Podcast
Questions and Answers
What is one of the treatment goals for heart failure in terms of reducing morbidity?
What is one of the treatment goals for heart failure in terms of reducing morbidity?
- Increasing afterload
- Relief of symptoms (correct)
- Decreasing blood volume
- Reducing preload
What is NOT the treatment goals for heart failure in terms of reducing morbidity?
What is NOT the treatment goals for heart failure in terms of reducing morbidity?
- Maintaining optimal blood glucose levels (correct)
- Relief of symptoms
- Avoid hospital admission
- Treat underlying or aggravating cause
Which class of drugs DO NOT reduces both preload and afterload in the treatment of heart failure?
Which class of drugs DO NOT reduces both preload and afterload in the treatment of heart failure?
- ACE inhibitors/ARBs
- Aldosterone antagonists
- Diuretics (correct)
- β-blockers
Which class of drugs improves cardiac contractility in the treatment of heart failure?
Which class of drugs improves cardiac contractility in the treatment of heart failure?
Which class of drugs DO NOT acts as arterial dilators in the treatment of heart failure?
Which class of drugs DO NOT acts as arterial dilators in the treatment of heart failure?
Which class of drugs reduces preload by reducing circulatory volume in the treatment of heart failure?
Which class of drugs reduces preload by reducing circulatory volume in the treatment of heart failure?
Which class of drugs reduces afterload by preventing A II formation or blocking the effects of A II in the treatment of heart failure?
Which class of drugs reduces afterload by preventing A II formation or blocking the effects of A II in the treatment of heart failure?
Which class of drugs increases myocardial contractility in the treatment of heart failure?
Which class of drugs increases myocardial contractility in the treatment of heart failure?
Which enzyme does Digoxin inhibit to increase intracellular Ca2+?
Which enzyme does Digoxin inhibit to increase intracellular Ca2+?
What is the main clinical use of Digoxin?
What is the main clinical use of Digoxin?
What is the half-life of Digoxin?
What is the half-life of Digoxin?
What are the cardiac adverse effects of Digoxin?
What are the cardiac adverse effects of Digoxin?
What is the recommended monitoring for Digoxin therapy?
What is the recommended monitoring for Digoxin therapy?
What are the possible reasons to monitor plasma concentrations of Digoxin?
What are the possible reasons to monitor plasma concentrations of Digoxin?
Which electrolyte imbalance increases the risk of digitalis-induced arrhythmia?
Which electrolyte imbalance increases the risk of digitalis-induced arrhythmia?
Which condition can lead to decreased elimination of Digoxin?
Which condition can lead to decreased elimination of Digoxin?
What is the recommended treatment for ventricular arrhythmias associated with Digoxin toxicity?
What is the recommended treatment for ventricular arrhythmias associated with Digoxin toxicity?
Which enzyme do bipyridines inhibit to increase cAMP levels?
Which enzyme do bipyridines inhibit to increase cAMP levels?
What is the main route of excretion for bipyridines?
What is the main route of excretion for bipyridines?
What are the common adverse effects of bipyridines?
What are the common adverse effects of bipyridines?
Which one of these drugs is NOT a β1 agonist?
Which one of these drugs is NOT a β1 agonist?
Which one of these is NOT a clinical use of β1 agonists?
Which one of these is NOT a clinical use of β1 agonists?
Which one of these adverse effects is NOT associated with β1 agonists?
Which one of these adverse effects is NOT associated with β1 agonists?
Which one of these is the primary mechanism of action (MOA) of beta-1 agonists?
Which one of these is the primary mechanism of action (MOA) of beta-1 agonists?
Which diuretic is recommended for mild edema?
Which diuretic is recommended for mild edema?
Which diuretic is recommended for moderate and severe edema?
Which diuretic is recommended for moderate and severe edema?
Which diuretic is reported to reduce morbidity and mortality when taken with ACE inhibitors and other agents?
Which diuretic is reported to reduce morbidity and mortality when taken with ACE inhibitors and other agents?
Which of the following best describes the mechanism of action of diuretics?
Which of the following best describes the mechanism of action of diuretics?
Which of the following is a common reason for diuretic resistance in heart failure management?
Which of the following is a common reason for diuretic resistance in heart failure management?
How do diuretics reduce peripheral and pulmonary edema in heart failure?
How do diuretics reduce peripheral and pulmonary edema in heart failure?
Which part of the nephron do thiazide diuretics primarily act on?
Which part of the nephron do thiazide diuretics primarily act on?
Which type of diuretic inhibits the sodium channels involved in sodium reabsorption in the cortical collecting duct?
Which type of diuretic inhibits the sodium channels involved in sodium reabsorption in the cortical collecting duct?
Where do loop diuretics primarily act in the nephron?
Where do loop diuretics primarily act in the nephron?
Which of the following is NOT an example of a loop diuretic?
Which of the following is NOT an example of a loop diuretic?
What is the main route of administration for loop diuretics?
What is the main route of administration for loop diuretics?
What is the main clinical use of loop diuretics?
What is the main clinical use of loop diuretics?
Which transporter do loop diuretics inhibit in the thick ascending limb of the loop of Henle?
Which transporter do loop diuretics inhibit in the thick ascending limb of the loop of Henle?
Which of the following is the primary route of administration for thiazide diuretics?
Which of the following is the primary route of administration for thiazide diuretics?
What is the peak effect duration of thiazide diuretics?
What is the peak effect duration of thiazide diuretics?
Which of the following is NOT a mechanism of action of thiazide diuretics?
Which of the following is NOT a mechanism of action of thiazide diuretics?
Which of the following is the primary indication for thiazide diuretics?
Which of the following is the primary indication for thiazide diuretics?
Which electrolyte imbalance is associated with an increased risk of digoxin toxicity due to thiazide use?
Which electrolyte imbalance is associated with an increased risk of digoxin toxicity due to thiazide use?
Which of the following adverse effects is mostly associated with loop diuretics?
Which of the following adverse effects is mostly associated with loop diuretics?
Which of the following adverse effects is mostly associated with thiazide diuretics?
Which of the following adverse effects is mostly associated with thiazide diuretics?
Which of the following adverse effect is for either loop diuretics or thiazide diuretics?
Which of the following adverse effect is for either loop diuretics or thiazide diuretics?
Which of the following adverse effects is NOT associated with K+ sparing diuretics?
Which of the following adverse effects is NOT associated with K+ sparing diuretics?
Which of the following adverse effects is associated with EnaC blockers?
Which of the following adverse effects is associated with EnaC blockers?
Which organ is primarily responsible for the metabolism of K+ sparing diuretics?
Which organ is primarily responsible for the metabolism of K+ sparing diuretics?
Which electrolyte imbalance is a concern when using K+ sparing diuretics in patients taking ACE inhibitors?
Which electrolyte imbalance is a concern when using K+ sparing diuretics in patients taking ACE inhibitors?
Which type of K+ sparing diuretic inhibits the aldosterone-induced production of sodium transport proteins (Aldosterone-dependent antagonists) at collecting tubules?
Which type of K+ sparing diuretic inhibits the aldosterone-induced production of sodium transport proteins (Aldosterone-dependent antagonists) at collecting tubules?
