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What is the primary cause of an acute periapical abscess?
What is the primary cause of an acute periapical abscess?
What characteristic is true regarding thermal stimulation in the presence of pulp necrosis?
What characteristic is true regarding thermal stimulation in the presence of pulp necrosis?
What clinical feature is associated with periodontitis in acute periapical abscess?
What clinical feature is associated with periodontitis in acute periapical abscess?
What type of radiographic finding is characteristic of an acute periapical abscess?
What type of radiographic finding is characteristic of an acute periapical abscess?
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Which of the following is NOT a complication of an untreated periapical abscess?
Which of the following is NOT a complication of an untreated periapical abscess?
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What histopathological finding is commonly seen in an acute periapical abscess?
What histopathological finding is commonly seen in an acute periapical abscess?
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How does a periapical abscess typically present when pus tracks palatally?
How does a periapical abscess typically present when pus tracks palatally?
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Which systemic manifestations may occur with an acute periapical abscess?
Which systemic manifestations may occur with an acute periapical abscess?
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What can happen to abscesses related to anterior maxillary teeth?
What can happen to abscesses related to anterior maxillary teeth?
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Which space may be involved when abscesses related to mandibular premolars or molars perforate the lingual plate of mandible?
Which space may be involved when abscesses related to mandibular premolars or molars perforate the lingual plate of mandible?
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What is a common treatment for an abscess related to a maxillary tooth?
What is a common treatment for an abscess related to a maxillary tooth?
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Which statement correctly describes cellulitis?
Which statement correctly describes cellulitis?
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What are common symptoms associated with cellulitis?
What are common symptoms associated with cellulitis?
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What is a common cause of Ludwig's angina?
What is a common cause of Ludwig's angina?
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What complication can result from the extension of cellulitis associated with maxillary teeth?
What complication can result from the extension of cellulitis associated with maxillary teeth?
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Which of the following is a characteristic sign of cavernous sinus thrombosis?
Which of the following is a characteristic sign of cavernous sinus thrombosis?
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What microorganisms are commonly associated with cellulitis infections?
What microorganisms are commonly associated with cellulitis infections?
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What component does streptokinase work to break down in the body?
What component does streptokinase work to break down in the body?
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What is the primary treatment for Ludwig's angina?
What is the primary treatment for Ludwig's angina?
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What does brawny induration in Ludwig's angina indicate?
What does brawny induration in Ludwig's angina indicate?
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What type of immune cells are predominantly found in a chronic periapical abscess?
What type of immune cells are predominantly found in a chronic periapical abscess?
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What is a common symptom of a chronic periapical abscess?
What is a common symptom of a chronic periapical abscess?
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Which of the following describes a key feature of periapical granuloma?
Which of the following describes a key feature of periapical granuloma?
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What indicates the presence of meningeal irritation in cavernous sinus thrombosis?
What indicates the presence of meningeal irritation in cavernous sinus thrombosis?
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What is the primary cause of lateral granuloma?
What is the primary cause of lateral granuloma?
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What is the most common cause of osteomyelitis?
What is the most common cause of osteomyelitis?
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Which of the following is NOT considered a cause of lateral granuloma?
Which of the following is NOT considered a cause of lateral granuloma?
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Which type of osteomyelitis is marked by the rapid destruction of bone due to a virulent bacterial strain?
Which type of osteomyelitis is marked by the rapid destruction of bone due to a virulent bacterial strain?
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Which clinical feature is associated with lateral granuloma?
Which clinical feature is associated with lateral granuloma?
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What is a common early symptom of acute osteomyelitis?
What is a common early symptom of acute osteomyelitis?
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What pathology occurs during acute osteomyelitis involving bone marrow?
What pathology occurs during acute osteomyelitis involving bone marrow?
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What is a common radiographical feature of a lateral granuloma?
What is a common radiographical feature of a lateral granuloma?
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Which histopathological feature is found in lateral granuloma?
Which histopathological feature is found in lateral granuloma?
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What demographic is often more affected by acute osteomyelitis?
What demographic is often more affected by acute osteomyelitis?
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What is a significant predisposing factor for developing osteomyelitis?
What is a significant predisposing factor for developing osteomyelitis?
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What is an expected complication of a periapical granuloma?
What is an expected complication of a periapical granuloma?
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What is typically seen in the granulation tissue of a periapical granuloma?
What is typically seen in the granulation tissue of a periapical granuloma?
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What happens to blood vessels in the bone during acute osteomyelitis?
What happens to blood vessels in the bone during acute osteomyelitis?
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Which zone of a periapical granuloma is typically more inflamed?
Which zone of a periapical granuloma is typically more inflamed?
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What type of treatment is commonly required for lateral granuloma?
What type of treatment is commonly required for lateral granuloma?
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What is the main feature of diffuse sclerosing osteomyelitis?
What is the main feature of diffuse sclerosing osteomyelitis?
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Which age group is primarily affected by focal sclerosing osteomyelitis?
Which age group is primarily affected by focal sclerosing osteomyelitis?
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What type of radiographic appearance is typical for focal sclerosing osteomyelitis?
What type of radiographic appearance is typical for focal sclerosing osteomyelitis?
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Which clinical feature is associated with chronic osteomyelitis with proliferative periostitis?
Which clinical feature is associated with chronic osteomyelitis with proliferative periostitis?
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What histological finding is common in the pathology of chronic low-grade osteomyelitis?
What histological finding is common in the pathology of chronic low-grade osteomyelitis?
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What is the primary cause of bone sclerosis in diffuse sclerosing osteomyelitis?
What is the primary cause of bone sclerosis in diffuse sclerosing osteomyelitis?
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Which of the following is a clinical feature of focal sclerosing osteomyelitis?
Which of the following is a clinical feature of focal sclerosing osteomyelitis?
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In what anatomical region is chronic osteomyelitis with proliferative periostitis most commonly found?
In what anatomical region is chronic osteomyelitis with proliferative periostitis most commonly found?
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Study Notes
Disease of Periapical Tissue
- Periapical area: The area surrounding the apical foramen of a dental root.
- Periapical Lesions: Inflammation surrounding the apical foramen of the dental root.
- Etiology of Periapical Lesions:
- Pulpitis
- Traumatic Injury
- Deep-seated restorations
- Infection through gingival crevice (periodontal diseases)
- Use of unsterilized instruments during root canal treatment
- Application of strong chemical antiseptics during root canal treatment
Periapical Periodontitis
- Inflammation of the periodontal ligament (PDL) around the apical portion of the root.
- Types:
- Acute periapical periodontitis
- Chronic periapical periodontitis
Acute Periapical Periodontitis
- Painful inflammation of the periodontium due to trauma, irritation, or infection through the root canal.
- Also called symptomatic apical periodontitis.
- Tenderness on percussion, severe pain causing difficulty closing teeth.
Etiology (Causes)
- Vital tooth:
- Occlusal trauma
- Wedging of foreign body
- Blow to the teeth
- Orthodontic pressure
- Non-vital tooth:
- Sequelae of pulpitis
- During root canal therapy (forcing irrigating irrigants or medicaments through the apical foramen)
- Extension of obturating material
- Perforation of the root
- Overinstrumentation
Clinical Features (Symptoms)
- Hot or cold substances do not cause pain
- Slight extrusion of the tooth from its socket
- Tenderness on mastication due to inflammatory edema in the PDL
- Severe pain due to external pressure forcing edema fluid against sensitized nerve endings
- Tender to percussion (preapical test).
- Widening of the PDL space (radiographic feature).
Histopathological Features
- Acute apical periodontitis characterized by an inflammatory reaction with engorged blood vessels and neutrophil accumulation.
- This inflammation is often transient if caused by acute trauma
- Progress to bone resorption if irritant not removed.
- Abscess formation possible with bacterial infection(acute preapical abscess/alveolar abscess)
Treatment
- Selective grinding if inflammation is due to occlusal trauma
- Extraction or root canal treatment (RCT) to drain exudate.
Acute Periapical Abscess (Dentoalveolar Abscess)
- An acute suppurative process in the dental periapical region.
- Accumulation of acute inflammatory cells at the apex of a non-vital tooth.
- Etiology:
- Infection (following pulpitis, hematogenous spread)
- Traumatic injury
- Pulp necrosis
- Mechanical/chemical manipulation in RCT
- More commonly, exacerbation of a chronic infection (e.g., periapical granuloma, "phoenix abscess").
Clinical Features for Acute Periapical Abscess
- History of pain (due to previous pulpitis)
- Large carious cavity/filling usually present
- Thermal stimulation does not cause pain (due to pulp necrosis)
- Tooth extremely painful and sensitive to percussion
- Tooth slightly extruded from its socket
- Intense throbbing pain as pus forms
- Systemic manifestations: Red and tender gingiva around the affected root, no swelling (inflammation confined within bone), lymphadenitis and fever possible
Radiographic Features
- Amorphous radiolucent area with irregular faintly margined borders in the periapical area of the dental root.
Histological Features of Acute Periapical Abscess
- Dense, almost solid masses of neutrophils (pus cells)
- Intermingling with inflammatory exudate, cellular debris, necrotic material, bacterial colonies, or histiocytes
- Dilated blood vessels in the PDL and bone marrow space
- Inflammatory infiltration of the bone marrow
- Resorption and degeneration of osteocytes (bone cells)
Complications of Untreated Periapical Abscess
- Pus may discharge directly into the oral cavity (sinus tract).
- Pus tracking palatally forming a palatal abscess
- Abscesses in molar regions penetrating buccal cortical plate
- Inflammatory edema and suppuration spreading into surrounding soft tissues (face, neck, cellulitis)
- Abscesses may drain through skin surface.
- Perforation of bone above tooth attachments, potentially leading to the eye or upper lip.
- Potential discharge into the maxillary sinus
- Potential involvement of sublingual, submandibular, and lateral pharyngeal spaces.
Cellulitis
- Diffuse inflammation of soft tissues, tending to spread through tissue spaces and fascial spaces .
- Clinically: painful, swelling of involved soft tissues, malaise, elevated temperature, trismus (difficulty opening mouth), swelling due to inflammatory edema, suppuration and abscess formation.
- Potential eye involvement is a serious concern (potential cavernous sinus thrombosis)
- Etiology is often related to spreading infection.
Chronic Periapical Abscess
- Features: May be a chronic condition or a sequela of acute infection. No acute symptoms, but tooth is felt high in socket, intraoral sinus formation, possible salty taste.
- Microscopically: Pus cavity with chronic inflammatory cells, lymphocytes, plasma cells, macrophages; fibrous tissue and capillaries are present; a fibrous capsule at the periphery
Periapical Granuloma
- Localized granulation tissue mass surrounding the apical foramen of a non-vital tooth; most common periapical lesion.
- Resulting from pulp death and diffusion of bacterial toxins from root canals into surrounding periradicular tissue (through apical and lateral canals).
- Possible presence of lateral/accessory root canals leading to lateral granuloma
- Treatment typically involves apicectomy and root canal treatment.
Chronic Periapical Periodontitis
- Occurs as a result of pulp death; diffusion of bacterial toxins from the root canals into the surrounding periradicular tissue occurs through apical and lateral canals.
- Most common periapical lesion.
- Chronic periapical inflammation characterized by granulation tissue.
Osteomyelitis
- Acute or chronic inflammatory process in bone marrow spaces or cortical surfaces; usually bacterial infection.
- Classification includes acute suppurative, chronic suppurative, chronic low-grade (sclerosing), focal sclerosing, and proliferative periostitis.
- Etiology includes dental infection, jaw fracture, or hematogenous spread.
- Predisposing factors include radiation damage, Paget's disease, osteoporosis, and systemic diseases (e.g., malnutrition, acute leukemia, uncontrolled diabetes, sickle cell anemia, chronic alcoholism).
- Acute osteomyelitis: Rapidly destructive inflammatory process; virulent bacterial strains commonly cause acute spreading of inflammatory process through medullary spaces in bone
- Acute osteomyelitis pathogenesis, pathology: inflammation, exudate spread, vascular thrombosis, necrosis, bacterial pus-filled pockets, lifting of periosteum (worsening necrosis) and osteoclastic activity (bone resorption).
Chronic Osteomyelitis
- Often a sequella of acute osteomyelitis.
- Characterized by a persistent low-grade infection, bone destruction, and granulation tissue formation, with little suppuration.
- Local bone sclerosis is more likely than in acute osteomyeliti.s and it is less likely to cause rapid spread of infectious agents
- Common sites are posterior mandible, and the disease process is characterized by low-grade pain, bad taste from pus, swelling with increased pain and discharge , along with increased mobility of affected teeth in some cases.
Diffuse Sclerosing Osteomyelitis
- Chronic low-grade inflammation of bone
- Occurs as a response to a low-grade stimulus
- Pathogenesis; bacterial infection causing formation of a mass of chronically inflamed granulation tissue, which then results in sclerosis of surrounding bone with dense bone trabeculae and fibrotic marrow tissue (infiltrated by a small number of lymphocytes and plasma cells).
- Clinical features; usually asymptomatic adults, commonly involves the mandible
Focal Sclerosing Osteomyelitis
- Focal bony reaction to low-grade periapical infections or pulpal inflammation
- Primarily affects children and young adults
- Usually asymptomatic
- Radiographically, often associated with a narrowed periodontal space or periapical area , which is denser then the surrounding bone
- Pathology; characterized by a dense mass of sclerotic bone trabeculae, scanty fibrotic marrow tissue, with an infiltration of a small number of lymphocytes and plasma cells.
Chronic Osteomyelitis with Proliferative Periostitis (Garré's)
- Characterized by hard bony swelling around the jaw, often at the periphery
- Essentially periosteal osteosclerosis with reactive vital bone expanding the surface of affected bone
- Primarily children and young adults
- Radiographically shows duplication of the hard cortex - known as onion skin appearance
- Histologically, shows a supra-cortical but subperiosteal mass of parallel layers comprised of reactive new bone or osteoid. Connective tissue usually fibrous, with occasional lymphocycte and plasma cells patches.
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Description
This quiz explores diseases affecting the periapical tissues, including definitions and types of periapical lesions and periodontitis. It discusses the etiology behind these conditions and symptoms associated with acute periapical periodontitis. Test your knowledge of dental health and terminology in this specialized area!