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Questions and Answers
What do adrenergic agents do?
What do adrenergic agents do?
Which term is synonymous with adrenergic agents?
Which term is synonymous with adrenergic agents?
What is the function of the alpha-1 receptor?
What is the function of the alpha-1 receptor?
Treatment of nasal congestion or hypotension; causes mydriasis during ophthalmic examinations.
What is the role of the alpha-2 receptor?
What is the role of the alpha-2 receptor?
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What is beta-1 receptor used for?
What is beta-1 receptor used for?
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What medical conditions are treated by beta-2 receptors?
What medical conditions are treated by beta-2 receptors?
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What do adrenergic antagonists do?
What do adrenergic antagonists do?
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What do cholinergic agents mimic?
What do cholinergic agents mimic?
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What do cholinergic-blocking agents inhibit?
What do cholinergic-blocking agents inhibit?
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How do benzodiazepines function?
How do benzodiazepines function?
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What is the effect of barbiturates?
What is the effect of barbiturates?
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What is the goal of anti-seizure pharmacology?
What is the goal of anti-seizure pharmacology?
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What does GABA stand for?
What does GABA stand for?
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What is Phenobarbital's unique ability?
What is Phenobarbital's unique ability?
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What do hydantoin and phenytoin-like drugs desensitize?
What do hydantoin and phenytoin-like drugs desensitize?
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What do succinimides do?
What do succinimides do?
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What is the mechanism of action for tricyclic antidepressants?
What is the mechanism of action for tricyclic antidepressants?
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What do selective serotonin reuptake inhibitors (SSRIs) do?
What do selective serotonin reuptake inhibitors (SSRIs) do?
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What do serotonin-norepinephrine reuptake inhibitors (SNRIs) inhibit?
What do serotonin-norepinephrine reuptake inhibitors (SNRIs) inhibit?
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What do monoamine oxidase inhibitors (MAOIs) inhibit?
What do monoamine oxidase inhibitors (MAOIs) inhibit?
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What is the function of glutamate inhibitors/serotonin receptor antagonists?
What is the function of glutamate inhibitors/serotonin receptor antagonists?
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What do CNS stimulants do?
What do CNS stimulants do?
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What do phenothiazines do?
What do phenothiazines do?
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What is the action of nonphenothiazines?
What is the action of nonphenothiazines?
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What do atypical antipsychotics block?
What do atypical antipsychotics block?
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What do opioid agonists do?
What do opioid agonists do?
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What do opioid antagonists compete with?
What do opioid antagonists compete with?
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What do NSAIDs inhibit?
What do NSAIDs inhibit?
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What are triptans/ergot alkaloids thought to do?
What are triptans/ergot alkaloids thought to do?
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What do esters/amides block?
What do esters/amides block?
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What do inhalation gaseous agents produce?
What do inhalation gaseous agents produce?
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What is the function of a depolarizing blocker?
What is the function of a depolarizing blocker?
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Study Notes
Adrenergic Agents and Related Terms
- Adrenergic Agents: Stimulate the sympathetic nervous system, inducing fight-or-flight response symptoms.
- Sympathomimetics: Another term for Adrenergic Agents, highlighting their ability to mimic sympathetic nervous system actions.
- Alpha-1 Receptor: Used for treating nasal congestion and hypotension, causing mydriasis (pupil dilation) during eye exams.
- Alpha-2 Receptor: Primarily used for hypertension treatment; reduces norepinephrine release, lowering blood pressure.
- Beta-1 Receptor: Critical in treating cardiac arrest, heart failure (HF), and shock.
- Beta-2 Receptors: Effective in managing asthma symptoms and preventing premature labor contractions.
- Adrenergic Antagonists: Block adrenergic receptors, with the capability to target either alpha or beta receptors.
Cholinergic Agents and Blockers
- Cholinergic Agents: Mimic parasympathetic nervous system actions, promoting rest-and-digest responses.
- Parasympathomimetics: Another name for Cholinergic Agents, reflecting their similar functions.
- Cholinergic-Blocking Agents: Inhibit parasympathetic impulses and suppress the fight-or-flight response.
- Anticholinergics: The class of drugs that includes Cholinergic-Blocking Agents.
Anxiolytics and Anticonvulsants
- Benzodiazepines: Bind to GABA receptor-chloride channel complex, promoting calmness and sedation.
- Barbiturates: Intensify GABA's effects across the brain by binding to the GABA receptor-chloride channel.
- Anxiety and Insomnia Treatment: Mechanism believed linked to D2 dopamine receptors and serotonin receptor activities.
- Anti-seizure Pharmacology: Aims to suppress neuronal activity to prevent abnormal firing.
- GABA: The primary inhibitory neurotransmitter in the brain.
- Phenobarbital: Allows the suppression of abnormal neuronal discharges without sedation.
Seizure Medications
- Hydantoin and Phenytoin-Like Drugs: Desensitize sodium channels, delaying neuron depolarization.
- Succinimides: Suppress seizures by delaying calcium influx into neurons.
Antidepressants
- Tricyclic Antidepressants: Inhibit the reuptake of norepinephrine and serotonin at presynaptic neurons.
- Selective Serotonin Reuptake Inhibitors (SSRIs): Slow the reuptake of serotonin into presynaptic nerve terminals, enhancing mood.
- Serotonin-norepinephrine Reuptake Inhibitors (SNRIs): Raise serotonin and norepinephrine levels, improving mood.
- Monoamine Oxidase Inhibitors (MAOIs): Prevent monoamine oxidase from terminating neurotransmitter actions.
CNS Stimulants and Antipsychotics
- Glutamate Inhibitor / Serotonin Receptor Antagonist: Alters neuronal ionic activity and impacts neurotransmitter release and uptake.
- CNS Stimulants: Heighten alertness and increase focus by stimulating specific CNS areas.
- Phenothiazines: Prevent dopamine and serotonin from occupying essential receptor sites in neurologic pathways.
- NonPhenothiazines: Block postsynaptic D2 dopamine receptors for antipsychotic effects.
- Atypical Antipsychotics: Block both D2 and serotonin (5-HT) receptors alongside alpha-adrenergic receptors.
Pain Management and Opioids
- Opioid Agonists: Bind to opioid receptors, resulting in various biological responses.
- Opioid Antagonists: Compete with opioids for receptor sites, blocking their effects.
- NSAIDs: Inhibit cyclooxygenase, preventing the formation of pain-inducing prostaglandins.
- Triptans / Ergot Alkaloids: Serotonin agonists thought to constrict specific intracranial blood vessels, relieving migraine pain.
- Esters / Amides: Block sodium channels, inhibiting neuronal pain impulses.
- Inhalation Gaseous Agents: Create analgesic effects through interactions with the CNS's pain mechanisms.
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Test your knowledge on key pharmacological terms with these flashcards focusing on drug classes. Each card presents a word along with its definition, providing a quick way to learn and reinforce your understanding of pharmacology concepts. Perfect for students and professionals alike!