislodged?
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When performing tracheostomy care, which step is essential during insertion?
When performing tracheostomy care, which step is essential during insertion?
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Which monitoring tool is essential for confirming tracheostomy tube placement?
Which monitoring tool is essential for confirming tracheostomy tube placement?
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What is a potential complication of a fully inflated cuff in a tracheostomy tube?
What is a potential complication of a fully inflated cuff in a tracheostomy tube?
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What is a primary characteristic of hypovolemic shock?
What is a primary characteristic of hypovolemic shock?
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Which of the following treatments is most important for managing anaphylactic shock?
Which of the following treatments is most important for managing anaphylactic shock?
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Which condition is associated with cardiogenic shock?
Which condition is associated with cardiogenic shock?
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What is a common manifestation of neurogenic shock?
What is a common manifestation of neurogenic shock?
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Which medication is primarily used to raise blood pressure in obstructive shock?
Which medication is primarily used to raise blood pressure in obstructive shock?
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What is indicated by a decreased cardiac output in the context of shock?
What is indicated by a decreased cardiac output in the context of shock?
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In which type of shock does severe vasodilation occur, leading to hypotension?
In which type of shock does severe vasodilation occur, leading to hypotension?
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What immediate action should be taken for a tension pneumothorax?
What immediate action should be taken for a tension pneumothorax?
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Which of the following is a complication of sepsis?
Which of the following is a complication of sepsis?
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What is the purpose of using inotropic medications in cardiogenic shock?
What is the purpose of using inotropic medications in cardiogenic shock?
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Which condition is characterized by a normal pH level but abnormal PaCO2 and HCO3 levels?
Which condition is characterized by a normal pH level but abnormal PaCO2 and HCO3 levels?
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What is the primary cause of respiratory alkalosis?
What is the primary cause of respiratory alkalosis?
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What is a significant manifestation of metabolic acidosis?
What is a significant manifestation of metabolic acidosis?
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Which symptom is NOT typically associated with hyperglycemia in diabetes mellitus?
Which symptom is NOT typically associated with hyperglycemia in diabetes mellitus?
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What is the primary treatment intervention for diabetic ketoacidosis (DKA)?
What is the primary treatment intervention for diabetic ketoacidosis (DKA)?
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In cases of adrenal insufficiency, what is the most effective treatment?
In cases of adrenal insufficiency, what is the most effective treatment?
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Which understanding is incorrect regarding the differences between DKA and HHS?
Which understanding is incorrect regarding the differences between DKA and HHS?
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What strategy is utilized to treat respiratory acidosis?
What strategy is utilized to treat respiratory acidosis?
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In Syndrome of Inadequate Antidiuretic Hormone Secretion (SIADH), which of the following is a hallmark manifestation?
In Syndrome of Inadequate Antidiuretic Hormone Secretion (SIADH), which of the following is a hallmark manifestation?
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What is a common complication of untreated diabetes mellitus type II?
What is a common complication of untreated diabetes mellitus type II?
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Which of the following is NOT a recommended intervention for a patient with respiratory alkalosis?
Which of the following is NOT a recommended intervention for a patient with respiratory alkalosis?
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Which intervention would best help manage hypoglycemia?
Which intervention would best help manage hypoglycemia?
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What is the expected consequence of metabolic alkalosis on potassium levels?
What is the expected consequence of metabolic alkalosis on potassium levels?
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Study Notes
Airway Management
- A patent airway, adequate ventilation, and adequate perfusion are essential for survival
- Oropharyngeal airway (OPA) and nasopharyngeal airway (NPA) are emergency airway devices, endotracheal airway (ETA) with intubation is preferred
- Signs and symptoms of airway obstruction include stridor, choking, wheezing, nasal flaring, restlessness, and loss of consciousness
- Interventions for airway obstruction include Heimlich maneuver, repositioning the head, intubation, tracheostomy, and cricothyroidotomy
Tracheostomy Tube
- Provides long-term airway assistance
- Components include cuff, pilot balloon, and obturator
- Cuff seals the airway and is inflated using the pilot balloon which prevents aspiration
- A flat pilot balloon indicates a deflated cuff, which may lead to trach dislodgement
- The obturator is used to guide the tube back into place if dislodged
- Always keep the obturator at the bedside
Tracheostomy Care
- Insertion requires a respiratory therapist, vital sign and SpO2 monitoring, patent IV, sedation, supine positioning, bedside suction, and emergency equipment
- Post-operative care includes confirming placement with a chest X-ray, end-tidal CO2 monitoring, removing the obturator, ensuring balloon and cuff inflation, securing the trach with suture, and monitoring vital signs, SpO2, and ventilator settings
Pulmonary Embolism
- A clot occlusion in a pulmonary vessel
- Causes an inflammatory response, leading to increased right ventricle workload and potential pulmonary hypertension
- Risk factors include immobility, recent surgery, history of DVT, malignancy, obesity, oral contraceptives, smoking, prolonged travel, heart failure, pregnancy, and clotting disorders
- Signs and symptoms include chest pain, hemoptysis, hypoxia, tachypnea, use of accessory muscles, respiratory alkalosis, anxiety, tachycardia, hypotension, cough, wheezing, crackles, fever, syncope, confusion, and accentuation of pulmonic heart sounds
- Diagnostics include elevated BNP, PT/INR, and aPTT, spiral CT with pulmonary angiography, echocardiography, D-dimer, troponin, chest X-ray, ECG, and V/Q scan
- Treatments options include oxygen, rest and activity balance, inferior vena cava filter placement, pulmonary embolectomy, fibrinolytic therapy, heparin, and enoxaparin
- Concerns associated with PE treatment include risk of bleeding and obstructive shock
Ventilation
- Endotracheal tube insertion is performed by a CRNA, anesthesiologist, nurse practitioner, physician assistant, or physician for mechanical ventilation
- Indications for ventilation include hypoxia, hypercapnia, trauma, and edema
- Nursing considerations for ventilation include positioning, pre-oxygenation, sedation, and paralytics
- Verification of ET tube placement requires chest X-ray, CO2 detection, bilateral chest rise, lung and abdominal auscultation, and improvement in oxygenation parameters
- Intubation attempt should be no longer than 30 seconds
- Continued ET tube verification is essential by marking the tube to teeth/lips
Modes of Ventilation
- Mechanical ventilation modes include Assist Control (AC), Pressure Control (PC), Airway Pressure Release Ventilation (APRV), and Synchronized Intermittent Mandatory Ventilation (SIMV)
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Assist Control (AC)
- Provides complete control of oxygenation parameters including tidal volume, FiO2, RR, and PEEP
- Most common ventilation setting
- Ventilator inflates the lungs with the ordered tidal volume on inspiration; patient exhales once tidal volume is reached
- Complications include barotrauma/pneumothorax and respiratory alkalosis
- Nursing Interventions include increasing sedation for respiratory alkalosis, adding paralytics, and ensuring peak pressure alarm is activated for barotrauma
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Pressure Control (PC)
- Ventilator inflates lungs with the ordered pressure on inspiration; patient exhales once pressure is reached
- Complications include respiratory acidosis and hypoventilation
- Patients are heavily sedated and may be on paralytics
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Airway Pressure Release Ventilation (APRV)
- Goal is to improve oxygenation and open alveoli, commonly used for ARDS patients
- Combines AC and PC ventilation principles
- Increases tidal volume, allowing for spontaneous breathing
- May or may not require sedation
- Uses inverse ratio ventilation where inspiration is extended and expiration is shortened
- Bilevel ventilation provides two different pressures: one for inspiration and a lower pressure for exhalation
- Complications include hypercapnia and respiratory acidosis
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Synchronized Intermittent Mandatory Ventilation (SIMV)
- Delivers preset tidal volume with a preset number of breaths, allowing spontaneous breaths in between
- Encourages independent breathing and combats muscle atrophy
- SIMV takes over when the patient is unable to complete their breath, whereas AC continues to deliver preset settings
- Used for weaning from mechanical ventilation
Acid-Base Balance
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Respiratory Acidosis
- Caused by inadequate breathing (hypoventilation) resulting in hypoxia
- Signs and symptoms include dyspnea, rapid shallow respirations, hyperkalemia, dysrhythmias, decreased BP, and disorientation
- Treatments include addressing the cause (hyperkalemia treatment) and increasing RR/improving ventilation
- Interventions include elevating head of bed (HOB), breathing exercises, incentive spirometry, oxygen therapy, and metabolic panel monitoring
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Respiratory Alkalosis
- Caused by ineffective breathing (hyperventilation)
- Signs and Symptoms include increased RR and depth, increased HR, decreased or normal BP, hypokalemia, numbness/tingling of extremities, seizures, hyper reflexes, and muscle cramps
- Treatments include treating the cause (hypocalcemia and hypokalemia) and decreasing RR
- Interventions include using a brown paper bag to rebreathe CO2, IV fluids, breathing exercises, pain and anxiety management, sedation, and metabolic panel monitoring
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Metabolic Acidosis
- Caused by excessive hydrogen ions (DKA, renal failure, dehydration, diarrhea)
- Manifestations include decreased BP, hyperkalemia, muscle twitching, warm flushed skin, N/V, decreased muscle tone and reflexes, and Kussmaul respirations
- Treatments include treating the cause (hyperkalemia treatment) and increasing RR
- Interventions include IV fluids and sodium bicarbonate for DKA, dialysis for renal failure, diphenoxylate/atropine for diarrhea, and antiemetics for vomiting
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Metabolic Alkalosis
- Caused by loss of hydrogen ions (body fluids), due to vomiting, excessive gastric suction, and diuretics
- Signs and symptoms include restlessness followed by lethargy, dysrhythmias (tachycardia), compensatory hypoventilation, confusion, N/V/D, tremors, and hypokalemia
- Treatment/interventions include antiemetics, IV fluids and electrolyte replacements, dialysis for severe cases, and treatment of hypocalcemia and hypokalemia
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Important Notes
- Acidosis leads to hyperkalemia
- Alkalosis leads to hypokalemia
Endocrine
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Diabetes Mellitus Type 1 (DM I)
- Autoimmune disease requiring lifelong insulin therapy
- Signs and symptoms include the 3 Ps (polyuria, polydipsia, polyphagia), weight loss, weakness, and fatigue
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Diabetes Mellitus Type 2 (DM II)
- Beta cells produce less insulin and body cells become insulin resistant
- Can be managed with diet, lifestyle changes, and weight loss
- Risk factors include family history, overweight, and advanced age
- Signs and symptoms include prolonged wound healing, visual problems, fatigue, and recurrent infections
Complications of Diabetes
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Hypoglycemia
- Blood sugar less than 70 mg/dL
- Signs and symptoms include anxiety, sweating, hunger, confusion, blurred/double vision, shakiness, irritability, cool clammy skin
- Treatments include 15g of simple carbs (fruit juice or regular soft drink), recheck in 15 minutes, and 50% dextrose 20-50 mL IV push for immediate resolution
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Diabetic Ketoacidosis (DKA)
- Blood glucose levels >300 to 600 mg/dL
- Presence of ketone bodies in blood and urine
- Monitor Cr, BUN, Hct, and BMP
- Metabolic acidosis, Kussmaul respirations, and fruity breath
- Manifestations include polyuria, polydipsia, blurred vision, weakness, headache, hypotension, weak/rapid pulse, and hyperkalemia
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Hyperosmolar Hyperglycemic State (HHS)
- Blood glucose levels > 600
- Altered mental status (usually unresponsive)
- Associated with acute illness, uncontrolled diabetes, and glucocorticoid use
- Manifestations include serum osmolality >320, hypotension, tachycardia, variable neurological symptoms
- Management includes fluids, electrolyte correction, and insulin
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DKA vs HHS
- DKA is faster onset than HHS.
- DKA: pH is acidic, HHS: pH is normal.
- DKA: Ketones are present, HHS: Ketones are absent or minimal.
Adrenal Insufficiency
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Addison's Disease
- Treated with steroid therapy
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Cushing's Disease
- Treated with surgical removal of the adrenal gland
- Lifelong steroid therapy is required because the adrenal glands no longer produce cortisol
Pituitary Disorders
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Diabetes Insipidus
- Hyposecretion of antidiuretic hormone (ADH)
- Manifestations include polydipsia, polyuria, and dehydration
- Treatment includes fluid replacement and desmopressin
- Monitor vital signs, neurologic status, cardiovascular status, electrolytes, I/O, weight, and urine specific gravity
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Syndrome of Inadequate Antidiuretic Hormone Secretion (SIADH)
- Hypersecretion of antidiuretic hormone (ADH)
- Manifestations include fluid retention, edema, serum hypoosmolality, concentrated urine with normal/increased intravascular volume
- Diagnosis based on serum and urine osmolarity
- Treatment: treat underlying cause, fluid restriction (unless third spacing is present), furosemide, seizure and fall precautions, and laboratory monitoring
Shock
- Stages of Shock: initial, compensatory, progressive, and refractory
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Types of Shock
- Hypovolemic shock
- Cardiogenic shock
- Distributive shock
- Obstructive shock
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Hypovolemic Shock
- Low circulating blood due to trauma or fluid loss
- Management includes labs (ABGs, H&H, metabolic panel, lactate), 100% O2 via non-rebreather, intubation, large bore IVs (18 gauge), central line, and fluid rehydration
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Cardiogenic Shock
- Heart is unable to pump correctly due to MI, valvular dysfunction, or severe HF
- Manifestations include decreased cardiac output, decreased BP, narrow pulse pressure, and pulmonary congestion
- Treatment includes labs, oxygen, fluids, vasoactive medications, inotropes, diuretics, and morphine
- Limit activity and treat the underlying cause
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Distributive Shock
- Severe vasodilation causing decreased BP and inadequate blood flow
- Subtypes: Sepsis, Anaphylaxis, Neurogenic Shock
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Anaphylactic Shock
- Allergic response
- Management includes removing the trigger, 100% O2, IV fluids, epinephrine, corticosteroids, bronchodilators, and antihistamines
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Neurogenic Shock
- Spinal cord injury above T5
- Severe hypotension and bradycardia due to vasodilation
- Management includes IV fluids, vasoactive drips, atropine, heparin/lovenox, and transcutaneous/transvenous pacing
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Obstructive Shock
- Obstruction that prevents proper blood flow
- Subtypes: Tension pneumothorax, Cardiac tamponade, Severe valvular disease
- Management: neuro checks, intubation/ventilation, ABGs, vasoactive meds, anticoagulants, thrombolytics, tension pneumothorax treatment (chest tube), pericardiocentesis, suction thrombectomy, and valvular replacement
Vasoactive Medications
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Dobutamine
- Positive inotropic effect, used for heart failure, not titratable
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Dopamine
- Increases HR and BP, first-line treatment for shock
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Epinephrine
- Increases HR and BP
- Continuous infusion is a last resort due to vasoconstrictive effects
- Use alongside dopamine if necessary
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Norepinephrine
- Increases BP only
- Also known as Levophed
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Phenylephrine
- Increases BP only
- Short-term use, also known as Neo or Neo-Synephrine
Sepsis
- Sepsis 6: O2, blood cultures, antibiotics, fluids, lactate, and urine output
- Complications of sepsis include disseminated intravascular coagulation (DIC) and multiple organ dysfunction syndrome (MODS)
- Sepsis can arise from infection or systemic inflammatory response syndrome (SIRS)
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Description
Test your knowledge on respiratory rates and ventilation methods with this quiz. It covers key concepts like APRV, SIMV, and the implications of different ventilation strategies. Understand the nuances of respiratory support in clinical settings.