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Questions and Answers
What is the primary cause of increased blood glucose levels in diabetes mellitus?
What is the primary cause of increased blood glucose levels in diabetes mellitus?
- Decreased glucose excretion by the kidneys
- Increased dietary intake of glucose
- Increased glucose production by the liver
- Impaired insulin function leading to decreased glucose utilization (correct)
Which of the following is a consequence of increased fat mobilization in diabetes mellitus?
Which of the following is a consequence of increased fat mobilization in diabetes mellitus?
- Increased protein synthesis
- Metabolic acidosis (correct)
- Decreased ketone formation
- Decreased lipid levels
What is the primary reason for impaired protein utilization in diabetes mellitus?
What is the primary reason for impaired protein utilization in diabetes mellitus?
- Decreased protein catabolism
- Decreased amino acid transport into cells (correct)
- Increased insulin levels
- Increased protein synthesis
Which of the following is a consequence of increased glucose excretion by the kidneys in diabetes mellitus?
Which of the following is a consequence of increased glucose excretion by the kidneys in diabetes mellitus?
What is the primary cause of atherosclerosis and cardiovascular complications in diabetes mellitus?
What is the primary cause of atherosclerosis and cardiovascular complications in diabetes mellitus?
Which of the following is a consequence of long-standing diabetes mellitus?
Which of the following is a consequence of long-standing diabetes mellitus?
What is the primary cause of protein deposits in the basement membrane in diabetes mellitus?
What is the primary cause of protein deposits in the basement membrane in diabetes mellitus?
What is the primary reason for increased protein catabolism in diabetes mellitus?
What is the primary reason for increased protein catabolism in diabetes mellitus?
Which of the following is a consequence of impaired inflammatory processes in diabetes mellitus?
Which of the following is a consequence of impaired inflammatory processes in diabetes mellitus?
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